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Adhesive Capsulitis Presentation May 2009

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    Learning OutcomesAnatomy of the shoulder

    Definition

    Aetiology

    Pathophysiology

    Clinical Phases

    Physical presentation Treatment and medication

    Follow-up

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    Anatomy of the shoulder

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    Anatomy cont

    Posterior view: Anterior view:Teres major

    www.instantanatomy.net

    Subscapularis

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    Definition In 1934, Codman stated, "This entity [FS] is difficult to

    define, difficult to treat, and difficult to explain fromthe point of view of pathology.

    Two principal features:

    gradual onset of pain and reduced range of motion(active and passive) of the GHJ in a capsular pattern

    Uncertain aetiologyDias et al (2005)

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    Epidemiology No regional variations

    Prevalence is reported to be 2-5%, with an 11%prevalence in individuals with diabetes. For patientswith T1DM, the risk is approximately 40%.

    FS affects women more frequently than men and themean age of onset is over 40

    Brue et al (2007)

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    Aetiology Idiopathic disorder.

    risk factors: trauma, diabetes, hyperthyroidism, and

    dyslipidemia, CVA with upper-extremity paresis,brachial plexus injury, cervical spinal cord injury, andParkinson disease.

    repetitive movements of the upper extremities

    active GHJ synovitis in relation to a systemicinflammatory rheumatologic disorder.

    surgery to the shoulder, with post-op immobilizationDias et al (2005)

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    Pathophysiology GHJ synovial capsule is often involved

    hyperplastic fibroplasia and excessive type III collagensecretion that lead to soft-tissue contractures (of thecoracohumeral ligament, soft tissues of rotatorinterval, the subscapularis muscle, the subacromial

    bursae). late phase dx

    Why the pain precedes the contracture and why itresolves before the contracture remains unclear.

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    Pathophysiology cont Neurological mechanisms: peripheral alpha-

    adrenoreceptor hyperresponsiveness, dorsal-rootreflexes (DRRs), central nervous system (CNS) factors,myxoid globular degeneration, and sympatheticautonomic hyperactivity.

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    Three Clinical Phases Phase 1 - freezing stage: insidious onset of

    predominantly nocturnal pain (without inflammatory,exudative cellular joint processes). End of ROM can

    increase pain. Lasts 2-9 months.

    Phase 2 - frozen stage: pain gradually subsides andprogressive limitation in ROM occurs in a capsular pattern. ADLs can be severely affected. Last 3-12 months.

    Phase 3 thawing stage - pain progressively decreases,and limitations in ROM spontaneously recover over 12-24months.

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    Physical presentation Initial night pain associated with movements of the

    shoulder e.g., combing one's hair, reaching overheadfor a seat belt (both use AB + ext rot), reaching forone's back pocket (ext and int rot).

    Pain can be as high as 10/10 (VAS)

    In ~90% of patients, the pain lasts for 1-2 years beforesubsiding.

    Patients with a painful FS have pain during resistedcontraction of all of the rotator cuff tendons

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    Physical presentation cont progressive limitation of PROM,

    characterized by a painfulcapsular end-feel (externalrotation, followed by abduction,medial rotation and flexion).

    Patients typically lift the entireshoulder girdle when trying tolift the arm.

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    Treatment Patient education

    dx process, including recovery time

    importance of a HEP to increase ROM and function

    Occupation

    Deterrence (early mobilization and individuals who dorepetitive activities should pay special attention totheir posture and the ergonomics of their workstation)

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    Treatment Jewell et al, (2009): a randomized clinical trial investigated

    the effectiveness of joint mobilization and exercise forpatients with FS

    The treatments that increased the likelihood ofpainreduction and improved function were:

    - Joint mobilization performed by a PT and exercise

    The treatments that decreased the likelihood of painreduction and improved function were:

    - Ultrasound, Massage, Iontophoresis,

    Phonophoresis

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    TreatmentVermeulen et al (2000) showed that passive

    mobilization at the end of range was more effective for

    improving ROM and function than in the pain-freezone. However, the overall difference between theinterventions was small.

    In addition, patients appeared to achieve greatestimprovement in ROM when treatment wasadministered early (Liaw et al, 2000).

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    Maitlands mobilisations

    Accessory: a movement that cannot be performed

    independently by the patient but is critical to normalmovement e.g. glide, slide, spin, translate

    Physiological: movements that a person can carry out

    actively e.g. flexion/extension, abduction/adduction,medial/lateral rotation, circumduction

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    Grading oscillatory joint mobilisation

    treatment techniques

    R1

    Grade 1

    Grade II

    Grade III

    Grade IV

    Beginning

    of range

    End of

    expected

    rangeMaitland

    freezing

    stage

    frozen &

    thawing

    stage

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    Examples: mobilisations at GHJ

    AP glide in abduction

    AP glide when shoulder is in flexion Lateral glide in abduction

    Distraction to GHJ in abduction

    passive physiological to increase range of lateralrotation and flexion

    Use hold and relax technique to increase flexion

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    Maitlands Mobilisations

    Desired effects:

    Recover full range painless movement

    Stretching a stiff joint to restore range

    Stretching to lengthen contracted, fibrosed or

    shortened muscle tissue

    Relieve pain

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    Successful treatment

    * Increased ROM and decreased pain at end of range

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    Other treatment

    physical therapy associated with an intra-articularinjection of corticosteroid improves function andROM more rapidly (p < 0.005) than does intra-

    articular corticosteroid injection alone (Carette et al,2003)

    Medication for pain relief

    OT give advice for performing ADLs (eg, dressing,bathing, grooming).

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    Other treatment cont

    Buchbinder et al (2004)conducted a randomised

    double-blind placebo controlled trial supporting theuse of hydrodilatation for FS. Significantimprovements in function,pain, and ROM at 3 weekswere found, and this benefit was maintained at 6

    weeks.

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    Other treatment cont

    Jones et al (1999) conducted a randomized controlled

    trial compare the effectiveness of a singlesuprascapular nerve block with that of series of intra-articular corticosteroid injections. Pain decreased andROM increased more rapidly with the nerve block.

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    Follow-up

    Bilateral FS may find that even basic ADLs are achallenge. (eg, toileting, hygiene, dressing, driving,fastening a seat belt).

    Further outpatient care (based on the patient's currentstatus and functional goals)

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    Conclusion FS is of insidious onset, with progressive pain and

    decreased ROM in a capsular pattern

    Physical therapy treatment involves jointmobilisations and exerciseand has been shown toincrease the likelihood of pain reduction andimproved function in patients with FS

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    References Jewell, et al (2009). Interventions associated with an increased or decreased

    likelihood of pain reduction and improved function in patients with adhesivecapsulitis: a retrospective cohort study. Phys Therapy, 89(5):419-29.

    Carette et al (2003). Intraarticular corticosteroids, supervised physiotherapy, or

    a combination of the two in the treatment of adhesive capsulitis of theshoulder: a placebo-controlled trial.Arthritis Rheum., 48(3):829-38

    Vermeulen et al (2000). End-range mobilization techniques in adhesivecapsulitis of the shoulder joint: A multiple-subject case report. Phys Ther.,80(12):1204-13.

    Liaw SC (2000). The effect and timing of physiotherapy on change in range ofmotion and function in frozen shoulder. Physiother Singapore, 3(3):82-6.

    Roy et al (2007). Adhesive Capsulitis , Available at

    http://emedicine.medscape.com/article/326828-overview

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    References Jones et al (1999). Suprascapular nerve block for the treatment of frozen

    shoulder in primary care: a randomized trial. Br J Gen Pract., 49(438):39-41.

    Buchbinder et al (2004). Arthrographic joint distension with saline and steroidimproves function and reduces pain in patients with painful stiff shoulder:

    results of a randomised, double blind, placebo controlled trial. Annals of theRheumatic Diseases; 63:302-309

    Dias et al (2005). Frozen shoulder. BMJ, 331:1453-1456

    Brue et al (2007). Idiopathic adhesive capsulitis of the shoulder: a review.Journal of Knee Surgery, Sports Traumatology, Arthroscopy, vol 15(8), 1433-7347,

    Hengeveld et al (2005). Maitlands peripheral manipulation (4th ed.).Elsevier/Butterworth Heinemann, Michigan

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    Any Questions?


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