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Interpretation of Normal ECG *medical

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    ECG or EKG:

    Interpretation of normalECG

    Dr. Kyaw Min

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    To get differentperspectives ofthe heart

    http://www.photokina-show.com/0458/nikonians/events/nikoniansnudeshoot/http://www.photokina-show.com/0458/nikonians/events/nikoniansnudeshoot/http://www.photokina-show.com/0458/nikonians/events/nikoniansnudeshoot/http://www.photokina-show.com/0458/nikonians/events/nikoniansnudeshoot/http://www.photokina-show.com/0458/nikonians/events/nikoniansnudeshoot/
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    Electrocardiograph

    electrodes, electrode cables

    amplifiers filters,

    control modules,

    paper recordingmodule

    special graph paper

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    V1-V2 anteroseptal wall II, III, aVF inferior wall

    V3-V4 anterior wall I, aVL lateral wall

    V5-V6 anterolateral wall V1-V2posterior wall(reciprocal)

    Electrocardiograp

    hic view of theheart

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    A normal adult 12-lead ECG. Sinus rhythm is present with a heart rate of 75

    beats per minute.

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    Waves

    Positive

    Negative

    Segments

    Intervals

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    Paper speed: 25 mm/secVoltage

    5 mm

    = 0.2s

    5 mm= 0.5mV

    Voltagecalibration:

    2 largesquares (10mm)

    = 1 mV

    1 large square = 0.2 s

    300 large squares = 300 x 0.2 s = 60s(1 min).

    http://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svghttp://en.wikipedia.org/wiki/Image:ECG_Paper_v2.svg
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    vertical

    horizontal

    Each large(5mm)square : 0.5mV0.2s

    Each small(1mm)square : 0.1mV0.04s

    t

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    aves, nterva s ansegments

    in normal ECG

    http://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svg
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    wave0.08 to 0.1 s; 0.25 mV or less)

    represents the wave ofdepolarization that spreadsfrom SA node throughoutthe atria.

    upright in all leads; butinverted in aVR.

    atrial repolarization isubmerged in QRS complex)

    http://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svg
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    QRS complex(0.06 to0.1 s)

    (1- 3 small squares)-represents ventriculardepolarization.

    - In most people, QRS

    complex is tallest in leadII, but in leads I and III, itis also predominantlyupright (i.e. R wave is

    greater than S).

    -Prolonged QRS complex(> 0.1s) in Bundle

    branch block

    http://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svg
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    Q waveSmall septal Q wave in

    any of leads II, aVL, or V5 V6, usually less than 3 mmdeep and less than 1 mmacross.

    -represents the normaldepolarization ofinterventricular septumfrom left to right.

    -A small Q wave is also

    common in lead III innormal eo le in which

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    T wave-represents ventricular

    repolarization- In a normal ECG, Twave is alwaysinverted in aVR, and

    often in V1, butusually upright in allthe other leads.

    http://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svg
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    U waveSometimes a small

    U wave may beseen following Twave (lastremnants of

    ventricularrepolarization andslow repolarizationof papillarymuscles)

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    -R intervalormal range is 0.12 to 0.2 s

    3 5 small squares)

    the period of timerom the onset of theP wave to the beginning

    f the QRS complex.represents the timeaken for depolarizationf atria, and conduction

    through AV node andis-Purkinje system.

    long PR interval reflects slow conduction

    eart block, bradyarrhythmia)

    http://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svg
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    -T interval (0.2 to 0.4 s)represents the time for

    both ventriculardepolarization andrepolarization.

    At high heart rates,Q-T interval decreases.

    Long QT syndrome(K+ channel mutation,yocardial ischemia

    lectrolyte abnormalities)

    http://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svg
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    T segmentthe part between S wave and

    T wave.is the time at which theentire ventricle is depolarized.

    hould be horizontal andisoelectric.

    oughly corresponds to

    heplateau phase ofentricular depolarization

    -Important for diagnosis of ventricular

    ischemia (depressed or elevated)

    http://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svg
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    segmentn elevation of the ST segment

    the hallmark of an acuteyocardial infarction.orizontal ST segment depressionmore than 2mm indicates

    chaemia.

    http://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svg
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    Cardiac axis

    Depolarization wave of theheart normally spreads through

    the ventricles from 11 oclock

    to 5 oclock,

    i.e. towards leads I, II and III.

    So the deflections in lead VR

    are normally mainly downward

    (negative) and lead II mainly

    upward (positive).

    Direction of the axis can bederived most easily from the

    QRS complex in leads

    I, II and III.

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    The cardiac axis

    By near-consensus, the normal QRS

    axis is defined as ranging from -30

    to +90.

    -30 to -90 is referred to as a left axis

    deviation (LAD)+90 to +180 is referred to as a right

    axis deviation (RAD)

    Abnormalities of axis can hint at:Ventricular enlargement and/or

    conduction defects (i.e. hemiblocks)

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    If right ventricle becomes hypertrophied, the axiswill swing

    towards the right : the deflection in lead I becomesnegative and

    Right axis deviation

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    When left ventricle becomes hypertrophied, the axismay swing

    to the left, so that the QRS complex becomesredominantl

    Left axis deviation

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    Uses of ECG

    Recording of rate and rhythm diagnosis of cardiac arrhythmias

    detection of conduction

    abnormalities (e.g. heart block,accelerated conduction)

    screening tool for ischaemic heart

    disease (during a cardiac stresstest)

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    Uses of ECG

    It guides therapy and riskstratification for patients withsuspected acute myocardial

    infarction

    It helps detect electrolytedisturbances (e.g.

    hyperkalaemia andhypokalaemia)

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    RR interval = one ventricular cycle Ventricularate heart rate

    PP interval = one atrial cycle atrial rate

    http://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svghttp://en.wikipedia.org/wiki/Image:SinusRhythmLabels.svg
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    Heart Rate Determination Method 1

    Most rates can be calculated this way. Find an Rwave on a heavy line (large box) count off

    "300, 150, 100, 75, 60, 50"for each large box you land on until you reach the

    next R wave. Estimate

    the rate if the second R wave doesn't fall on aheavy black line.

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    Heart Rate Determination Method 2

    Use this method if there is a regularbradycardia,

    i.e. - rate < 50.

    If the distance between the two R waves is

    too long to use the common method 300/[# large boxes between two R

    waves].

    The number of large boxes between first and second Rwaves = 7.5. Thus 300/7.5 large boxes = rate 40.

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    Heart Rate = ?

    Heart Rate = 300/5 = 60/min

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    rate 100 = sinus tachycardia

    http://www.ecglibrary.com/sbrady.htmlhttp://www.ecglibrary.com/stach.htmlhttp://www.ecglibrary.com/stach.htmlhttp://www.ecglibrary.com/sbrady.html
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    Diagnosis of cardiac arrhythmias

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    A normal 12-lead ECG and rhythm strip (Longlead II).Heart Rate = 300/4 = 75 bpm

    Rhythm :

    regular,

    Sinus rhythm

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    Cardinal features of Sinus rhythm

    The P wave is upright in lead II,inverted in aVR

    Each P wave is usually followed by aQRS complex

    The heart rate is 60 99 beats/min

    rate 100 = sinus tachycardia

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    Abnormalities of cardiacrhythm

    Look at the P waves and the widthof the QRS complex

    Supraventricular rhythms havenarrow QRS complexes

    Ventricular rhythms have wideQRS complexes (due to slower

    pathway through the Purkinjefibers)

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    Rate: 150 -250 /m

    Rate: 250 -350 /m

    Rate: 350+ /m

    Abnormally shaped P wave

    Arrhythmias

    (saw-tooth waves)

    (Abnormal P waves,one P wave/QRS)

    Atrial fibrillation with a totally

    irregular ventricular rate.

    Atrial rate 300-500/min.

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    Supraventricular rhythms Atrial tachycardia:

    QRS complex rate greater than150/min;

    Abnormal P waves, usually with short

    PR intervals, usually one P wave perQRS complex, but sometimes P waverate 200-240/min with 2:1 block

    Rate: 150 -250 /m

    (Abnormal P waves,one P wave/QRS)

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    Supraventricular rhythms

    Atrial flutter: P wave rate 300/min,

    saw-toothed pattern, 2:1, 3:1 or 4:1block

    Rate: 250 -350 /m

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    Supraventricular rhythms Atrial fibrillation:

    the most irregular rhythm of all,

    QRS complex rate characteristicallyover 160/min without treatment, butcan be slower

    no P waves identifiable, but there is avarying completely irregular wavy

    baseline

    r

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    Ventricularpremature beat(extrasystole)

    Ventricular

    tachycardia

    (fast rate, no P wave,

    wide bizarre QRS)

    r

    Arrhythmi

    as

    Ventricularfibrillation

    (erratic, wavy

    Defibrillator

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    Ventricular extrasystoles:

    Early QRS complex;

    No P wave,

    QRS complex wide (greater than 120ms);abnormally shaped; followed by a

    compensatory pause Abnormally shaped T wave,

    Next P wave is on time

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    Ventricular tachycardia:

    No P waves;

    QRS complex rate greater than160/min; accelerated idioventricularrhythm

    Wide bizarrely shaped QRS complex

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    Ventricular fibrillation:

    The most frequent cause of sudden

    death in patients with myocardialinfarction

    In the absence of emergency

    treatment, lasts a few minutes; fatal Look at the patient, not the ECG

    Ventricularfibrillation

    (erratic, wavybaseline)

    Defibrillator

    C di l di i i

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    CardiacPhysiology

    Electrocardiography

    Diagnosis

    Ventricular Fibrillation

    Ischemia

    Electric Shock

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    Detection of conductionabnormalities (e.g. heart block,accelerated conduction)

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    First degree block:One P wave per QRS complexPR interval greater than .2 s

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    Mobitz Type 2:(2:1 or 3:1 block)Occasional non-conducted beatsTwo or three P waves per QRS complex

    Normal P wave rate,PR intervals are constantQRS is dropped intermittantly

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    Progressive PR lengthening thennon-conducted P wave,And then repetition of the cycle

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    Bundle Branch Block

    If there is abnormal conductionthrough either the left or right bundlebranches (bundle branch block),

    there will be a delay in thedepolarization of part of theventricular muscle

    The extra time taken fordepolarization of the whole of theventricular muscle causes widening

    of the QRS complex

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    ird degree block (complete block):relationship between P waves and QRS complexes

    wave and QRS march out separatelyually, wide QRS complexes

    ual QRS complex rate less than 50/minmetimes narrow QRS complexes, rate 50-60/min

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    Screening tool for ischaemic heartdisease (during a cardiac stresstest)

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    Exercise or stress ECG

    L d

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    3 mm (0.3 mV) of horizontal ST-segment depression, indicating apositive test for ischemia.

    After 4 min ofexercise

    At rest

    LeadV4:

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    It guides therapy and riskstratification for patients withsuspected acute myocardialinfarction

    Anterior Q wave infarction

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    Deep Q waves, ST segment elevation. Later T inversion reciprocal ST depressions in leads III, and aVF.

    Anterior Q wave infarction

    Inferior Q wave infarction

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    may be associated with reciprocal ST depressions in leadsV1 to V3.

    Inferior Q wave infarction

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    Myocardial Infarction

    Sequence of ECG changes

    Normal ECG

    Raised ST segments

    Appearance of Q waves

    Normalization of ST segments

    Inversion of T waves

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    Ischemia T inversion

    Injury ST segment elevation Infarct pathologic Q wave

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    It helps detect electrolyte

    disturbances (e.g. hyperkalaemiaand hypokalaemia)

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    (Very tall, slender peaked T wave)

    (T inversion, prominent U wave)

    Electrolyte abnormalities

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    Electrolyte abnormalities

    Low K+

    : T wave flattening and theappearance of a hump on the end ofthe T wave called a U wave.

    High K+: tall, wide, peaked T waveswith the disappearance of the STsegment. The QRS complex may bewidened.

    (Effects of abnormal magnesium levelsare similar.)

    Low Ca2+: prolongation of the QTinterval

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    How to report an ECG

    Rate

    Rhythm

    Conduction intervals

    Cardiac axis

    A description of QRS complexes

    A description of the ST segments andT wave

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    DescriptionHeart rate 110/min,

    Sinus rhythm

    Normal PR interval (140 ms)

    Normal QRS duration (120 ms)

    Normal cardiac axis

    Normal QRS complexes

    Nor T waves

    InterpretationNormal ECG

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