ECG IN MISCELLANEOUS CONDITIONS
PRESENTED BY: Dr. Areej Al-jabaly
A: Drugs Effects
B: ELECTROLYTE
C : DISEASES
D: NORMAL VARIANTS
A: DRUGS EFFECTS Digoxin :
Therapeutic Effect *ST segment depression
( reversed tick )
* Shortening of the QT interval T wave inversion
Toxic Effect :
Any type of arrhythmia especiallventricular octopi
Quinidine: and related drugs like ( procinamide , Disopyramide , phenothiazine, Tricyclic, Antidepressant, Amiodarone )
* P wave widening * QRS widening
* Prolonged QT interval ( longer than half of the RR interval)
•
* Increase U wav amplitude *ST segment depression
* Increase U wav amplitude
B: ELECTROLYTE:
Hyperkalemia:
1 -Mild to moderate hyperkalemia (5 -7 mEq/L )
)* Tall symmetrical peaked ( tents T waves with narrow bas.
2 -More severe hyperkalemia (8 - 11 mEq/L )
* widening of QRS * PR interval prolonged
3 -Severe case > 11 * ECG resemble a sine wave * P wave disappearance
(atrial arrest)
Hypokalemia :
Mild( 3-3.5) to moderate ( 2.5 – 3) mEq /L
* Progressive ST segment depression
* Progressive decrease in T wave amplitude
* increase U wave amplitude
)Severe (< 2.5 mEq /L * Fusion of T and U wave
* Increase QRS duration and amplitude
* Increase P wave duration and amplitude
* QT interval usually slightly prolonged
Hypercalcaemia:
Marked shortening of the QT interval due to shortening of the ST segment
Hypocalcaemia:
Prolong the ST segment without affecting the T wave
C : DISEASES Renal failure :
Triad of *LVH (HTN)
* Peaked T wave (Hyperkalemia)
*Prolong of the QT interval (Hypocalcemia)
Pericardial Effusion: Triad of
* Low voltage QRS complexes (0.5mv or less)
* low to inverted T waves in most leads
* Total electrical alternans
:Thyroid disease
A: Hypothyroidism: * Low voltage ECG * Sinus bradycardia
* Inverted T waves without ST segment deviation in many or all leads ( slow and low ECG )
B:Thyrotoxicosis:
*Unexplained AF ( sinus tachycardia at rest)
* High voltage ECG * Decrease of QT interval
* Prominent U wave in association with tachycardia
Acute Pericarditis: * Diffuse ,Upward concave ST
elevation * PR depression (specific but
less sensitive ) *Almost associated with sinus
tachycardia
Acute Myocarditis :
* Non specific T wave change. * Depression or elevation of
ST segments . * Prolonged QT interval.
: CVA* Abnormal & widened T waves
that may be deeply inverted or tall & peaked.
* Prominent U waves.* Prolonged QT interval.
These changes are termed CVA pattern & usually resolved with
time .
:COPD * RAD
* Absent R wave in precordial leads.
* Prominent R wave in Rt precordial leads & ST segment depression when
there is RVH
* Prominent P wave in inferior leads (P pulmonale ) resulting
from Rt atrial abnormality .* Occasionally SI , SII , SIII
syndrome. * Rarely in 10 % of patients LAD
:Pulmonary embolism * Sinus tachycardia.
* Rt ventricular strain , appearance of ST-T changes
in VI ,VII. * SI QIII TIII more specific but
less sensitive ( due to acute Rt ventricular dilatation )
* ST depression. * Acute RBBB ( rSR' in VI)
result from Rt ventricular dilatation
:Amyloidosis * Low voltage of all wave in limb
leads * Marked LAD
* QS or minimal R wave in V1- V3 or V4
D: NORMAL VARIANTS
: Early repolarization syndrom * ST elevation:
1 -may raised to 2 mm above the baseline.
2 -It always follow the S wave .
* Tall R & ST-T change in the Lt
precordial leads. * Relatively tall & frequently
symmetrical T wave , rarely T wave inversion.
* No reciprocal changes except ST segment depression in
aVR.
Hypothermia : * J wave or ( Osborn wave ) it is
localized to the junction of the end of QRS complex and beginning of the end of ST segment
* Prolongation of QRS complexes
* Depression of ST segment* T wave depression
* Prolongation of QT interval* Sinus bradycardia
* First and second - degree heart block
* Ectopic rhythm
Obesity: * Displacement of heart by
elevated diaphragm to the left but within normal range QRS axis
* Increasing the distance between the heart and the recording electrodes although the true low voltage QRS amplitude is rarely appears
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