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353 Pregnancy

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    Nursing 353Maternal Risk Factors

    Fetal AssessmentFebruary 3rd, 2005

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    High Risk Pregnancy

    The life or health of the mother or fetus isjeopardized

    Examples include:

    GDM

    Previous loss

    AMA

    HTN

    Abnormalities with the neonate

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    Perinatal Mortality

    Overall maternal deaths are small

    Many deaths a preventable

    Education and prenatal care are veryimportant

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    Antepartum Testing

    FKCs BID

    UTZ FHR

    Gestation age

    Abnormalities IUGR

    Placental location and quality

    AFI

    Position

    BPP

    Doppler flow

    Fetal growth

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    Ultrasound

    Can be done abdominally or transvaginally

    1st trimester done to detect viability,calculate EDC

    2nd trimester done to detect anomalies,calculate EDC

    3rd

    trimester done to do BPP, fetal growthand well-being, AFI

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    Doppler Flow Analysis via UTZ

    Study blood blow in the fetus and placenta

    Done on high risk mothers:

    IUGR HTN

    DM

    Multiple gestation

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    AFI

    Polyhydramnios too much amniotic fluid

    Oligohydramnios too little amniotic fluid

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    Biophysical Profile

    Includes 5 components:

    Fetal breathing movements

    Gross body movements

    Fetal tone

    AFI

    NST - reactive

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    Amniocentesis

    Used with direct ultrasound

    Less than 1% result in complications

    Complications include: Fetal death, miscarriage

    Maternal hemorrhage

    Infection to fetus

    Preterm labor Leakage of amniotic fluid

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    Meconium

    Visual inspection of amniotic fluid

    Meconium is defined as thin and thick andparticulate

    Associated with fetal stress: hypoxia,umbilical cord compression

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    CVS

    Done between 9 -12 weeks

    Genetic studies

    Removal of small amount of tissue fromthe fetal portion of the placenta

    Complications: vaginal spotting,

    miscarriage, ROM, chorioamnionitis If done prior to 10 weeks, increased risk

    of limb anomalies

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    AFP

    Genetic test

    Done with mothers blood

    16-20 weeks gestation Mandated by state of California

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    EFM

    Third trimester goal is to continue toobserve the fetus within the intrauterineenvironment

    Goal: dx uteroplacental insufficiency

    NST vs. CST

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    NST

    90% of gross fetal body movements areassociated with accelerations of the FHR

    Can be performed outpatient

    Not as sensitive

    User friendly but must interpret strip

    Fetus may be in a sleep state or affectedby maternal medications, glucose etc.

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    NST

    To be reactive must meet criteria

    Must be at least 20 minutes in length

    Must have 2 or more accelerations thatmeet the 15 X 15 criteria

    Must have a normal baseline

    Must have LTV

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    NST

    To stimulate a fetus that is not meetingcriteria:

    Change positions of the mother LS, RS

    Increase fluids

    Acoustic stimulator

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    CST

    Done in the inpatient setting only!

    Has contraindications

    May be expensive if meds/IV needed Monitored for 10 minutes first

    Then may use nipple stimulation or

    oxytocin stimulation No late decelerations than negative CST

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    CST

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    Endocrine and Metabolic Disorders

    #1 Diabetes Mellitus

    Disorders of the thyroid

    Hyperemesis

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    Diabetes

    Hyperglycemia

    May be due to inadequate insulin action ordue to impaired insulin secretion

    Type 1 insulin deficiency

    Type 2 insulin resistance

    GDM glucose intolerance duringpregnancy

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    DM

    10th week fetus produces it own insulin

    Insulin does not cross the placental barrier

    Glucose levels in the fetus and directlyproportional to the mother

    2nd and 3rd trimesters decreased

    tolerance to glucose, increased insulinresistance, increased hepatic function ofglucose

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    Diabetic Nephropathy

    Increased risks for:

    Preeclampsia

    IUGR

    PTL

    Fetal distress

    IUFD

    Neonatal death

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    DM

    Poor glycemic control is associated withincreased risks of miscarriage at time ofconception

    Poor glycemic control in later part ofpregnancy is assoc. with fetal macrosomiaand polyhydramnios

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    Polyhydramnios

    May compress on the vena cava and aortacausing hypotension, PROM, PPhemorrhage, maternal dyspnea

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    Macrosomia

    Disproportionate increase in shoulder andtrunk size

    4000-4500gms or greater

    Fetus will have excess stores of glycogen

    Increased risks of

    Shoulder dystocia C/S

    Assisted deliveries

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    IUGR

    Compromised uteroplacental insufficiency

    02 available to the fetus is decreased

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    RDS

    Increased RDS due to high glucose levels

    Delays pulmonary maturity

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    Neonatal Hypoglycemia

    Usually 30-60 minutes after birth

    Due to high glucose levels duringpregnancy and rapid use of glucose afterbirth

    Related to mothers level of glucose control

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    Labs with DM

    HBA1c

    1 hour PP

    FBS

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    Diet

    Sweet success diet

    Well balanced diet

    6 small meals / day Have snack at HS

    Never skip meals

    Avoid simple sugars

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    Insulin

    Regular/Lispro and NPH

    2/3 dose in am and 1/3 dose in pm

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    Monitoring Glucose Levels

    FBS

    1 hour PP

    HS 5 checks / day

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    Fetal Surveillance

    NSTs done around 26 weeks, weekly

    At 32 weeks done biweekly with NST/BPP

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    Infections and DM

    Infections are increased:

    Candidiasis

    UTIs

    PP infections

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    DM

    Increased risk of IUFD after 36 weeks

    Increased congenital anomalies

    Cardiac defects

    CNS defects

    Spina bifida

    anencephaly

    Skeletal defects

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    DM and labor

    Continuous fetal monitoring

    Blood glucose levels in tight control

    Be prepared for CPD

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    GDM

    Women with GDM at risk of developingDM later on in life

    NSTs around 28 weeks

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    Hyperthyroidism

    Typically caused by Graves disease

    S/S:

    Fatigue

    Heat intolerance

    Warm skin

    Diaphoresis

    Weight loss

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    Should be treated in pregnancy

    Tx with PTU

    Beta blockers

    May lead to thyroid storm if untreated

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    Hypothyroidism

    Usually caused by Hashimotos

    S/S:

    Weight gain

    Cold intolerance

    Fatigue

    Hair loss

    Constipation

    Dry skin

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    Cardiovascular Disorders

    The heart must compensate for theincreased workload

    If the cardiac changes are not welltolerated than cardiac failure can develop

    1% of pregnancies are complicated byheart disease

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    NY Heart Association Classes

    Class I

    Class II

    Class III Class IV

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    Cardiac output is increased

    Peak of the increase 20-24 weeksgestation

    Cardiac problems should be managed withcardiologist

    Mortality with pulmonary hypertension andpregnancy is more than 50%

    Diet: low sodium

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    Nursing Care

    Avoiding anemia

    Avoid strenuous activity

    Monitor for: cardiac failure and pulmonarycongestion

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    During Labor

    Side lying position

    Prophylactic antibiotic

    EpiduralAttempt vaginal delivery

    If anticoagulant therapy is needed:

    Heparin Lovenox

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    MVP

    Common and usually benign

    May experience syncope, palpitations anddyspnea

    Prophylactic antibiotics given beforeinvasive procedure or birth

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    Anemia

    Most common iron deficiency

    Hgb falls below 12 (most labs)

    Typically seen in the end of 2nd trimester

    Iron supplementation

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    Folic Acid Deficiency Anemia

    Increases risk of NTD, cleft lip

    Recommended dose 400 mcg/day

    Supplemented in cereal and many otherfoods

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    Sickle Cell Anemia

    Abnormal hemoglobin SS types in theblood

    People have recurrent attacks of fever and

    pain in the abdomen and extremities

    Caused from tissue hypoxia, edema

    African-Americans

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    Thalassemia

    Common anemia

    Insufficient amount of Hgb is produced tofill the RBCs

    Mediterranean region

    Genetic disorder

    May be associated with LBW babies andincreased fetal death

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    Asthma

    Common with FH

    1-4% of pregnant women have Asthma

    Possible adverse events associated with

    asthma: LBW

    Perinatal mortality

    Preeclampsia Complicated labor

    Hyperemesis

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    Asthma Continued

    Goal is to relieve the attack, prevent theasthma attack, and maintain 02

    Should be managed with OB and ENT

    May require tx: albuterol, steroids, O2

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    Epilepsy

    Seizure disorder

    May result from developmentalabnormalities or injury

    20% have an increase in seizure activityduring pregnancy

    Risks: more seizures, risk of vaginalbleeding, abruptio placentae, fetus mayexperience seizures

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    Epilepsy Continued

    Use of antiepeleptic meds duringpregnancy has been linked to risks for thefetus

    Smallest therapeutic dose to be given

    Daily folic acid supplementation

    Managed with OB and neurologist

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    RA

    Chronic arthritis

    Pain upon movement and swelling in jointspaces

    More often in women

    2/3 of women with RA find the severity ofsymptoms decrease during pregnancy

    Typically give baby ASA

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    SLE

    Inflammatory disease, autoimmuneantibody production

    Advised to wait until in remission for 6

    months to become pregnant

    15-60% of women will developexacerbation of SLE during pregnancy or

    postpartum

    Tx: ASA and steroids

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    Cholelithiasis

    More often in women

    Pregnancy makes women more vulnerable

    Surgery often delayed until after delivery

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    Appendicitis

    Dx may take more time to find

    Sxs: abdominal pain, nausea, vomiting,loss of appetite

    Increases incidence of PTL or SAB

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    TORCH

    Toxoplasmosis protozoan infection,neonatal effects jaundice,hydrocephalus, microcephaly

    Other- Heb A or B, Group B, Varicella, HIV

    Rubella (German measles) if contractedin 1st Trimester fetus may have congenital

    deformities

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    TORCH

    CMV- transmitted person to person, maycause CNS damage to fetus

    Herpes Simplex (HSV 2) if initial

    infection occurs in pregnancy, higherincidence of perinatal loss. Fetus maypick up virus if present in the vagina

    during labor

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    Mental Health Disorders

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    Anxiety Disorders

    Most common mental disorders

    Include: phobias, panic disorders, OCD,PTSD

    Tx: relaxation techniques, breathingexercises, imagery

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    Depression in Pregnancy

    6% of women develop depression for the1st time during pregnancy

    Tx: counseling and tx with SSRIs

    Wellbutrin only med named as Category B

    Many women opt to DC meds duringpregnancy

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    Substance Abuse in

    Pregnancy

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    Smoking

    Risks of any amount of smoking include:

    SAB

    SGA

    Bleeding

    IUFD

    Prematurity

    SIDS

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    Alcohol

    Many women reluctant to tell health careprovider

    Risks:

    LBW

    Mental retardation

    Learning and physical deficits

    With FAS severe facial deformities

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    Alcohol during Pregnancy

    Risks to mother:

    HTN

    Anemia

    Nutritional deficits

    Pancreatitis

    Cirrhosis

    Alcoholic hepatitis

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    Marijuana

    Crosses the placenta and causes increasedcarbon monoxide levels in mothers blood

    May cause fetal abnormalities

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    Cocaine

    In the US, 10-15% of all pregnant womenuse cocaine

    Problems associated with use: polydrug

    use, poor health, poor nutrition, STIs,infections, HIV

    Poverty big issue

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    Cocaine in Pregnancy

    Maternal effects: Cardiovascular stress

    Tachycardia

    HTN Dysrhythmias

    MI

    Liver damage

    Sz Pulmonary disease

    Death

    Fetal Complications:Abruptio placentae

    PTL

    Precipitous labor Risks for abdominal

    pregnancy

    Fetal complications

    after delivery

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    Opiates in Pregnancy

    Drugs include: heroin, Demerol, morphine,codeine, methadone

    Methadone is used to treat addiction to

    other opiates Possible effects on pregnancy and heroin

    use are: Preeclampsia, PROM, infections,PTL

    Tx: Methadone and psychotherapy

    Goal: prevent withdrawal symptoms

    h h

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    Methamphetamine

    CNS stimulant

    Most common use n the 18-30 yr oldrange

    Neonatal complications include:

    IUGR

    PRL/PTB

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    Postpartum Psychologic

    Complications

    B b Bl

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    Baby Blues

    Usually within 4 weeks of childbirth

    Many experience this

    PPD

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    PPD

    Intense sadness, crying all the time, moodswings, fears, anger, anxiety, irritability

    Incidence of PPD at 8 weeks 12% and

    8% at 12 weeks

    Many women feel guilty

    May need tx but usually resolves on own

    P t t P h i

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    Postpartum Psychosis

    Delusions, hurting self or the infant,emotional lability, insomnia,suspiciousness, confusion, obsessive

    concerns regarding the baby 1-2/1000 births

    35-60% recurrence with each subsequentbirth

    Usually symptoms appear within 8 weeksof birth

    M di l M t

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    Medical Management

    Supportive family

    Intense psychotherapy

    Emergency

    Tx: SSRIs

    SSRIs contraindicated while breastfeeding

    1 A client asks the nurse to again explain

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    1. A client asks the nurse to again explainthe purpose of the amniocentesis test.

    The nurse responds that one purpose ofthis test is to indicate the:

    A. Accurate age of the fetus

    B. Presence of certain congenital anomalies C. Biparietal diameter of the skull

    D. Hormone content of the amniotic fluid

    E. Mainly the presence of Downs syndrome

    2. The nurse explains to a new mother

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    that the condition of SGA is caused by:

    A. Placental insufficiency

    B. Maternal obesity

    C. Primipara

    D. Genetic predisposition

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    4. The nurse in the newborn nursery

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    4. The nurse in the newborn nurseryunderstands that assessing a newborn

    with a diabetic mother, initially the insulinlevel would be:

    A. Higher than in normal infants

    B. Lower than in normal infants

    C. The same as in normal infants

    D. Varied from baby to baby

    5. A client is admitted to L&D, at 38

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    5. A client is admitted to L&D, at 38weeks gestation. She is there forevaluation because she is experiencingpolyhydramnios. The nurse understandsthat this diagnosis means that:A. There is the normal amount of amniotic

    fluid, thinner in volume B. A less-than-normal amount of amniotic

    fluid is present

    C. An excessive amount of amniotic fluid is

    present D. A leak is causing the fluid to accumulate

    outside the amniotic sac


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