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Care of Clients With Problems in Oxygenation (Part 2)

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    CARE OF CLIENTS WITHPROBLEMS IN

    OXYGENATION(P ART 2)

    Jotham C. Marfil, RN

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    D IAGNOSTIC TESTS

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    CK-MB (CREATININE KINASE,MYOCAR D IAL MUSCLE)

    An elevation in value indicates myocardialdamage

    An elevation occurs within 4 to 6 hoursand peaks 18 to 24 hours following anacute ischemic attack

    Normal value is 0% to 5% of total; totalCK is 26 to 174 units/L

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    L ACTATE D EHY D ROGENASE (L D H)

    E levations in LDH levels occur 24 hoursfollowing myocardial infarction and peakin 48 to 72 hours

    Normally, LDH1 is lower than LDH2;when the serum concentration of LDH1 ishigher than LDH2, the pattern is

    indicated as flipped, signifyingmyocardial necrosis

    140 to 280 IU/L

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    TROPONIN

    Is composed of troponin C, cardiactroponin I, and cardiac troponin T

    Has a high affinity for myocardial injury;it rises within 3 hours and persists for upto 7 days

    Troponin I lower than 0.6ng/mLTroponin T 0 to 0.2ng/mL

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    C OMPLETE BLOO D COUNT

    RBC decreases in rheumatic heart diseaseand infective endocarditis and increasesin conditions characterized by inadequatetissue oxygenation

    The WBC increases in infectious andinflammatory diseases of the heart andafter MI to dispose necrotic tissueresulting from infarction

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    E levated hematocrit level can result from

    vascular volume depletion

    Decreases in hematocrit and hemoglobinlevels can indicate pneumonia

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    S ERUM LIPI D S

    The lipid profile measures serumcholesterol, triglyceride, and lipoproteinlevels

    Is used to assess the risk of developingcoronary artery disease

    Serum cholesterol lower than 200mg/dLLDL lower than 130mg/dLHDL 30 to 70 mg/dL

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    B- TYPE NATRIURETIC PEPTI D E (BNP)

    Is released in response to atrial andventricular stretch; it serves as a markerfor congestive heart failure

    Should be lower than 100pg/mL

    The higher the level, the more severe thecongestive heart failure

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    ELECTROCAR D IOGRAPHY

    Noninvasive test that records theelectrical activity of the heart and isuseful for detecting cardiac dysrhythmias,location and extent of MI, and cardiachypertrophy and for evaluation of theeffectiveness of medications

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    INT E RV E NTIONS

    Determine the clients ability to lie still;advise the client to lie still, breathenormally, and refrain from talking duringthe test

    Reassure the client that an electrical shockwill not occur

    Document any cardiac medications theclient is taking

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    E CHOCAR D IOGRAPHY

    Noninvasive procedure based on theprinciples of ultrasound and evaluatesstructural and functional changes in theheart

    Heart chamber size is measured, ejection

    fraction is calculated, and flow gradientacross the valve is determined

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    E XERCISE TESTING (STRESS TEST )

    Noninvasive test that studies the heartduring activity and detects and evaluatescoronary artery disease

    Treadmill testing is the most commonlyused mode of stress testing

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    INT E RV E NTIONS

    Obtain an informed consent if required

    Provide adequate rest the night before theprocedure

    Instruct the client to eat a light meal 1 to

    2 hours before the procedure

    Instruct the client to avoid smoking,alcohol and caffeine before the procedure

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    Instruct client to wear nonconstrictive,

    comfortable clothing and supportiverubber-soled shoes for the exercise stresstest

    Instruct the client to notify the physicianif any chest pain, dizziness, or shortnessof breath occurs during the procedure

    Instruct client to avoid taking a hot bathor shower for at least 1 to 2 hours afterthe procedure

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    D IGITAL SUBTRACTION ANGIOGRAPHY

    This test combines x-ray techniques and acomputerized subtraction technique withfluoroscopy for visualization of thecardiovascular system

    A contrast media (dye) is injected

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    INT E RV E NTIONS

    Assess for allergies to seafood, iodine, orradiopaque dyes. Premedicate client withantihistamines or corticosteroids to

    prevent a reaction

    Obtain informed consent

    Monitor vital signs

    Assess injection site for bleeding ordiscomfort

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    M AGNETIC RESONANCE IMAGING

    Noninvasive diagnostic test that producesan image of the heart or great vesselsthrough interaction of magnetic fields,

    radio waves, and atomic nuclei

    Provides information on chamber size andthickness, valve and ventricular function,and blood flow through the great vesselsand coronary arteries

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    INT E RV E NTIONS

    E valuate client for the presence of pacemaker or other implanted items thatpresent a contraindication to the test

    E nsure client has removed all metallicobjects such as watch, jewelry, clothingwith metal fasteners, and metal hairfasteners

    Inform client that she or he mayexperience claustrophobia while inscanner

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    SICKLE CELL ANEMIA

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    Constitutes a group of diseases termedhemoglobinopathies, in which hemoglobin A is partly or completely replaced byabnormal sickle hemoglobin S

    Caused by inheritance of a gene for astructurally abnormal portion of thehemoglobin chain

    Hemoglobin S is sensitive to changes in theoxygen content of the RBC

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    Insufficient oxygen causes the cells to

    assume a sickle cell shape and the cellsbecome rigid and clumped together,obstructing capillary blood flow

    Situations that precipitate sickling includefever and emotional or physical stress; anycondition that increases the need for oxygenor alters the transport of oxygen can resultin sickle cell crisis

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    At risk are those having parents

    heterozygous for hemoglobin S or being of African American descent

    Sickle cell crises are acute exacerbations of the disease, which vary considerably inseverity and frequency ; these include vaso-occlusive crisis, splenic sequestration, andaplastic crisis

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    V ASO -O CCLUSIV E CRISIS

    Caused by stasis of blood with lumping of the cells in the microcirculation, ischemia,and infarction

    Fever, painful swelling of the hands, feet,and joints, and abdominal pain

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    S PL E NIC S EQ U E STRATION

    Caused by the pooling and clumping of blood in the spleen (hypersplenism).

    Profound anemia, hypovolemia, and shock

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    A PLASTIC CRISIS

    Caused by the diminished production andincreased destruction of RBC, triggered byviral infection or the depletion of folic acid

    Profound anemia and pallor

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    INT E RV E NTIONS

    Maintain adequate hydration and bloodflow with IV administered NSS and withoral fluids

    Administer oxygen and blood products asprescribed

    Administer analgesics as prescribed(ATC)

    Administration of meperidine (Demerol) isavoided

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    Assist the child to assume a comfortable

    position so that the child keeps theextremities extended to promote venousreturn

    E levate the bed of the head 30 degrees,avoid putting strain on painful joints, anddo not raise the knee gatch of the bed

    E ncourage consumption of high-calorie,high protein diet, with folic acidsupplementation

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    Administer antibiotics as prescribed toprevent infection

    Monitor for signs of complications,including increasing anemia, decreased

    perfusion, and shock

    Instruct the child and parents about theearly signs and symptoms of crisis and themeasures to prevent crisis

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    IRON D EFICIENCY ANEMIA

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    Iron stores are depleted, resulting in adecreased supply of iron for themanufacture of hemoglobin in RBC

    Commonly results from blood loss,increased metabolic demands, syndromes of GI malabsorption and dietary inadequacy

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    S IGNS AND S YMPTOMS

    Pallor

    Weakness and fatigue

    Irritability

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    INT E RV E NTIONS

    Increase the oral intake of iron

    Instruct the child and parents in foodchoices that are high in iron

    Administer iron supplements as

    prescribed

    Give iron supplements between meals formaximum absorption

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    Give iron supplements with a multivitaminor fruit juice because vitamin C increases

    absorption

    Do not give iron supplements with milk orantacids because these items decrease

    absorption

    Teach the child and parents that a liquidiron preparation stains the teeth andshould be taken through a straw

    Inform parents/client on side effects(constipation, black stools, foul aftertaste)

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    HEMOPHILIA

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    Refers to a group of bleeding disorders

    resulting from a deficiency of specificcoagulation proteins

    Factor VIII deficiency (hemophilia A orclassic hemophilia)

    Factor IX deficiency (hemophilia B or

    Christmas disease)

    Result as an X-linked recessive disorder

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    Most frequently transmitted by the union

    of an unaffected male with a trait-carrierfemale; however, it can result from theunion between an affected male and anormal or carrier female

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    S IGNS AND SYMPTOMS

    Abnormal bleeding in response to trauma orsurgery (usually detected after circumcision)

    E pistaxis

    Joint bleeding causing pain, tenderness,swelling and limited ROM

    Tendency to bruise easily

    Platelet test is normal; clotting factorfunction may be abnormal

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    INT E RV E NTIONS

    Monitor for bleeding and maintainbleeding precautions

    Prepare to administer replacement factorsas prescribed

    Monitor for joint pain; immobilize the

    affected extremity if joint pain occurs

    Assess neurological status (child is at riskfor intracranial hemorrhage)

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    Control joint bleeding by immobilization,elevation, and the application of ice; inaddition, apply pressure (15 minutes) forsuperficial bleeding

    Instruct parents how to control bleeding

    Instruct the parents on activities to beavoided by the child, emphasizing

    avoidance of contact sports and the needfor protective devices while learning towalk

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    KAWASAKI D ISEASE

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    Is known as mucocutaneous lymph node

    syndrome and is an acute systemicinflammatory illness

    The cause is unknown but may be associatedwith an infection from an organism or toxin

    Cardiac involvement is the most serious

    complication; aneurysms can develop

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    S IGNS AND SYMPTOMS

    Fever

    Conjunctival hyperemia

    Red throat acute stage

    Swollen hands, rash,and enlargement of thecervical lymph nodes

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    Crackling lipsand fissures

    Desquamation of theskin on the tips of thefingers and toes

    subacute stageJoint pain

    Cardiac manifestations

    Thrombocytosis

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    Irritability may last up for up to 2 monthsafter the onset of symptoms

    Peeling of the hands and feet may occur

    Pain in the joints may persist for severalweeks

    Stiffness in the morning, after naps, and incold temperatures may occur

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    INT E RV E NTIONS

    Monitor temperatures frequently (refer if 101F or higher)

    Assess heart sounds, rate, and rhythm

    Assess extremities for edema, redness, anddesquamation

    E xamine eyes for conjunctivitis

    Monitor mucous membranes forinflammation

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    Monitor strict intake and output

    Administer soft foods and liquids that areneither too hot nor too cold

    Weigh the child daily

    Provide passive range of motion exercises tofacilitate joint movement

    Administer ASA as prescribed

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    Administer immune globulin intravenouslyas prescribed to reduce the duration of thefever and the incidence of coronary arterylesions and aneurysms

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    CORONARY ARTERYD ISEASE

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    Narrowing or obstruction of one or morecoronary arteries as a result of atherosclerosis, which is an accumulation of lipid-containing plaque in the arteries

    Causes decreased perfusion of myocardialtissue and inadequate myocardial oxygensupply

    Symptoms occur when the coronary arteryis occluded to the point that inadequateblood supply to the muscle occurs causingischemia

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    Coronary artery narrowing is significant if

    the lumen diameter of the left main arteryis reduced at least 50%, or if any majorbranch is reduced at least 75%

    The goal of treatment is to alteratherosclerotic progression

    Cardiac catheterization provides the mostdefinitive source for diagnosis

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    S IGNS AND SYMPTOMS

    Chest pain

    Palpitations

    Dyspnea

    Syncope

    Cough or hemoptysis

    E xcess fatigue

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    When blood flow is reduced and ischemiaoccurs, ST segment depression, T waveinversion, or both is noted; ST segmentreturns to normal when the blood flowreturns

    With infarction, cell injury results in STsegment elevation, followed by T waveinversion and an abnormal Q wave

    Blood lipid levels may be elevated

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    INT E RV E NTIONS

    Instruct the client regarding the purposeof diagnostic medical and surgicalprocedures and pre procedure and postprocedure expectations

    Assist the client to identify risk factorsthat can be modified

    Assist the client to set goals to promotelifestyle changes to reduce the impact of risk factors

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    Instruct the client regarding a low-calorie,low sodium, low cholesterol, and low fatdiet with an increase in dietary fiber

    Stress to the client that dietary changes

    are maintained for life

    Provide community resources to the clientregarding exercise, smoking cessation,and stress reduction as prescribed

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    S URGICAL PROC E DUR E S

    PTCA to compress the plaque against thewalls of the artery and dilate the vessel

    Laser angioplasty to vaporize the plaque

    Atherectomy to remove the plaque fromartery

    Coronary artery bypass grafting toimprove blood flow to the myocardialtissue at risk for ischemia or infarction

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    ANGINA

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    Chest pain resulting from myocardial

    ischemia caused by inadequatemyocardial blood and oxygen supply

    Caused by an imbalance between oxygensupply and demand

    Causes include obstruction of coronaryblood flow resulting from atherosclerosis,coronary artery spasm, or conditionsincreasing myocardial oxygenconsumption

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    P ATT E RNS OF ANGINA

    Stable Anginay Also called exertional angina

    y

    Occurs with activities that involve exertionor emotional stress; relieved with rest ornitroglycerin

    y

    Usually has a stable pattern of onset,duration, severity and relieving factors

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    U nstable Anginay

    Also called preinfarction angina

    y Occurs with an unpredictable degree of exertion or emotion and increases in

    occurrence, duration, and severity over time

    y Pain may not be relieved with nitroglycerin

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    I ntractable Angina is a chronic,incapacitating angina unresponsive tointerventions

    P reinfarction Anginay

    Associated with acute coronary insufficiency

    y Lasts longer than 15 minutes

    y Symptom of worsening cardiac ischemia

    y Occurs after an MI, when residual ischemiamay cause episodes of angina

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    S IGNS AND SYMPTOMS

    Pain

    Dyspnea

    Pallor

    Sweating

    Palpitations and tachycardia

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    Dizziness and faintness

    Hypertension

    Digestive disturbances

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    INT E RV E NTIONS

    Assess pain

    Provide bed rest

    Administer oxygen at 3L/min by nasalcannula as prescribed

    Administer nitroglycerin as prescribed

    Obtain a 12-lead E CG

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    Provide a continuous cardiac monitoring

    Assist the client in identifying angina-precipitating events

    Instruct client to stop activity and rest if chest pain occurs and to take nitroglycerinas prescribed

    Instruct client to seek medical attention if pain persists

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    Assist client to identify risk factors thatcan be modified

    Provide dietary instructions

    Provide community resources to the clientregarding exercise, smoking cessation,and stress reduction

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    MYOCAR D IAL INFARCTION

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    Occurs when myocardial tissue is abruptlyand severely deprived of oxygen

    Ischemia can lead to necrosis of myocardial tissue if blood flow is notrestored

    Infarction does not occur instantly butevolves over several hours

    Obvious physical changes do not occur inthe heart until 6 hours after theinfarction, when the infarcted areasappears blue and swollen

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    Not all clients experience the classic

    symptoms of an MI

    Women may experience atypicaldiscomfort , shortness of breath, or fatigue

    An older client may experience shortnessof breath, pulmonary edema, dizziness,altered mental status, or a dysrhythmia

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    S IGNS AND SYMPTOMS

    Pain

    Nausea and vomiting

    Diaphoresis

    Dyspnea

    Dysrhythmias

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    Feelings of fear and anxiety

    Pallor

    Cyanosis

    Coolness of extremities

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    INT E RV E NTIONS

    Obtain a description of the chestdiscomfort

    Assess vital signs

    Assess cardiovascular status

    Place client in a semi-Fowlers position

    Administer oxygen at 2 to 4L/min by nasalcannula as prescribed

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    E stablish an IV access route

    Administer nitroglycerin as prescribed

    Administer morphine sulphate as

    prescribed to relieve chest discomfort

    Obtain a 12-lead E CG

    Monitor thrombolytic therapy, which maybe prescribed for the first 6 hours of thecoronary event

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    Administer beta blockers as prescribed

    Assess distal peripheral pulses and skintemperature

    Monitor intake and output

    Assess RR and breath sounds for signs of heartfailure

    Monitor BP closely

    Provide reassurance to the client and family

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    Maintain bed rest for the first 24 to 36hours as prescribed

    Allow the client to stand to void or use a bedside commode if prescribed

    Provide ROM exercises

    E ncourage client to verbalize feelingregarding the MI

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    RAYNAU D S D ISEASE

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    Vasospasms of the arterioles and arteriesof the upper and lower extremities

    Vasospasms cause constriction of thecutaneous vessels

    Attacks are intermittent and occur withexposure to cold or stress

    Affects primarily fingers, toes, ears, andcheeks

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    S IGNS AND SYMPTOMS

    Blanching of the extremity, followed bycyanosis during constriction

    Reddened tissue when the vasospasm isrelieved

    Numbness, tingling, swelling, and a coldtemperature at the affected body part

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    INT E RV E NTIONS

    Monitor pulses

    Administer vasodilators as prescribed

    Assist the client to identify and avoidprecipitating factors such as cold andstress

    Instruct the client to avoid smoking

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    Instruct the client to wear warm clothing,

    socks and gloves in cold weather

    Advise client to avoid injuries to fingersand hands

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    BUERGER S D ISEASE

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    Thromboangiitis obliterans

    An occlusive disease of the median andsmall arteries and veins

    The distal upper and lower limbs areaffected most commonly

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    S IGNS AND SYMPTOMS

    Intermittent claudication

    Ischemic pain occurring in the digits whileat rest

    Aching pain that is more severe at night

    Cool, numb, or tingling sensation

    Diminished distal pulses

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    INT E RV E NTIONS

    Instruct the client to stop smoking

    Monitor pulses

    Instruct the client to avoid injury to theupper and lower extremities

    Administer vasodilators as prescribed


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