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Cardiac Symptoms 2

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    CARDIAC

    SYMPTOMS

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    CHEST PAIN

    ANGINA (ISCHEMIC CARDIAC PAIN): Squeezing,crushing, strangling, constricting pain in center of chest.Pain may radiate to left shoulder, left arm, right shoulder,

    jaw.

    Stable (Typical) Angina: Angina upon effort, or angina

    induced by increased blood pressure or increasedheart-rate. Angina is relieved by nitroglycerin, although

    nitroglycerin is not specific to this type of angina.

    Levine's Sign: Patient makes fist and holds it up to

    his chest, to describe the pain.

    Second-wind Phenomenon: If patient repeatssame activity after the attack, he may not feel the

    attack again the second time.

    Walk-through Angina: The pain subsides as patient

    continues the activity.

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    Atypical Angina:Atypical presentation of typicalangina.

    Atypical Symptoms: Sharp or stabbing pain, rather

    than crushing pain.

    Atypical Causes:Angina with change in position,for example, rather than angina strictly upon effort.

    Angina Equivalents: Other symptoms that are

    caused by myocardial ischemia.

    Exertional dyspnea.

    Nausea, indigestion.

    Dizziness, sweating.

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    Variant Angina (Prinzmetal Angina):Paradoxic angina occurring during rest but usually

    not during exercise. It is caused by coronary artery

    spasm. It can be hard to spot because it can coexistwith typical angina.

    Characteristic ECG findings can help distinguish

    variant angina from typical angina.

    Nitroglycerin will probably still relieve pain, as it

    relaxes coronary arteries.

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    Unstable Angina: Angina even at rest, or anginathat has recently gotten worse. It is associated with

    sharply increased risk for myocardial infarct within 4

    months.

    Angina Decubitus is a specific term for anginaoccurring at rest.

    Myocardial Infarct: Typical presentation =Unstable angina lasting longer than 15 minutes, that

    is not relieved by nitroglycerin.

    Silent MI's and MI's with atypical presentation do

    occur.

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    NON-ISCHEMIC CARDIAC PAIN:Mitral Valve Prolapse: Usually asymptomatic, but may

    present with an intermittent, sharp, sticking pain over

    left precordium.Pericarditis: The patient feels relief by shallow

    breathing and by sitting up and leaning forward.

    Dissecting Aneurysm: Sudden, severe tearing pain,

    radiating to the abdomen, neck, or back, depending onwhere the aneurysm is going.

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    :Pulmonary Embolism: May be asymptomatic, or thepatient may feel a dull tightness if the embolus is large

    enough.Paroxysmal Dyspnea is the most common symptom of

    pulmonary embolism.

    Pleurisy: Pain upon breathing. May be caused bypulmonary embolism, pneumonia, bronchitis, or pleural

    effusion.

    Pulmonary Hypertension: Dyspnea is a morecommon symptoms than pleuritic pain.

    Pneumothorax: Pain may be confused with pain of an

    MI.Mediastinal Emphysema: Free air in the mediastinumproduces chest tightness and dyspnea.

    Hamman's Sign: Crunching, rasping sound heard

    synchronous with the heartbeat, indicative ofmediastinal em h sema.

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    GASTROINTESTINAL CHEST PAIN:Esophageal Spasm: Substernal chest pain anddysphagia.

    Esophageal Reflux (GERD): Chest pain relievedby antacids.

    Gallstone Colic: Colicky RUQ pain radiating to backand to right shoulder. Occasionally it may be confused

    with angina.

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    CHEST WALL PAIN:TIETZE'S SYNDROME

    (COSTOCHONDRITIS): Inflammation ofCostochondral joints. Pain is often localized and can

    be elicited by palpating the sternum over the involved

    ribs.

    HERPES ZOSTER:Pain may precede the

    appearance of the rash. Both pain and rash follow

    dermatomal distribution.

    DACOSTA'S SYNDROME: Psychogenic painusually localized to the cardiac apex. May be

    associated with anxiety.May also see palpitations, hyperventilation,

    dyspnea, weakness, depression, or other signs of

    anxiety.

    Vertebral Column Disease: It may occasionallylead to anterior chest ain.

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    DYSPNEA

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    DyspneaDyspnea is defined as an abnormally uncomfortableawareness of breathing

    it should be regarded as abnormal only when it occurs at

    rest or at a level of physical activity not expected to cause

    this symptom.

    The sudden development of dyspnea suggests pulmonaryembolism, pneumothorax, acute pulmonary edema,

    pneumonia, or airway obstruction

    In contrast, in most forms of chronic heart failure,

    dyspnea progresses slowly over weeks or months. Such aprotracted course may also occur in a variety of unrelated

    conditions, including obesity, pregnancy, and bilateral

    pleural effusion.

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    QUANTITATION OF DYSPNEA

    - amount of exertion

    - condition of individual

    SUDDEN UNEXPECTED DYSPNEIC EPISODE ARREST -

    Pulmonary Emboli

    Spontaneous Pneumothorax

    Anxiety

    EXERTIONAL DYSPNEA: Dyspnea on exertion is a commonsymptom of mild or severe Congestive Heart Failure.

    DYSPNEA at REST:

    Pulmonary causes of dyspnea (PE, COPD,

    pneumothorax) often occur at rest. With cardiac

    problems, dyspnea usually does not occur at rest, or it

    is overshadowed by angina.

    Anxiety Dyspnea: Difficulty breathing due to anxiety

    occurs only at rest.

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    ORTHOPNEA: Dyspnea occurring with patient in thesupine position. Orthopnea is a sign ofCongestive Heart

    Failure that is more severe than that associated with

    exertional dyspnea.CAUSE: Supine position increases pulmonary blood

    flow ------> exacerbate pulmonary congestion and

    pulmonary edema. The problem is relieved by resuming

    a more upright position.

    Two-Pillow, Three-Pillow Orthopnea: Terms todescribe the severity of the orthopnea. Three pillow is

    worse than two-pillow.

    PAROXYSMAL NOCTURNAL DYSPNEA (PND):

    Similar to orthopnea, except it has sudden onset and occursonly after the patient has been lying down at rest for at least

    an hour.

    Unlike orthopnea, It is not relieved immediately by sitting

    up.

    Patient is usually able to return to sleep, eventually.

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    ORTHOPNEA

    LV Failure

    Asthma

    COPD

    Dilat. paralysis of diaphragm

    TREPOPNEA

    Dyspnea in R orL decubitus

    Heart Failure

    PLATYPNEADyspnea in upright position

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    PULMONARY EDEMA: Pulmonary edema is usually a

    manifestation of left-ventricular heart failure. Peripheral edema

    associated with CHF is a manifestation of right-sided heart

    failure (Cor Pulmonale).SYMPTOMS: Severe symptoms. Extreme anxiety, dyspnea,

    air hunger, cold sweats, fear of impending death.

    SIGNS: Pink, frothy sputum, and bubbly breath sounds.

    VALVULAR HEART DISEASE: Mitral Stenosis is associated

    with dyspnea.CONGENITAL HEART DISEASES:

    Tetralogy of Fallot: Exertional dyspnea is common.

    Ventricular Septal Defect: Tachypnea and sweating. Late

    cyanosis.

    CARDIAC -vs- PULMONARY DYSPNEA:OTHER CAUSES OF SHORTNESS OF BREATH:

    Kussmaul Respiration: Intense hyperventilation

    (respiratory alkalosis) occurring with Diabetic

    Ketoacidosis, as a compensatory mechanism to relieve

    the metabolic acidosis.

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    DIFFERENTIAL DIAGNOSISINTERSTITIAL OBSTRUCTIVE DISEASE

    LARGE EXTRATHORACIC AIRWAY OBSTRUCTION

    - ACUTE -

    Aspiration of foreign bodyAngioneurotic edema of glottis

    - CHRONIC -

    Tumors

    Fibrotic Stenosis after tracheotomy or intubation

    - manifests as stridor & retraction of supraclavicular fossae

    with inspiration

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    OBSTRUCTION OF INTRATHORACIC AIRWAY

    - ACUTE INTERMITTENT WITH WHEEZING - Asthma

    - CHRONIC COUGH WITH EXPECTORATION -

    Chronic bronchitis - generalized

    Bronchiectasis - localized

    I. EMPHYSEMA

    - diagnosis of exertional dyspnea

    - dyspnea at rest

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    II. DIFFUSE PARENCHYMAL LUNG DISEASE -

    - ACUTE - Pneumonia

    - CHRONIC - Sarcoidosis, Pneumoconiosis

    - Decreased lung volume

    - Decreased lung complianceIII. PULMONARY VASCULAR OCCLUSIVE DISEASE

    - Pulmonary Embolism

    - Abnormal ABG with normal volumes of lung

    IV. DISEASES OF CHEST WALL

    - Severe Kyphoscoliosis, Spondylitis- Neuromuscular Disorders

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    V. HEART DISEASE

    - increased pulmonary capillary pressure, obstructive disease

    of pulmonary vein, pulmonary capillary hypertension

    - Mechanism:

    - increased pulmonary vascular hydrostatic pressure --->Transudation ---> decreased lung compliance ---> stimulation

    of J receptor in alveolar interstitial space

    - fatigue of respiratory muscles

    - metabolic acidosis

    - Orthopnea- PND (?)

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    DIAGNOSISHEART FAILURE- previous history of heart e.g. AMS

    - S3 & S4

    - Left ventricular hypertrophy, Jugular venous distention,

    Edema

    Cardiac Asthma - diaphoresis, more bubbly airway sound,common occurrence of cyanosis

    COPD- dyspnea is more gradual than CHF-

    Except - with infection, Pneumothorax or exacerbation

    of asthma

    ANXIETY NEUROSIS- different from pulmonary embolism- - sighing respiration,

    bizarre pattern of breathing

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    PULMONARY EDEMACARDIOGENIC

    - decreased compliance, increased resistance to small airway, increased

    lymphatic flow

    NONCARDIAC CAUSE- Hypoalbuminemia

    - liver disease, NS, protein losing enteropathy

    - increased negativity of interstitial

    pressure

    - sudden massive evacuation of large pneumothorax- lymphatic blockage secondary to fibrotic and inflammatory disease

    or lymphangitic carcinomatosis

    - disruption of alveolocapillary membrane

    - Alloxan administration

    - diffuse pulmonary infection, aspiration shock (Gram-

    negative infection, sepsis, pancreatitis, cardiopulmonarybypass)

    OTHER CAUSES

    - narcotic overdose

    - exposure to high altitude

    - neurogenic pulmonary edema- CNS disorder

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    PALPITATIONS:An unpleasant awareness of one'sown heart-beat. Often described as fluttering, or skipping a

    beat.

    Paroxysmal Atrial Tachycardia: May cause palpitationswith an instantaneous onset.

    Premature Ventricular Contractions (PVC's): May be

    experienced as palpitations or a skipped beat. The

    premature contraction is followed by a compensatory

    pause, to allow for ventricular filling.

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    FATIGUE: Non-specific finding often found with heart disease.

    FATIGUE CAUSED BY HEART DISEASE: It usually occurs

    later in the day or in the evening. Fatigue early in the

    morning is usually not associated with heart disease, unlessthe patient was aroused from REM sleep.

    The heart disease gets worse, as the patient

    experiences onset of fatigue earlier in the day.

    OTHER CAUSES OF FATIGUE: Lots. Chronic illness of

    many types, anemia, psychological causes.

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    SYNCOPE: Fainting, transient loss of consciousness.VASOVAGAL EVENTS: Most common cause of

    syncope, it is caused by excessive stimulation of the

    Vagus nerve ------> excessive bradycardia andreduced blood-flow to the brain.

    Anxiety: It is usually associated with acute anxiety

    or excessive emotion. The Vagal hyperactivity is

    thought to be a hypersensitive response to

    sympathetic outflow.

    CARDIOVASCULAR CAUSES:

    1. Arrhythmias:

    STOKES-ADAMS SYNDROME: Syncope

    caused by reduced cardiac output secondary toan arrhythmia.

    Both severe tachycardia and bradycardia can

    reduce cardiac output, leading to syncope.

    Severe tachycardia reduces cardiac output by

    reducing ventricular filling time.

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    2. Cardiac Outflow Tract Obstruction:

    Aortic Stenosis may lead to syncope.

    Myxomas, benign myocardial tumors, may cause

    outflow obstruction and lead to syncope.Tetralogy of Fallot is associated with fainting attacks.

    Myocardial Ischemia

    3. Carotid Sinus Syncope: Hypersensitivity of the Carotid

    Sinus in elderly men is common cause of syncope.

    4. Impaired Vasomotor Reflexes: Impairment of

    Baroreceptors. Syncope is associated with orthostatic

    hypotension.

    5. Decreased Blood Volume

    FLUID REMOVAL:Micturition Syncope: Syncope occurring with micturition

    but at no other time. Associated with removal of fluid from

    the body.

    POST-TUSSIVE SYNCOPE: Syncope after a bout of coughing, or

    after the Valsalva maneuver, may occur in patients with COPD.

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    COUGH

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    COUGH

    DIAGNOSTIC EVALUATION

    - Acute or Chronic

    - Productive or not

    ACUTE - infections

    CHRONIC - postnasal drip

    CHARACTERISTICS OF COUGH

    BARKING - PertussisRELATED TO MEALS -Tracheoesophageal Fistula

    Hiatal Hernia,Esophageal Diverticulum

    RELATED TO CHANGE IN POSITION

    Lung Abscess

    Localized BronchiectasisPUTRID SPUTUM - Lung Abscess

    PINK FROTHY SPUTUM - Pul. Edema

    MUCOID MASSIVE SPUTUM - Alveolar Cell CA

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    PHYSICAL EXAMINATION

    INSP. STRIDOR & WHEEZING -

    Laryngeal Disease

    INSP. & EXP. RONCHI - Tracheal & major airway involvement

    COARSE INSP. RALES - Interstitial Fibrosis &/or Edema

    FINE RALES - Pneumonitis &/or Pulmonary Edema

    COMPLICATION

    Syncope

    Rupture of Emphysematous BlebsRib Fracture

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    HEMOPTYSIS

    - Recurrent hemoptysis should be managed as if it

    were the initial hemoptysisETIOLOGY

    1. INFLAMMATORY

    Bronchitis

    Bronchiectasis

    Pulmonary TuberculosisAbscess Pneumonia (Klebsiella)

    2. NEOPLASTIC

    Lung CA

    Bronchial Adenoma

    3. OTHERS

    Pulmonary Embolism

    Left Ventricular Failure

    Mitral Stenosis

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    TRAUMATIC -

    Foreign body

    Contussion

    Primary Pulmonary HPN

    Arteriovenous Malformation (?)Eisenmenger Syndrome

    VASCULITIS -

    Wegeners Granulomatosis

    Goodpastures Syndrome

    AMYLOID4. HEMORRHAGIC DIATHESIS

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    DIFFERENCE OF HEMOPTYSISWITH HEMATEMESIS

    HEMOPTYSIS

    - tingling sensation in throat

    - bright red

    - frothyHEMATEMESIS

    - nausea & abdominal discomfort

    - magenta-colored

    J Rare in Metastatic CancerJ Although it may occur in Viral or Pneumococcal Pneumonia,

    still think of other Conditions.

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    END OF LECTUREEND OF LECTURE


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