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Cvs Inspection

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    INSPECTIONOF

    CARDIOVASCULAR

    SYSTEMDr. Ch.VIJAY

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    Patient must be stripped to waist.

    Examined in good light.

    Examined in both upright and in lying down

    position.

    Examiner should sit or stand directly facing thepatient.

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    The examination includes 2 parts :-

    A) INSPECTION OF NECK

    B ) INSPECTION OF CHEST

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    VENOUS PRESSURE

    ARTERIAL PULSE

    ENGORGEMENT OF LEFT EXTERNAL

    JUGULAR VEIN

    DETECTION OF GOITRE

    WEBBING OF NECK

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    Venous pressure measured >3 cm above the sternal angle is consideredelevated

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    NORMAL JUGULAR VENOUS WAVES

    a wave - atrial systolex descent onset of atrial relaxationc wave - small positive notch in the'x' descent due to bulging of the AVring into the atria in isovolumetricventricular contraction.

    v wave - after the x' descent - slowpositive wave due to right atrial fillingfrom venous returny descent- rapid emptying of the RAinto RV due to TV opening

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    VENOUS PULSATIONS ARTERIAL PULSATIONS

    b/w SCM 2 heads &clavicleSuperficial ,widespreadVisibility>>>palpabilityObliterable

    Multiple pulsationsChanges with -respiration -position -abd.pressureUpper limit visible 2 peaks/heart beat2 descents,rapid

    Medial to SCM

    Deeper,localised

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    CAROTID PULSATIONS Carotid shudder A coarse vibration at the height of carotid

    pulse said to be diagnostic of combined AS+AR. Dancing carotids(Corrigans Sign) Massive pulsation of

    neck carotid arteries observed in AR. These pulsations are

    severe enough to cause visible movement of ears or headwith each beat of heart(Alfred de Mussets sign). Pulsating carotids are usually indicative of wide arterial

    pulse pressure. Kinked carotid artery

    Males suggestive of coarctation of aorta.Females have a small pulsatile oval swelling in persons

    with hypertension, atheroma, and kyphoscoliosis. Prominent pulsation on right side of neck, in hypertension,

    is referred to as Rowntrees sign. contd

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    EPIGASTRIC PULSATIONS

    Epigastric pulsation may be cardiac, aortic, or hepatic

    in origin.

    Character Cardiac pulsations Aortic pulsations Hepatic pulsations

    Relation to apical

    thrust

    Synchronus with

    apex

    Soon after apex Soon after apex

    Thrust orretraction

    More of retraction More of thrust More of thrust

    Location High up inepigastrium

    Low down inepigastrium

    Right of themidline

    Causes MSLt sided pleuraleffusion

    NervousnessTransmittedpulsations byabdominal lumpAneurysm ofabdominal aorta

    Enlarged pulsatingliver in TR or TS

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    DISTENDED SUBCUTANEOUS ARTERIES :-

    Dilated and tortuos superficial arteries under the skin

    of the chest and back are a characteristic feature ofcoarctation of the aorta.

    Suzmans sign?

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    Size, shape and type of chest

    Shape of the precordium

    Apical thrust

    Other pulsations of the precordium

    Other pulsations of the chest wall

    Suprasternal or episternal pulsation

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    They have a direct bearing on presence of atypical orabnormal physical signs in the chest as in funnelchest, rachitic chest, and scoliosis, or the straight backsyndrome.

    It is also responsible for diseasedcondition of the heart, as inemphysema and severe

    kyphoscoliosis.

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    Bulging or retraction of the precordium may be dueto diseases outside the heart; they should be ruledout first, before implicating heart as the cause.

    PRECORDIAL BULGING :- A good sign forrecognizing bulge in male is lateral displacementand elevation of left nipple in comparison withright.

    Causes :

    1) Skeletal deformities2) Diseases of lung and pleura

    3) Diseases of heart or precordium

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    BACKWARD BULGE :-

    1) Pectus excavatum

    2) Shield chest

    PRECORDIAL FLATTENING :-

    1) Old pericarditis or adherent pericardium

    2)

    Fibrosis or collapse of lung3) Scoliosis or kyphoscoliosis

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    APEX BEAT v/s PMI :

    The term PMI is often used as a synonym for an apex beat. It actually meanspoint of maximum impulse i.e. the site of the

    loudest murmur.

    The maximal precordial pulsations

    may be due to

    Dilated pulmonary artery

    Large RV

    Ventricular aneurysm

    Aortic aneurysm

    Hence THE DEFINITION OF APEX BEAT is thelowermost and outermost point of definite cardiacimpulse, which can be appreciated.

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    NORMAL VARIATIONS :-

    A. Infancy or childhood, apex is in 4th intercostal space.

    B. In thin, narrow chested, and elderly subjects, seen in6th intercostal space.

    C. In obesity, abdominal distension and during

    pregnancy, it may be displaced slightly outward andupward by the raised diaphragm.

    D. Apical thrust is normally invisible in few persons dueto

    1) Heart being situated behind a rib2) Thick chest wall

    3) Pendulous breast

    4) Emphysematous chest

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    POSTURAL SHIFT :-

    A. Mere shifting in bed from left lateral to the rightlateral position may shift the apex as much as 11/2 to2 inches.

    B. A change from recumbent to the upright position oreven taking a deep breath may alter the position ofthe thrust.

    C. Failure of apical thrust to shift in this manner(with

    change in posture or on inspiration) is a sign ofadherent pericardium.

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    APICAL THRUST MUST BE OBSERVED FOR :

    Presence or absence

    Location, whether normal or displaced

    Extent, whether localized or diffuse

    Direction of movement during systole, whetheroutward or inward (thrust or retraction)

    Lack of mobility or fixation Other characteristics

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    Displacement of apical thrust:- Due toA) Extrinsic or Extra cardiac causes

    I) Extra thoracic scoliosis, straight back syndromeII) Intra thoracic Displaced

    i. sideways pleural effusion, pneumothoraxii. downwards aortic aneurysm, mediastinal

    new growthIII) Intra abdominal ascites, meteorism, massive

    abdominal tumour or advanced pregnancy.B) Intrinsic or cardiac causes

    I) Congenital dextrocardiaII)Acquired hypertension, aortic and mitral valve

    disease

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    Extent of apical thrust:- Diffuseness of thrust is

    seen inA) Thin chest wallB) Hyperdynamic heart conditionsC) Severe valvular regurgitationD) Left to right shuntsE) Complete AV blockF) Hypertrophic obstructive cardiomyopathyG) Retraction of lung from fibrosis or collapse

    A double systolic outward thrust is characteristic ofHOCM. It is also seen in mitral valve prolapse and LV

    dyskinesia as in acute MI.

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    Force of apex thrust:- force is visibly increased inA) Thin chest wallB) Retracted lungC) Hyperdynamic heartD) LV hypertrophy as in hypertension or AR

    Cardiac causes of invisible apex :-A) Weak action of heart as in MI or acute myocarditisB) Pericardial effusionC) Dilation of heartD) Dextrocardia

    Skodas sign :- (Negative cardiac impulse)It is sucking in or retraction during systole of the apical region. It may be due

    toA) Hyperdynamic heart with apex situated behind a ribB) Hypertrophied right ventricle, with forward thrust in the midprecordial

    area and retraction of apexC) Adhesive pericarditis, a diagnosis justified only when retraction involves

    both ribs and interspaces (BROADBENTS SIGN)

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    Best observed by tangential inspection of precordial area, preferablywith patient recumbent with lowering his eyes level to anteriorwall of patients chest.

    Physiological diffuse pulsation (wavy or peristaltic cardiacimpulse) : Seen in

    Thin chest individuals Hyperdynamic hearts During fever or after exercise Retracted lungs

    Physiological para-apical retraction : A systolic retraction ofchest wall between the apical region and sternum, due to thesucking in effect of RV systole

    It may be mistaken for abnormal apex.It is however situated medial to true apex and is a retraction rather

    than a outward movement. Contd

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    Left parasternal pulsation : A systolic heaving of the mid precordial area, maximal

    between 3rd and 6th ribs is characteristic of massive RV

    hypertrophy. A central lift may be due to systolic expansion of left

    atrium from mitral regurgitation. A heave in the left parasternal region may be due to LV

    hypertrophy.

    Rocking or see saw movements : seen in massivehypertrophy of right or left ventricle.

    In RV hypertrophy, an inward movement of the apex is

    associated with an outward movement of the midprecordium

    during systole

    In LV hypertrophy, the phenomenon is reversed

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    Diffuse systolic retraction : A diffuse retraction of

    precordial area, involving ribs and interspaces is due tothe

    Tricuspid regurgitation

    Adhesive pricarditis

    Aortic regurgitation

    Lateral retraction in lateral decubitus position due to Large RV or severe TR (with initial outward

    movement)

    Constrictive pericarditis: systolic retraction (with initialoutward movement), followed by diastolic thrust

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    High thoracic pulsations : Observed in 2nd rightintercostal space or behind the upper part of thesternum indicative of

    Aneurysm of the ascending or transverse part of aortic

    arch Dilatation of the aorta

    AR

    Pulsations involving the 2nd or 3rd left interspace due to :

    Dilatation of pulmonary artery as in PDA or septaldefects,MS or aneurysmal dilatation of P.artery.

    Retraction of left lung from fibrosis or collapse.

    Aneurysm of descending thoracic aorta

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    Pulsations of sternoclavicular joint: Due to Right side suggests right sided aortic arch

    Either side occurs in aortic dissection or aneurysm

    Systolic outward pulsation of upper half of sternum is dueto aneurysm of ascending aorta

    Pulsation to right of sternum is due to dilated and unfolded

    ascending thoracic aorta and rarely due to large right atrium

    Pulsations in atypical situations

    CAUSE LOCATION

    Empyema necessitates Pulsatile swelling in lateral aspect of chest wallLymphosarcoma Highly vascular tumour in mid sternum

    Descending thoracic aorta aneurysm Back

    Innominate artery aneurysm Supraclavicular region or upper part of thorax

    Coactation of aorta Interscapular and intercostal regions

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    It may be seen in

    Hyperdynamic heart.

    Anemia.

    Aneurysm of aorta. Dilatation of aorta as in atheroma or syphilitic

    aortitis.

    Raised or uncoiled aorta as in hypertension.

    Elongation and flexion of the innominate artery.

    Anomalous right subclavian artery.

    Thyroidea ima artery.

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