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HEART BLOCK
PRESENTER--- Dr Sibadatta Das
MODERATER----Dr Indu Khurana
INTRODUCTION
A "heart block" is a disease in the electrical system of the heart
.Heart Block is a type of bradycardia (too-slow heartbeat) that also is called atrioventricular, or AV block.
Cunducting system of heart
S.A.NODE
A.V. NODE
BUNDLE OF HIS
PURKINJE FIBRES
INTRA ATRIALTRACT OFBACHMAN
Intranodaltracts
MEASUREMENT
Done mainly by 2 way 1 ECG (Non interventional) 2 his bundle electrogram (interventional)
ELECTRO CARDIO GRAM
Extra corporial measurement of summed electrical activity of intigrated myocardial tissue
Interpreted as different waves like P Q R, S ,T etc.
Atrial depolarization
Ventricular depolarization
Ventricular repolarization
HIS BUNDLE ELECTROGRAM Recording of electrical activity
of heart by intra cardiac ring electrode placed near the tricuspid valve
From comparative studyWith ECG accurate site of block
can be calculated
Activation of AV node
Transmission Of impulseThroughHis bundle
Ventriculardepolarization
Comparative study
This is done by calculating 3 intervals
PA Int:-conduction from SA node to AV node normal— (27 ms)
AH Int:-AV nodal conduction time ( 92 ms)
HV Int:-bundle conduction ( 43 ms)
DISEASE TARGETS
SA NODE DISTURBANCES
Occurs mainly in 2 ways 1. sinus failure (failure of
impulse generation)
2. sinus exit block (failure of conductance of impulse to atrium)
manifestation
Mostly presented in 2 way 1.SICK SINUS SYNDROM mostly found in old patients Presented as sudden dizziness , fatigue , &
syncope ECG: persistent bradycardia i.e recognised by a pause which is multiple of
PP interval or progressive shortening of PPinterval followed by a pause
TACHY-BRADY syndrome
•reentry of ventricular stimulation causing
• atrial tachycardia followed by a pause.
•Then started with a slower rate .
•Again &again the cycle is repeated.
investigation
24 hr holter monitering is the chief mode. Interpretation shd be done cautiously as
hyper vagotonia in athelates & sleep sinus pause causes the same pattern
Accurate detection occurs throiugh sinus node electrogram and calculating the sinus recovery time indirectly
TREATMENT
Most of the patients are symptom free Symptomatic patients are difficult to treat Oral theophylin is some how effective But most of the patients require pace maker Permanent pace maker is the T/t of choice in
recurrent symptomatic Pt.s Dual chamber pace maker is preferred as SA
node dysfunction is usually associated with AV node dysfunction.
Affected AV Node
This is mainly affected by 3 ways
1. First degree AV block.
2. Second degree AV block.
3. Third degree AV block.
First degree AV block
All the impulses from atria reaches ventricle but delay occurs in conduction.
Etiology is not specified ECG is almost normal
except prolongation of PR interval hence it is sited as diagnostic criteria
PR interval > 0.2s is almost diagnostic
second degree AV block(intermittent AV block) Not all impulses arising from atria reach
ventricle 2 types
. Mobitz I
. Mobitz II
Mobitz I(Wenckebach AV block) PR interval gradually prolongs
till 1 P wave is dropped. After dropped P wave the PR
interval is shorter than the last long PR interval.
The difference between longest & shortest PR interval > 100 ms.
ETIOLOGY : inferior wall infarction drug intoxications like digitalis,
β blockers, Ca channel blockers etc.
Mobitz II There is sudden stoppage of
conduction without previous change in PR interval.
So, 2 to 3 consecutive P wave comes following 1 QRS complex accordingly it is named as 2:1 or 3:1 block.
ETIOLOGY : Anteroseptal infarction calcific disease of fibrous
skeleton of heart Typically it has a tendency to
proceed towards complete heart block
THIRD DEGREE HEART BLOCK
No atrial impulse conducted into ventricle
QRS complex comes alone in ECG
P wave has no relation with the QRS complex
Rate is as per AV node rhythm I;e
40-45/min with normal QRS complex
Or as per HIS-PURKINJE rhythm i.e 15-40 /min with wide QRS complex.
Bundle branch block
Etiology – long standing HT severe valvular disease cardio myopathy
Typical manifestations found in ECG is opposite direction of T wave to that of QRS complex due to reverse direction of repolarization
2 types
RIGHT BUNDLE-BRANCH BLOCK
LEFT BUNDLE-BRANCH BLOCK
RIGHT BUNDLE-BRANCH BLOCK
•Electrical impulse cannot travel
through it to the right ventricle
• Activation reaches the right •ventricle by proceeding from the• left ventricle.
•It then travels through the septal
and right ventricular muscle mass •This progress is, of course, slower •leads to a QRS-complex wider than
0.1 s •QRS >0.12s is diagnostic
RBBB causes an abnormal terminal QRS-vector that is directed to the right ventricle (i.e., rightward and anterior). This is seen in the ECG as a broad terminal S-wave in lead I.
Another typical manifestation is seen in lead V1 as a double R-wave. This is named an RSR'-complex
LEFT BUNDLE-BRANCH BLOCK
The situation in left bundle-branch block (LBBB) is similar, but activation proceeds in a direction opposite to RBBB.
Again the duration criterion for complete block is QRS > 0.12s
.
Because the activation wave front travels in more or less the normal direction.
However, because of the abnormal sites of initiation of the left ventricular activation front and the presence of normal right ventricularactivation the outcome is complex seen as a broad and tall R-wave, usually in leads I, aVL, V5, or V6.
OTHER COMBINATIONS OF BLOCK BIFASCICULAR BLOCK
RBB + Lt. Ant. Fascicle block
or
Rt. Ant. Fascicle block TRIFASCICULAR BLOCK
Bi fascicular block + PR prolongation etc.
CONCLUSION
Still an enigma for medical science Highly unpredictable in character Through out monitoring is essential Non interventional T/t option is rare is present day
scenario. Hope is there for something better in future days.
REFERENCES
Text book of physiology 1st- Indu khurana Review physiology 22nd-Ganong Physiology 13th- Samson wright Internal medicine16th- Harrison Clinical diagnosis of ECG- K.P. Mishra