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PMHFAV OF lAPORATORIE? °'" *"*****SPECIAL ANALYSES REPORT

fACHIIY

fMOCHAM COLINAMC IYPE IR

LAIIIUOE4 10 lONCHUOCII W OAK 19 74 TlMEK

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BUREAU OF LABORATORIES D.n t«.l..J_____ > jcffiSPECIAL ANALYSES REPORT

tfclAUIISHMISI CASl w;:CCKJNIV PROGRAM COLL NAM* tYPf 1M S1OANALV&

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LL CARDS) 4-16 LATITUDE 4-10 LONGmjOE II-W ' DATE t*-24EH 1 ClM I Fit j W 1 0 I Vi 1 i 1 i i 1 1 i Oi i ._ i i sr/pl'Src;

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COUNTY MUNICIPALITY* PROGRAM COLL NAME/PHONE NUMBER

CARD (31 IO CODE (ALL CARDS) 4-tB LATITUDE 4- to LONGITUDE H-1> GATE 19-24, ./* Cnty | Mur. T EH Ca» Fie. 1 L * \ 0 r

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HEUOTVE POINT U

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, J.— - SPECIALTABUSMMENT . ; CASE

COUNTY MUMCIPAUTY PROGRAM

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COLL NAME/PHONE NUMBER __ TYPE tH STO ANALYSIS

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tt

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Cr*» Ilk* IT I E* I CtW j Ft* • I

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frtflMJ d/TTshmQUANTITATIVE RESULTS

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f^l1-131 y COMMONWEALTH OF PENNSYLDEPARTMENT OF ENVIRONMENTAL R

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ESTABLISHMENT CASK rVCJUrv

OAOe-r L(C, ir «w "r viar»c*<- «33 R CAX. UcS GemftaciWi. <31OCOUNTY MUNIQPAUTY PROGRAM COLL NAME/PHONE NUMBER

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COMMONWEALTH OF PENNSYLVANIA ———DEPARTMENT OF ENVIRONMENTAL RESOURCES


CASE 1 FACILITY

PROGRAM COLL KAMC/PHONf NUMBER

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409R02

PRELIMINARY INVESTIGATIONOF VOLATILE ORGANIC CHEMICAL CONTAMINATION

OF GROONDWATER NEAR TOE WESTINGHOUSE ELECTRIC CORPORATION 'ELEVATOR COMPONENTS PLANT - GETTYSBURG, PA ^ - // N

Hatch 23, 1983

Executive Summary

Hi oh levels of volatile organic compounds, includingtrichloroethylene and 1,1,1-trichloroethane have been detectedin certaisi domestic water supply wells adjacent to theWestinghouse Electric Corporation Elevator Components Plant inGettysburg, Pa. As a user of organic solvents in theirmanufacturing processes, Westinghcuse was inunediately consideredto be the source of the contamination by many nearby residents,as well as by state and federal environmental regulatoryagencies. In response to these allegations, Westinghouseretained R. E. Wright Associates, Inc. (REWAI) to conduct apreliminary hydrogeologic investigation, capable ofdetermining if the plant is the source of the contamination, orif it originated from off site.

Overview of REWAI Plan of Study

REWAI's investigation broadly consisted of four main tasks:t

A. Review all available data relative to site geology and•||yxlr ogeology.

B. "Drill and construct four on-site groundwatermonitoring wells.

. C. Sample and analyze selected on-site soil and water• samples.

409R02 2

D. Interpret all findings and develop conclusionsconcerning contaminant source. ' , j

Tasks A and B abov-3 were designed to allow definition of thethree-dimensional geologic and hydrogeologic environment, andthus determine parameters controlling contaminant migration.Task C was designed to determine the magnitude and extent ofcontaminants in on-site soils, groundwater, and drainagesystems, and Task D provided for a determination of the mostlikely contaminant source, based upon the data generated fromTasks A through D.

Results of RSWAI Investigations

*•

Site Geology - The plant is located on a poorly bedded,fine-grained red shale known as the Gettysburg Formation. Thestrata within this rock unit have been tilted downward to thewest at approximately 24 degrees below horizontal, so that theorientation of their intersection with ground surface is at an ^-ângle approximately 20 degrees east of north. The rocks arethus said to strike N 20 E and dip 24 degrees west. West of theplant, and roughly parallel to the Reading Railroad Line, adiabase dike has intruded vertically into the shale and forms aphysical barrier, hydrologically isolating bedrock on eithersida of it.

The bedropk,. surface lies buried beneath an average five feet ofsoil at tffff plant site; however, a contour map of the bedrock

**j'-.Vsurface could be made using information provided by soils andfoundation borings completed prior to construction of theWestinghouse complex. Contour mapping revealed an unevenbedrock surface which generally slopes from northwest tosoutheast toward Route 34. Two features of interest found inthe bedrock surface were a trough running from southwest to

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409R02

northeast beneath the southern end of the plant and a depressionlocated approximately 500 feet behind 'the north end of theplant.

S i t e Hy d r o_g e o 1 o ay - Two distinct groundwater flow systemsare present at the plant site. These are a shallow flow systemwhich exists in the soil and generally follows the bedrockinterface and a deep groundwater flow system which exists withinthe bedrock. It is important to note that while these are twodistinct systems, they are not separate, and thus inter-communicate. Flow directions in both systems are generally fromnorthwest to southeast.

Because of the fine-grained nature of the shale bedrock,groundwater does not flow through it in a homogeneous andisotropic fashion, but rather tends to migrate alongdiscontinuities in the rock mass such as bedding planes and

I / fracture zones. Primary movement is along the fracture zones.

Sampling for Volatile Organic Compounds - A total of 18water or soil samples were collected at various locations onWestinghouse property, including the four*new wells drilled as apart of this investigation, the existing Westinghouse well, anda spring located between the main plant entrance and Route 34.All samples were analyzed by standard purge and trap gaschromatography for the following compounds:

chloride*

dichloroethylene1,1 dichloroethane1,2 dichloroethane1,1,1-trichloroethanetrichloroethylenetetrachloroethylene

SoniP 305392

409R02 4

Summaries o£ the chemical analysis are attached as Tables I and

"•

CQnclu3j.Qrig of P.SWAT. Investi,gahion

Significant contamination of soils and groundwater has beendocumented on-site and is primarily associated withtrichloroethylene and 1,1,1-trichloroethane, both of which havebeen or are currently being used on-site. Because theseorganic compounds are heavier than water, they will tend togravitate and migrate along the bedrock-soil interface and theninto the secondary openings in the bedrock. While thecontaminant migration is influenced by the direction ofgroundwater flow, it is not always coincident with it. However,the slope of the bedrock surface and concentrations ofcontaminants on site and along fractures zones result in theconclusion that Westinghouse is the primary source of volatileorganic chemical contamination of the groundwater in thevicinity of the Gettysburg Plant.

Recommepdatipns

Recommended action to be taken by Westinghouse is divided intotwo categories: Emergency Action and Long-Term Action.Emergency Action recommendations are provided as a means fordealing with public concerns over consumption of contaminateddrinking water, enhancing tha public image of Westinghouse in theeyes of tlf local residents and the regulatory agencies, limitingthe spr^js&rof contaminants, and fully understanding therequirementa for assessment and abatement that will be requiredby the regulatory agencies. Long-Term Actions are provided as anoverview of what most likely will be required to complete a

88305393

409R02 . 5

Groundwater Quality Assessment and Abatement Program as requiredby RCRA.

Emergency Actione>

A. Provide drinking water to all affected or potentiallyaffected residents along Route 34 up to theintersection of Boyd School Road and east and west ofRoute 3 along Boyd School Road. On a temporarybasis, this could be accomplished by providingbottled water or installing in-line filtration.However, an extension of the public water line andhookup of residents should be undertaken as soon aspossible. It is further recommended thatWestinghouse legally retain the rights to any privatewells once public water has been supplied to allowfuture monitoring and sampling during the assessmentand abatement program. Additionally, all private

i wells should be secured to prevent usage by"" homeowners to prevent creation of pumping centers

that will influence the spread of contaminants.

B. Packers should be installed in new monitoring wellsKW-1 and MW-3 to preclude any chance of contamination

v> being transferred between two different water-bearingV >\.v"& zones. These packers would be temporary until/such

• / .time as further testing could be completed to• .-A "•' »•^determine flow patterns within these wells and

•s|eohtaininant contributions from each zone.

C. . Conduct an environmental inventory of the Gettysburgplant to validate the integrity of current operationsto assure that there is no ongoing source ofcontamination. included would be a liquid positive

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409R02

displacement test of the line leading from the fillpipe to the 1,1,1-trichloroethane storage area.; Itis recognized that an air pressure test had beenperformed on this line, but because only a small leakcan cause significant groundwater contamination, themore accurate test recommended should be performed.

D. Determine which regulatory agency, EPA or DER, willoversee and control assessment and abatementactivities and then meet with them to discuss thefollowing: . . ..•.;• "

*•'»•-'' .,

.

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I."y1i Extent of problem as currently known.

v^-> yô*" 2. Emergency action taken, or to be taken.

3. Requirements and goals of an assessment andabatement plan, including:

a) Soils and rock drilling, sampling, and_ well construction.

b) Pumping tests to determine aquifer charac-t . \>r.uA teristics.

c) Disposal of and determination of need todelist pumping test discharge water.

d) Additional water quality sampling tocharacterize natural variation over time.

E. Subsequent to meeting with the regulatory agency, andknowing its requirements, a formal Groundwater

409R02 '

Quality Assessment and Abatement Plan should beprepared and submitted to the agency for approval.

Long-Term Acjrjon - Upon approval of the Groundwater QualityAssessment and Abatement Plan by the regulatory agency,Long-Term Action would constitute the implemention of bothparts of the Plan. As both programs will have to be givencareful consideration to meet the goals set forth byWestinghouse and the regulatory agency, our recommendations atthis time should only be considered as a "straw man" upon whicha formal plan can be developed. Based upon our prior experiencewith similar contamination incidents and requirements of theregulatory agencies, we would recommend the following:

A. Implement a groundwater quality assessment plan whichwould include:

1. Soil drilling and sampling, and constructionof shallow groundwater monitoring wells todetermine the exent of soil contamination andgroundwater contamination of the shallowgroundwater system. (Proposed drilling loca-tions are shown on Drawing I8409-007-B.)

2. Drilling additional bedrock monitoring wells todefine the boundaries of the contaminant plumeand to establish a more complete samplingnetwork. (Proposed locations are shown onDrawing I8409-007-B.)

3. Sampling of all monitoring points at least fourmore times to establish variation in contaminant

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409R02 8

concentrations over time to enable developmentof an appropriate abatement system. ' , ;

4. Conducting continuous rate ( ± 48-hour) pumpingtests on two or more wells to define aquifercharacteristics, and to enable definition ofsize and migration of the contaminant plume,interaction of on- and off-site wells, and toaid in development of an appropriate abatementsystem.

5. Determine size and rate of migration ofcontaminant plume using data derived from stepsoutlined above and assisted by computer modelingwhere appropriate.

B. Determine need for soil removal and disposal usingdata derived during the assessment program andrequirements of regulatory agency.

C. Establish the best means for groundwater removal at arate capable of containing and abating contamination.This could be either by single purge well removal ormultiple purge well removal for the deep groundwatersystem. For the shallow groundwater systems, purgewells or interceptor trenches may be considered.

ji-'f _.-.D. J£$|Uaing the information derived from the assessment

lllgrograia, the number of groundwater withdrawal points,?'the volume of water to be treated, the influent waterquality and the effluent water quality criteria will

IT.tS.AR305397

409R02

be known, and an evaluation of groundwater treatmentalternatives can be made. These would include:

1. Granular activated carbon.

2. Biological treatment.

*

3. Air-stripping by packed column or spray field.

4. Modifications or combinations of the above.

This evaluation should include cost, feasibility,maintenance, and effectiveness.

E. Treated water will have to be disposed of andalternatives for disposal will have to be evaluatedsimilar to alternatives for treatment. .Disposalalternatives include:

1. Discharge to municipal sanitary sewer.

2. Discharge to surface water course.

3. Subsurface reinjection - with oc without in-situbioreclaxaation.

F. ...fjiiySJfe-. & final step, the Groundwater Quality Abatement>gram would be formalized and implemented. ThisLd include:

1. Design of treatment and disposal systems.

2. Procurement of all necessary permits.

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409R02 . 10

3. Installation of waste water treatment anddisposal system. '

.4. Regular monitoring of system performance asrequired by regulatory agencies to include:

. Influent water quality.

. Effluent water quality.

. Water quality in adjacent monitoring wells.

Estimated Coats

The recommended emergency action is estimated to involveapproximately $16,000 of REWAI charges to proceed through onemeeting with DER or EPA. As Westinghouse has been entirelyresponsible for emergency water provision to affected homes, noestimate has been made for the cost of supplying water.

Further, until the appropriate assessment plan is formulated, itis inappropriate to estimate total cleanup costs, except to saythat our experience on similar problems permits us to estimatethat a $300,000 to $400,000 cost may be incurred to remedy theproblem at your Gettysburg plant. This would be exclusive ofongoing operational and maintenance costs which can be expectedfor several years to come.

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VOLUME X v-_- _'- L,"" •_

COMMONWEALTH OF PENNSYLVANIAENVIRONMENTAL HEARING BOARD

WESTINGHOUSE ELECTRICCORPORATION

Versus Docket 88-319-CP-F

COMMONWEALTH OF PENNSYLVANIADEPARTMENT OF ENVIRONMENTALRESOURCES

Verbatim transcript of hearingheld in Hearing Room B, 101South Second Street, Harrisburg,Pennsylvania, on Tuesday,

January 21, 199210:00 a.m.

BEFORE: HONORABLE. TERRANCE J. F I TZ PATRICKMember

APPEARANCES:

DICKIE, McCAMEY & CHILCOTETwo PPG PlacePittsburgh, Pennsylvania 15222-5402

BY: DAVID J. ARMSTRONG, ESQUIREAnd

KENNETH KOMOROSKI, ESQUIRE

For - Westinghouse Electric Corporation

CAPITAL CITY REPORTING SERVICEBOX 11908 FEDERAL SQUARE STATION

HARRISBURG, PA. 17108TELEPHONE (717) 533^-2195

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1

2 ..APPEARANCES (Continued):

3 IIMARY PECK, ESQUIRE

4 || Assistant CounselSoutheast Region

5 || Department of Environmental ResourcesLee Park

6 || 555 North LaneSuite 6015

7 || Conshohocken, Pennsylvania 19428

8 And

9 || JUSTINA M. WASICEK, ESQUIREBureau of Regulatory Counsel

10 || Third Floor, City TowersThird & Chestnut Streets

n Harrisburg, Pennsylvania 17120

12 For - Department of Environmental Resources

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INDEX TO WITNESSES

i DIRECT CROSS REDIRECT RECROSS

For The Commonwealth;

Gary Hull " 1884 1911 1922 1925,

Barbara L. Harper, Ph.D. 1927 2032

* * *

' .

1 IPINDEX TO EXHIBITS

IDENTIFIED ADMITTED

Department Exhibits:

5 C-122 Toxicological Profile for 1932 1955Triehioroethylene

6C-131 Sample Results of Westing- 1962

7 house and its consultantsfrom Work Plan, "Summary

8 of Historical On-SiteAnalytical Data and

9 Summary of ResidentialWell Analytical Data"

10C-135 Testing Consent Order for 1963

n 1,1,1-Trichloroethane andResponse to the Interagency

12 Testing Committee

13 C-136 Research Article, Kukong- 1965vriyapan, "Interference

14 with HepatocellularSubstrate Uptake by

15 1,1, 1-trichl oroethaneand Tetrachloroethylene'

16C-137 Review Article, Bruckner, 1966

17 et al, "Metabolism,Toxicity, and Carcino-

18 genicity of Trichloro-ethylene"

19C-138 Occupational Exposure to 1967

20 Trichl oroethy lene, NIQSH

21 C-139 Ozone Protection - Methyl 1979Chloroform ban offers

22 single greatest step,Environmental Policy

23 Alert, 1989

24

25

.1 1873

2 C-1'40 Research Article, Steup, 1967et al, "Pretreatment with

3 Drinking Water SolutionsContaining Trichloro-

4 ethylene or ChloroformEnhances the Hepatoxicity

5 of Carbon Tetrachloridein Fisher 344 Rats"

6C-141 DER Hygienic Information 1978

7 Guide No. 6, Trichloro-ethylene (1971)

8C-142 Von Oettingen, "The 1968

9 Halogenated Hydrocarbonsof Industrial and Toxi-

10 " cological Importance" (1964)

ll C-143 Toxicology and Hygiene of 1969Industrial Solvents, Lehman

12 and Flury (1943)

13 C-144 Volatile Contaminants 1976of Drinking Water,

U 247 Science 141,Abelson, 1990

15C-145 ATSDR Profile for 1970

16 1,1,1-Trichl oroethane

17 C-146 Computer data for 1971Triehioroethylene

18C-147 Computer Data for 1972

19 1,1,1-Trichloroethane

20 C-150 Health Assessment Document 1972for Trichl oroet.hyl ene

21C-151 Guengerich, "Role of Human 1973

22 Cytochrome P-450 II E 1in the Oxidation of Many

23 Low Molecular WeightCancer Suspects"

24C-152 Cooper, "Poisoning by 19.74

25 Drugs and Chemicals"

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C-153 "Phase-out of Methyl 1980 —^Chloroform Use Calledfor by NRDC in ProtectOzone Layer"

C-154 Fan, "Trichl oroethy lene: 1975Water Contamination andHealth Risk Assessment"

C-200 Curriculum Vitae of 1932 1955Barbara L. Harper, Ph.D.

Defendant's Exhibits

D-31 March 9, 1988 letter from 2075EPA to Lee M. Thomas,Administrator of EPA

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JUDGE FITZPATRICK: Ms. Wasicek, are you

going to proceed this time?

MS. WASICEK: Yes, Your Honor.

JUDGE FITZPATRICK: Go ahead.

MS. WASICEK: For our next witness, the

Commonwealth calls Dr. Barbara Harper.

BARBARA L. HARPER, having

been duly sworn, was called

as a witness and testified

as follows:

DIRECT EXAMINATION

15

]6 BY MS. WASICEK:

17 Q Dr. Harper, could you please state your

name for the record?

A Barbara Lynn Harper.

Q What is your office address, Dr. Harper?

A Fourth Floor, Fulton Building, DER.

Q Can you tell me what your position is?

A I am the Chief of Advanced Science and

Research for DER.

Q Do you hold a general title?

fiR305l»08

2 A Public Health Toxicologist is the cla

3 title.

Q How long have you held that position?

5 A Over two and a half years, since I came

6 he re in June of 1989.

7 Q What are your professional duties as

8 part of your employment with the Department of Envi-

9 ronmental Resources?

10 A Quite a number of things. I was brought

n here to develop a team of advanced science profession-

12 als. That's what Advanced Science and Research is.

13 They're supposed to be all doctoral - level people in a

14 wide variety of expertise, whatever is identified as j

15 the most pressing needs at the time for DER.

16 I provide expertise in risk assessment,

17 toxicology, both in terms of developing policy and in

18 terms of preparing technical guidance.

19 I review some risk assessments or advise

20 other programs if there are particular questions that

21 they need advice on relating to health effects of

22 chemicals, risk assessment, and that kind of thing.

23 I review scientific literature in gerier-

24 al and keep up with various professional societies and

25 the state of the art of toxicology; participate on DER

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advisory and technical committees; review some of the

regulations that we develop if they pertain to toxics.

Those are the internal kinds of things.

External things -- maintain a liaison

with, for instance, the Pennsylvania Medical Society,

the medical community, academic community; represent

DER on various national or state professional society

type committees; answer public questions on health

effects of chemicals, toxicology, and also internal

ones as those questions arise.

That's all I can think of right off.

Q Does your work involve the calculation

of levels of chemicals?

A At times, yes, we get into that level of

detail as far as actually calculating risk levels.

Q Can you tell the court what the manage-

rial parts of your job duties are?

A In developing our team of advanced sci-

ence professionals, I would supervise those. Right

now, we have a Ph.D. Mathematician-Statistician, and

we're getting a Health Physicist, and we have an Envi-

ronmental Economist position posted. All those would

be doctoral level.

Q Are any of your duties including the

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supervision of how current these people keep in their

fields?

A That's actually a requirement, that they

must continue their professional development and keep

up, say, in whatever their area of expertise is.

Q In addition to your employment with the

Department, do you have other professional functions?

A I am an Adjunct Assistant Professor of

Environmental Toxicology at Penn State, Middletown* *" '

Campus.

I am preparing a course to teach this

spring on environmental risk assessment.

Q What do your academic responsibilities

as Adjunct Professor involve?

A It involves not just teaching a course,

but supervising -- they have a Master's Degree program

there, and I am supervising a couple of students for

that.

If something is related to DER, DER can

provide expertise, I will be liaison for other type of

research activities for those students.

Q Are you on any committees as part of

your academic responsibilities?

A Non-DER committees?

flR3G5l4ll

I—I1 1 . 1931-,

2 ._„: Q Just as part of your adjunct professor-^-X

3 ship?

4 A There is an Environmental Pollution

5 Control Program Committee at Penn State. I am one of

6 three DER representatives on that. It's composed of

7 industry people, government, and faculty, to develop

8 the program according to the job market, basically,

9 what the students really need to be trained in.

10 . Q Tell me generally, what is toxicology?

n A It ' s a study of chemica'l effects on

12 ' anything. It can be on a cell, a whole organism,

13 ecosystem.

<• 14 Q What kinds of studies are involved in v j

15 toxicology?

16 A A whole variety of things. Experimental

17 methods can include in vivo which is in whole animal

18 type of experiments, or in vitro which is, like, cul-

19 tured cells, or test-tube kinds of systems.

20 It involves metabolic studies, how the

21 chemical is metabolized in the body and what other

22 risk factors there are in the expression of health

23 effects.

24 It can involve things related to carci-

25 nogenicity or mutagenicity or teratogenicity.

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Shall I define those as I go along?

that.

Q We could probably wait until later to do

Does the field of toxicology.also in-

clude various routes of exposure?

A It can. Toxicology, I think of as, once

the chemical gets to the cell or to the organism, it's

the study of the effects.

A very related field with a lot of over-

lap is risk assessment, which includes how the chemi-

cal actually gets either to the organism or within the

organism to the target tissue.

Q Does your practice of toxicology include

risk assessment?

A Yes. That's one of my primary activi-

ties at DER.

Curriculum Vitae ofBarbara L. Harper, Ph.D. --produced and marked foridentification as Common-wealth Exhibit No. C-200.

BY MS. WASICEK:

Q I show you a copy of a document marked

C-200. Can you please identify that document?

A That's my C.D. or Curriculum Vitae.

Q Is that an accurate representation of

«B305M3

2 you?1 professional and academic experiences and quali-

3 fications?

4 A Yes.

5 Q Dr. Harper, are you a member of any

6 national committees?

7 A A couple. On Pages 4 and 5 -- on Page

8 5, there are a couple of committees.

9 One that I represent DER on is the Ches-

10 apeake Bay Environmental Toxics Committee, which is a

11 committee that is awarding research funds for fate and

12 transport and risk assessment models for the Chesa-

13 peake Bay. Pennsylvania is a member of that, since

14 Pennsylvania is one of the watershed states. \^/

15 ASTM is American Society for Testing and

16 Materials. It's also a national committee. I was

17 invited to be a member of that, and two subcommittees,

18 the Exposure Assessment Group and Risk Characteriza-

19 tion Group.

20 Q Going back just a minute. Can you tell

21 me who the other members of the Chesapeake Bay Pro-

22 gram, Environmental Effects Committee, are?

23 A Other toxicologists from NASA and from

24 NOAA and a couple from other states.

25 Q what does "NOAA" stand for?

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mittee?

1934

National Oceanic Atmospheric Administra-

What are the members of the other com-

A The ASTM Committee is composed of again.

toxicologists. There's some from EPA and other gov-

ernment agencies. There's some from academia.

There's some from state governments and there are a

few from environmental groups, kind of a public par-

ticipation. There are quite a few from industry.

It's really a consensus kind of committee.

Q What are their purposes?

A To develop methods for risk assessment

in conjunction with some of the EPA efforts. The

whole purpose is that all these agencies or represent-

atives agree ahead of time on the best methods to use.

Q Have you in the past been involved in

other committees of international stature regarding

exposure assessment?

A I was on one, the International Program

for Chemical Safety which was sponsored by World

Health Organization in 1983 to 1985. That was a pro-

cess that took several years. It was to compare some

of the short-term test methods for predicting carcino-

SR3Q5M5

1 193'

2 geni-city between labs and then all gathering together"—""^

3 and comparing results on identical sets of chemicals .

4 and making recommendations as to if there are varia-

5 tions between the tests, what those were due to.

6 Q Did the committee also discuss topics

7 such as mutagenesis, lab research, things like that?

8 A That's really what I mean by short-term

9 tests. The mutagenicity test, testing for mutagenici-

10 ty in a test tube is really a short-term way to pre-

11 diet which chemicals will turn out to be carcinogen if'

12 tested in long-term bioassays.

13 Q Are you familiar with the work of other

<• 14 professional toxicol ogis ts?

15 A Yes. A big part of what I need to do is

16 keep up with everybody else.

17 Q How do you familiarize yourself with the

18 work of other toxicol ogists?

19 A I keep up with the literature in gerier-

20 al, talk to them at meetings. I attend meetings when-

2i ever possible. Primarily through the literature.

22 Q Do you interact with toxicologists in

23 the faculty or on the committees?

24 A Inasmuch as toxicologists are members of

25 most of those committees, yes. I am familiar with a

1 1936

2 number of industry toxicologists. For instance, Equa-\

3 Tech, one of our DER advisory committees, has some

4 toxicologists from industry. I attend seminars at the

5 Medical Center, for instance, those are basically

6 toxicology seminars there, too.i

7 Q Do you track information data bases

8 published by EPA, anything like that?

9 A There are two types of data bases. One

10 is the literature'itself in which you actually keep up

u with what articles are published. And then there are

12 EPA or National Library and Medicine data bases which

13 for IRIS, for instance, it summarizes EPA's consensus

u on a chemical specific basis. That's an informational

15 kind of data base. So, I keep up with both of those.

16 Q Could you tell the Judge whether you

17 hold any certifications?

18 A I am a Dipl ornate of American Board of

19 Toxicology.

20 Q When were you certified?

21 A November of 1989.

22 Q What is the American Board of Toxicolo-

23 gy?24 A It's a non-profit group. It's really

25 the only way to judge the qualifications of somebody

6R3Q5U7

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2 who-wants to call himself or herself a toxicologist .^ — '

3 Q Does it set national standards for toxi-

4 cology?

5 A It is a national -- not certification in

6 the sense of a medical license, but it does set stan-

7 dards for toxicol ogists .

3 Q How does one become certified by the

9 American Board of Toxicology?

10 A One has to present credentials -- for

H instance, how long you've been doing toxicology or -

12 what courses you've taken, and then sit for a two-day

13 exam.

14 Q Does the American Board of Toxicology \ j

15 require updating of credentials?

16 A It requires a re-test every five years.

17 Q Did you take and'pass the test?

18 A Yes. The first time.

19 Q How is the American Board of Toxicology

20 regarded within the profession of toxicology?

21 A It's really the only way to really judge

22 somebody's credentials on a standardized basis, other

23 than something like an academic position.

24 Q Are you a member of any scientific soci-

25 eties?

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A Several. The premier ones are Society

of Toxicology, Society for Risk Analysis and the Amer-

ican Public Health Association.

Others are the Environmental Mutagen

Society, American Association for the Advancement of

Science. There are local chapters for several of

those and I am also a member of those.

Q Do you belong to Pennsylvania Public

Health Association?

A Yes, that's the local chapter of the

American Public Health Association.

Q Are you involved in any local chapters

of Society for Risk Analysis?

A Yes. There's a Philadelphia Chapter for

that and there's a Mid-Atlantic Chapter for the Soci-

ety of Toxicology.

Q Have you received any writing awards?

A One book award for the "Health Detect-

ive's Handbook."

Q Do you remember the year?

A I would have to look it up here. Are we

on a page?

Q No. That's all right if you don't re-

call. It's in your resume? . .

l 1939

2 _ A Yes, it's here somewhere.

3 Q What work have you done regarding

4 health-based chemical exposure standards?

5 A I've done toxicology research for ten or

6 fifteen years.

7 Q What has that research involved?

8 A Lots of different things.

9 Starting from my degree and going for-

10 ward from there?

n Q Perhaps you could just generally tell me

12 the general classes of what your research has invol-

13 ved?

1 - 1 4 A T h e general classes include some bio- ,

15 chemical genetics-immunology for a few years, and then

16 the rest of it has all been toxicology related. Muta-

17 genicity, carcinogenicity, metabolism, terato-

13 genicity -- basically the effects of chemicals on

19 either the genetic apparatus, DNA, or enzyme systems

20 in the body.

2) Q Has any of the research and teaching

22 involved chemicals within the class of volatile organ-

23 ic compounds?

24 A Within that class, actually most of my

25 research is related to that.

!

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. ..'. Q What is a volatile organic compound?

* A It can be as broad as it's any small

chemical that evaporates readily. In a more narrow

sense, we often think of it as TCE and chlorinated

solvents that are closely related.

Q Are volatile organic compounds referred

to as VOC's?

A That's an abbreviation.

Q Are TCE and TCA volatile organic com-

pounds?

A Yes.

Q What similarities are there between_

benzene and TCE and TCA?

A They are both small volatile solvents.

Q By what are they activated?

A They are activated in the body by the

same enzyme system, in the liver primarily.

Q Are there any overlapping effects among

these substances?

A TCE and its whole class, including TCA,

perchloroethylene, -1,1 and 1,2-dichloroethylenes and

dichloroethanes -- they're all very similar metaboli-

cally. They're al1 , activated and detoxified by the

same systems in the body.

SR3Q5U2I

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2 •--'' JUDGE FITZPATRICK: I'm going to show my

3 ignorance here, but what do you mean by "activated?"

4 THE WITNESS: Okay. In the process of

5 detoxification, the goal of the liver -- primarily the

6 liver -- is to make the substance more soluble so that

7 it can be filtered out by the kidney more readily.

8 The way it does that is to attach the foreign chemical

9 to a substance, a natural substance in the body. But

10 in order to attach it, it has to activate it or make

11 it more reactive so it will react with those other

12 mol ecules .

13 During the time in which it is activated

14 but before it is attached to the carrier molecules,

15 can attack the DNA, for instance. It can attack other

16 proteins. It can attack membranes. That's where a

17 lot of the toxic effects actually come from.

18 BY MS. WASICEK:

,9 Q Could you define for me what teratogene-

20 sis is?

21 A In a narrow sense, it's the study of

22 birth defects. In a little broader sense, it includes

23 any risk factors, things that you don't really consid-

24 er as over a birth defect, like low birth weight,

25 growth, retardation, and other things like that.

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---.. Q Does that involve exposure to chemicals?

A It can.

Q Could you define carcinogenesis for me?

A That's a study of the entire process of

cancer, from the-initial mutation in the DNA or the

initial DNA damage, all the way through progression

and selection for the tumor cells and eventually the

tumor itself .

Q Could you define mutagenesis for us?

A Mutagenesis is really a study of the DNA

damage, whether it's a chromosome damage or an actual

mutation, a variety of things. But it's all geared

toward the DNA itself.

Q Has your research and teaching included

work in each of these areas of teratogenesis, carcino-

genesis and mutagenesis?

A All three.

Q What have the subjects of your teaching

research involved?N.

A A variety of things. Some introductory

material, basic genetics and biochemistry. More spe-

cialized has to do with metabolism of foreign chemi-

cals and the damage that those chemicals can do.

Q Has it involved chemical interaction?

ft«305«i23

12 ----- . A To some extent, yes.

3 Q Has it involved oral exposure and inha-

4 lation?

5 A Both.

6 Q Has it involved animal studies?

7 A Animal studies and human studies.

8 Q Has it involved whole animal and

9 in vitro studies?

10 A Both.

11 Q Have you collaborated with other toxi-

12 cologists?

13 A That's primarily how you do research, is"i14 || as groups .

15 Q Have you been involved in short-term

16 tests to predict if a chemical will cause tumors in

17 animals or humans?

18 A That was a lot of the focus of our

1 9 1 1 research, using short-term tests to predict or to

20 confirm chemicals that would turn out or had turned

2i out to be carcinogenistic. To determine whether a

22 chemical is carcinogenic really takes long, expensive

23 animal bioassays. So, short-term tests that can pre-

24 diet the outcome are very valuable.

25 Q Have you done any studies involving huma

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A Some, yes, both occupational -- I was

involved in some exposure to formaldehyde at the Medi-

cal Center, and some of the work has involved compari-

son in genetic responses between smokers and non-smok-

ers .

Q Has it involved cancer or chemotherapy

with humans?

A One actually did. It was looking at

cells taken from people who were receiving chemothera-

py or not.

Q How would you use the results of your

research?

A The main way is as published in peer

review articles.

Q Why do toxicologists publish?

A Besides career advancement, to present

the information to other toxicologists after it has

been through the process of peer review.

Q Is one of the objectives of publication

to get information to affected people?

A Yes. The ultimate goal is to get the

information, for instance, to the physician who is

treating the patient or ultimately to the exposed

person themselves.

2 ----- Q You talked about peer review. What doeV*'

3 peer review mean?

4 A It's generally -- the data is presented

5 to other scientists, the data and the methods. For

6 most articles, before they are accepted for publica-

7 tion, they have to be reviewed by two or three other

8 scientists who are familiar with the methods.

9 Q Have you been involved in peer review

10 activity?

n A I have be en on an editorial board of a

12 journal and done some other manuscript review for

13 other journals.

14 Q Have you served as a consultant? ^ J

15 A I was a consultant to EPA for a couple

16 of years reviewing some aquatic toxicity documents.

17 That is the primary way.

18 Q Where did you attend college?

19 A Occidental College in Los Angeles.

20 Q What degrees do you hold?

21 A Biology.

22 Q What was your final degree?

23 A It was a B.A. in Biology.

24 Q Did you obtain any academic honors in

25 college?

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~ "' A It was cum laude with departmental hon-

ors which was based on research projects.

Q What was your field of concentration?

A In college, it was basically a pre-med

course.

Q And your degree was in biology?

A Right.

Q Can you tell me some of the science

courses you took in college regarding chemistry?• /

A There were two semesters of organic

chemistry, two semesters of general chemistry. I

would have to look and see, I don't remember --

Q Did you take any inorganic chemistry,

for instance?

A I think inorganic was a different

course.

Q Did you take any science courses in

college regarding biology -- would you name some of

the courses?

A Lots. Microbiology, immunology, neurol-

ogy. Various kinds of biochemical courses.

Q Did you study physiology?

A Anatomy and physiology, yes.

Q Did you study genetics?

SR305l*27

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2 "- A And genetics.

3 Q Did you take any physics courses in

4 college?

5 A Two semesters.

6 Q And this was equivalent to a pre-med

7 program there?

8 A Yes. The pre-meds and the non-pre-meds

9 were all in the same program. We all took the same

ip courses.

n Q Do you hold a graduate degree?

12 A A Ph.D.

13 Q Where did you obtain your Ph.D.?

14 A University of Texas at Austin.

15 Q When?

16 A 1974.

17 Q What was your Ph.D. in?

13 A It was in the Department of Zoology with

19 a specialty in Genetics.

20 Q What does zoology cover?

21 A Anything related to -- well, not just

22 animals. It can be a little broader than just ani-

23 mals. Anything from humans on down to very primitive

24 kinds of organisms.

25 Q Does it involve the study of common

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living animal systems?

A Of what?

Q Of living animal systems?

A Yes, extensively.

Q What graduate courses relating to toxi-

cology did you take leading to your Ph.D.?

A Let's see. There was mutagenesis, bio-

chemical enzyme systems, regular biochemistry. Proba-

bly some other things, as well as the broader kinds of

courses like immunology and genetics.

Q What relationship did your graduate

courses have to the field of toxicology?

A All of it is really required as a foun-

dation for toxicology.

Q What was your professional experience

prior to coming to DER?

A I was an Assistant Professor at the

University of Texas Medical Branch in Galveston.

Q What years were you there?

A As a faculty member, from 1981 to 1989.

Before that, I had two postdoctoral fellowships there,

also'.

Q Is the University of Texas Medical

Branch, a graduate medical school?

fl-R3-05«i29

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2 - A It has two programs: a medical school ""•

3 and a graduate school. They're both there.

4 Q In the period 1981 to 1989, what did you

5 teach?

6 A I gave various lectures to graduate

7 students and to medical students on areas relating in

8 general to toxicology. Then I also was course coordi-

9 nator and taught Issues in Preventive Medicine and

10 Community Health to our graduate students.

n Q Did your teaching involve issues of

12 environmental health?

13 A Yes. I was in the Department of Preven-

"- 14 tive Medicine, Community Health, the Division of En

15 ronmental Toxicology. So, our division really focused

16 on the overlap between environmental toxicology and

17 public health.

13 Q Did your teaching involve how environ-

19 mental factors interact?

20 A Yes, quite extensively.

2i Q Who were you teaching?

22 A Both medical students and graduate stu-

23 dents.

24 Q Graduate students in the field of sci-

25 ence?

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~ "' A Yes, within our own department and other

allied health fields, whatever graduate students

signed up for our courses.

Q Has your work involved how environmental

factors impact upon living organisms?

A That's really integral to all of it.

Q Did it involve direct action on DNA?

A Yes. Most of the laboratory research

really was involved with effects of chemicals on DNA.

Q Has your work involved interaction of

chemicals?

A Yes, in the sense that experimentally,

it's very common to give one chemical to an animal and

challenge it with another and see what effect the

first one had on the response to the second one.

Q Does that involve how the organism re-

lates to a target chemical?

A I'm not sure what that question .means.

Q Did ybur work involve how chemicals

interact together and singly in affecting how an or-

ganism responds to a target chemical?

A I'm not sure you're really asking the

right question.

Q Okay. Did you study organism's respons-

l 19

2 es"to chemicals?

3 A Yes, in several ways. Either at the DNA

4 level or at the enzyme level.

5 Q What did you teach in your Preventive

6 Medicine courses?

7 A The course itself was a broad course.

8 The segments I actually taught were all the environ-

9 mental, carcinogenicity section.

10 Q Did you teach anything about statistics?

n A A little bit about statistics, epidemi-

12 ology, general public health statistics, and a little

13 bit about how to use statistics in the lab.

14 Q What is pathophysiol ogy?

15 A It' s kind of a general name for how an

16 organism responds on a tissue level or an organ level

17 to, for instance, a chemical insult.

13 Q By a chemical insult, do you mean expo-

19 sure to a chemical?

20 A Yes. I mean exposure.

2i Q And it's within the body?

22 A Yes. .

23 Q In what courses, if any, did you discuss

24 pathophysiol ogy?

25 A. Both the Preventive Medicine course, the

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analogous course that we taught to medical students,

and then our own graduates took more specific genetic-

toxicology courses and I gave lectures in that.

Q Have you given lectures in regard to

particular chemicals?

A In most of those lectures, chemicals

were used as specific examples.

Q Did you give lectures on benzene?

A Benzene is a frequent choice of typical

environmental chemicals.

Q You said that you also were at the Uni-

versity of Texas Medical Branch prior to serving as a

teacher there. What was your experience prior to that

time?

A I had two postdoctoral fellowships. The

first one was in the Department of Human Biological

Chemistry and Genetics. We were studying some of the

biochemical mechanisms of cystic fibrosis.

The second one was in the Department of

Medicine, Division of Infection and Immunity, Division

of Infectious Diseases. That was related to some ofs

the biochemical mechanisms of the immune response.

Then I pulled those things together and\

focused more on genetic toxicology.

ftR3Q5«i33

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2 Q Were you then a research associate after

3 that time?

4 A Yes. There was a one-year period in

5 between the post-doc's and the faculty appointment.

6 Q As far as fellowships, did you teach?

7 A Yes. Graduate students always teach.

8 Q Do you remember what you taught?

9 A Again, it was miscellaneous lectures in

10 genetics and biochemical genetics, primarily.

11 Q And toxicology?

12 A A little bit as I was getting more into

13 the genetic toxicology area.

14 Q Have you participated in research act

15 ities?

16 A Extensively.

17 Q What was the focus of your research

18 activities generally?

19 A Most of them focused on responses of

20 animal to chemicals.

21 Q Has your research involved genotoxicity?

22 A That's sort of a shorthand for genetic

23 toxicology which is really how the chemical affects

24 the DNA.

25 Q Has your research involved the geno-

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toxrcity of benzene with metabolism?

A Yes, quite extensively.

Q How many research activities have you

been involved in that focused on mutagenesis or carci-

nogenesis?

A I listed about a dozen in here. Those

are the funded activities. One always participates in

extra experiments -- somebody else's research, for

instance.

Q Have you written or contributed to arti-

cles? .

A About 22 or so.

Q Were these articles peer reviewed?

A Most of them were.

Q What are abstracts, Dr. Harper?

A They're summaries of work. There's two

kinds of abstracts. One is the summary of the res-

earch paper.

The abstracts that are listed in here

are the summary of research that you present at a

professional meeting. Generally it's before you pub-

lish a paper.

Q How many abstracts have you written or

presentations have you given at these meetings?

195C

2 ,-, A I think there are 27 or 28.

3 Q Have you written any chapters in books?

4 A Aboutadozen.

5 Q What steps, if any, have you been taking

6 to keep up-to-date in your profession?

7 A Again, it's keeping up with colleagues,

8 keeping up with the literature, keeping up with, for

9 instance, EPA standard methods. I guess that kind of

10 summarizes it.

n MS. WASICEK: When you stated that Ex-

12 hibit C-200 was an accurate statement of your experi-

13 ence and background, I would move to admit Common-

• 14 wealth Exhibit C-200.

15 MR. ARMSTRONG: No objection, Your Hon-

16

17 JUDGE FITZPATRICK: Commonwealth's Ex-

18 hibit No. 200 is admitted into evidence.

19 Commonwealth ExhibitNo. C-200, previously

20 identified, admittedinto evidence.

21BY MS. WASICEK:

22Q Dr. Harper, are you familiar with the

23general class of VOC's?

24A Yes .

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1956

Q Will you list some examples of VOC's for

A They're all the chlorinated solvents

that we've been talking about here, and in a broader

sense, similar things like benzene, toluene, xylene --

lots of small volatile solvents.

Q Can you tell me whether any of the fol-

lowing are VOC's: TCE, is that a VOC?

A Yes.

Q Is TCA a VOC?

A Yes.

Q Is PCE a VOC?

A Yes.

Q How about tetrachloroethylene?

A .That is PCE or perchloroethylene.

Q How about dichloroethene?

A Yes, any of the isomers, and the

dichloroethanes, as well.

Q Do you have a lot of general exposure to

VOC's? Not personal human exposure. I mean, has your

work and study involved a lot of exposure to VOC's?

A Yes. Research has, and teaching has, as

well.

Q Why is that?

' 1 19'

2 — A They're very common. They're represent

3 tative of lots of chemical exposures. They are good

4 ways to describe how the body metabolizes and reacts

5 to chemical exposures.

6 Q Are you familiar with trichloroethy1ene

7 in particular?

8 A Yes.

9 Q Is that a widely discussed chemical?

10 A It's often used as a representative for

11 the c 1 ass .

12 Q The class of what?

13 A Of VOC's — chlorinated solvents.

14 Q What are other names for trichloroethj\^J

is ene?

16 A It's got a lot of trade names. It's

17 also called trichl oroethene, which is shorthand for

18 trichloroethylene. It's 1,1,2-trichloroethy1ene.

19 Q Is it also referred to as "tri" some-

20 times?

21 A Sometimes.

22 Q If I refer to "TCE," will you understand

23 that in the course of this hearing to be "trichloro-

24 ethylene?"

25 A Yes.

flR305i»38

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Q Are you also familiar with 1,1,1-tri-

chloroethane?

A Yes .

Q How are you familiar with it?

A Mostly from general reading, as well as

specifically, preparing for this case.

Q What are other names for 1,1,1-tri-

chloroethane?

The other common one is methyl chloro-

form.

Q Is it also called "TCA?"

A Yes.

Q If I refer to "TCA" during the course of

this hearing, will you understand that to be 1,1,1-

trichloroethane?

A Yes.

Q Have you taken steps to update yourself

regarding TCE and TCA?

A Yes .

Q What steps have you taken?

A Reviewed extensively the scientific

literature, occupational and medicine textbooks, data

bases, EPA informational data bases like IRIS.

MS. WASICEK: Your Honor, at this time,

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I would like to offer Dr. Harper as an expert in toxi-*

cology, including but not limited to health based

chemical exposure standards, chemical attributes and

effects of TCE and TCA and risk assessment.

MR. ARMSTRONG: Your Honor, we wish to

reserve any cross examination we have on the issue.

JUDGE FITZPATRICK: I believe the cus-

tomary way to do this is to have voir dire on the

•qualifications of Dr. Harper's expertise. Are you

saying that you wish to hold off until you do your

main cross on that?

MR. ARMSTRONG: Yes, Your Honor. I

don't know that I will have any. As a practical i

sue, I don't wish to waive it should some develop in

the balance of the course of the direct or in some of

my cross examination. I think the practical effect of

that is that I would not be interposing that objection

to opinion evidence offered in the direct, but I sim-

ply would like to reserve any questions I might have

on qualifications as part of my normal cross examina-

tion.

JUDGE FITZPATRICK: Ms. Wasicek?

MS. WASICEK: Your Honor, the only dif-

ficulty that might raise is that I would prefer to

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1960

have"a ruling from the Court now on whether the Court•

can accept Dr. Harper as an expert so that she can

give expert opinion, which is what her remaining tes-

timony will involve.

MR. ARMSTRONG: And, Your Honor, I do

not interpose any objection to the Court making such a

ruling at this time.

JUDGE FITZPATRICK.: Mr. Armstrong, I'm

going to ask you to go forward and take a position now

and question, and then either object or not. I'm very

concerned that we could possibly be in a situation

where we would end up wasting a lot of time because we

would have all the questions and we would be allowing

them and putting them on the record and then there

would be the possibility that you would move later and

say that you object to the expertise.

So, I would like you to go forward now

with voir dire or choose not to do so, one way or the

other.

MR. ARMSTRONG: Your Honor, if .those are

the alternatives, the only two alternatives given, I

have no alternative but to choose not to go forward.

JUDGE FITZPATRICK: So, you have no

objection to Dr. Harper's expertise?

12 ~~ MR. ARMSTRONG: On a motion for the

3 Court to find her to be an expert at this time, I have

4 nothing to say.

5 JUDGE FITZPATRICK: All right. I will

6 admit Dr. Harper as an expert in the fields which Ms.

7 Wasicek described.

8 MS. WASICEK: Thank you, Your Honor.

9 BY MS. WASICEK:

10 Q Dr. Harper, what do toxi col ogists gener-

11 ally reasonably rely upon in making inferences or

12 conclusions in their practice of toxicology?

13 A Several kinds of things. The first

14 would be peer reviewed literature based on experimeiK_X

15 tal evidence that has been published.

16 We would also review on EPA or other

17 government agencies scientists review of the peer

13 review literature.

19 There are some peer review data bases,

20 as wel 1.

, 21 Q So' toxicol ogists reasonably rely upon

22 government publications in regard to chemicals?

23 A The government publications that have

24 been put together by groups of scientists within agen-

25 cies, yes.

flR305i»l»2

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1962

Q Can you give me some examples of those?

A EPA has a series of Health Assessment

Documents.

ATSDR has a series of Toxicology Pro-

files on specific chemicals.

Those are the two main examples.

Q And toxicologists reasonably rely upon

data bases published through these agencies regarding

chemicals, like the IRIS data bases?

A Yes, because that has also been peer

reviewed.

Q Do they also rely on textbooks?

A Yes, because they're usually published

by leading people in the field and they also review

the peer reviewed literature that is already out

there.

Toxicological Profilefor Trichloroethylene --produced and marked foridentification as Common-wealth Exhibit No. C-122.

BY MS r WASICEK:

Q We have a series of documents that I

would like you to identify, Dr. Harper.

(Document handed to the witness.)

I ask you to identify that document?

6R305H3

l2 - A The number is C-122. It's the Toxico-

3 logical Profile for Trichloroethylene. It was pub-

4 lished by the ATSDR which is the Agency for Toxic

5 Substances and Disease Registry.

6 Q Have you reviewed this document?

7 A Yes.

8 Q Do you consider it reliable?

9 A Yes.

10 Q Is this a document that a toxicol ogist

11 reasonably relies upon in reaching a professional con-

12 elusion?

13 A Yes.

14 MS. WASICEK: I believe you have copies^—^

15 of all these already that were sent to you. Do you

16 have those handy?

17 MR. KOMOROSKI: I believe so.

18 Testing Consent Orderfor 1,1,1-trichloroethane

19 and Response to theInteragency Testing

20 Committee -- producedand marked for ident-

21 ification as CommonwealthExhibit No. C-135.

22(Document handed to the witness.)

BY MS. WASICEK:24

Q Please identify that document, Dr. Harp-25

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er?

A The number is C-135. It was published

in the Federal Register. Do you want the whole cita-

tion or just the title?

Q Just the title?i

A Testing Consent Order for 1,1,1-tri-

chloroethane and Response to the Interagency Testing

Committee.

Q . Have you reviewed that document?

A Yes.

Q Does it reflect agency consensus?

A It was EPA final rule on the testing for

1,1,1-trichloroethane.

Q Does that reflect a consensus within the

agency?. s

A I think it does.

Q Is this a document that a toxicologist

would reasonably rely upon in reaching a professional

conclusion?

A I would put it in a second tier. It is

a very good review as anything that is a consensus

review that is published in the Federal Register has

to be. It's not peer reviewed in the sense that ex-

perimental data is, but it is a reliable summary of

flR305i»U5

i—

.2 the effects of TCA.

3 Q Is it a document that a toxicologist

4 reasonably relies upon?

5 A Yes.

6 Research Article, Kukongvri-yapan, "Interference with

7 Hepatocellular SubstrateUptake by 1,1,1-Trichloro-

8 ethane and Tetrachloroethylene"-- produced and marked for

9 identification as Common-wealth Exhibit No. C-136.

10BY MS. WASICEK:

11Q I show you another document marked

12C-136. Could you identify that for me?

13A This is a research article. The title

14is, "Interference with Hepatocellular Substrate Uptake

15by 1,1,1-Trichloroethane and Tetrachloroethylene."

16Q Have you reviewed the article?

17A Yes .

18Q Is it reliable?

19A Yes .

20Q Is it peer reviewed?

21A Yes .

22Q Is this a document that a toxicologist

reasonably relies upon in reaching a professional24

conclusion?25

8R305H6

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1966

A Yes. This" is a typical kind of peer-

reviewed research article.

Review Article, Bruckner, et al,"Metabolism, Toxicity, andCarcinogenicity of Trichloro-ethylene" -- produced and markedfor identification as Common-wealth Exhibit No. C-137 .

7BY MS. WASICEK:

8Q I show you a document marked Exhibit

9C-137.- Could you identify that document for me?

10A This is a review article entitled, "Me-

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tabolism, Toxicity, and Carcinogenicity of Trichloro-

ethylene."

Q Who is the author?

A Bruckner, et al.

Q Have you reviewed this?

A Yes.

Q What is its reliability?

A It is quite reliable. It's published in

Critical Reviews of Toxicology, which is really one of

the best reviews that there is.


reasonably relies upon in reaching a professional

conclusion?

A Yes .

2 Occupational Exposure toTrichloroethylene, NIOSH --

3 produced and marked foridentification as Common-

4 wealth Exhibit No. C-138.

5 BY MS. WASICEK:

6 Q I show you a document marked

7 C-138. Would you identify that for me, please?

8 A This is a NIOSH document. It's enti-

9 tied, "Criteria for Recommended Standard, Occupational

10 Exposure to Trichl oroethyl ene." NIOSH is the National

n Institute for Occupational Safety and Health, which is

12 the research arm of OSHA.

13 Q Have you reviewed that document?

u A Yes . Sv—-'

15 Q Is this document reliable?

16 A Yes. It's an excellent review.

17 Q Is this a document that a toxicologist

13 reasonably relies upon in reaching a professional

19 conclusion?

20 A Yes.

21 Research Article, Steup, et al ,"Pretreatment with Drinking

22 Water Solutions ContainingTrichloroethylene or Chloro-

23 form Enhances the Hepatoxicityof Carbon Tetrachloride in

24 Fisher 344 Rats" -- producedand marked for identification

25 as Commonwealth Exhibit No. C-140.

flR305l»i»8

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1968

BY MS. WASICEK:Q I show you a document marked C-140.

Would you identify that for me, please?

A This is a research article. The title

is, "Pretreatment with Drinking Water Solutions Con-

taining Trichloroethylene or Chloroform Enhances the

Hepatoxicity of Carbon Tetrachloride in Fisher 344

Ra t s. "

Q Have you reviewed that?

A Yes.

Q Is it reliable?

A Yes. It has been peer reviewed.


reasonably relies upon in reaching a professional

conclusion?

A Yes.

Von Oettingen, "The HalogenatedHydrocarbons of Industrialand Toxicological Importance"(1964) -- produced andmarked for identificationas Commonwealth ExhibitNo. C-142.

BY MS. WASICEK:

Q I show you a document that has been

marked as C-142. Can you identify that for me?

A This is an excerpt from an occupational

1 . 19'

2 medicine textbook that was published in 1964. The•

3 title of the book was, "The Halogenated Hydrocarbons

4 of Industrial and Toxicological Importance."

5 Q Have you reviewed that?

6 A Yes.

7 Q Is it reliable?

8 A Yes.

9 Q Who was Von Oettingen?

10 A That's one of the big names in toxicolo-

11 gy of that time period, which is the 60's.

12 Q Is this a document that toxicologists

13 reasonably rely upon in reaching a professional con-~114 elusion?

15 Yes .

16 Toxicology and Hygieneof Industrial Solvents,

17 Lehman and Flury (1943) —produced and marked for


19BY MS. WASICEK:

20Q I show you a document marked C-143. Can

21you identify that for me, please?

22A This is also an excerpt from an occupa-

23tional medicine book, 1943. The book title is "Toxi-

24 cology and Hygiene of Industrial Solvents."25

flR305i»50

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Q Have you reviewed that document?

A Yes.

Q Is this a reliable document?

A Yes.

Q Are the editors well known?

A Yes.

Q Is this a document that toxicologists

reasonably rely upon in reaching a professional con-

clusion?

A Yes.

ATSDR Profile for1,1,1-Trichloroethane --produced and markedfor identification asCommonwealth ExhibitNo. C-145 .

BY MS. WASICEK:

Q I show you a document marked C-145. Can

you identify that for me, please?

A This is the Toxicological Profile for

1,1,1-Trichloroethane published by ATSDR.

Q Is this a government document?

A Yes.

Q Is there any peer review involved?

A Yes. It's peer reviewed in the draft

form before it's published like this in the final

fl-R30Sfc5t

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'"Ii. j

form.

Q Ha

A Ye

Q Is

reasonably rely

elusion?

A Ye

CcTrPtfcCcNc

BY MS. WASICEK:

Q I

marked C-146 .

A Tl-

roethy lene.

Q He

A Ye

Q Is

A Ye

Q Is

reasonably rely

elusion?

A Ye

Have you reviewed this document?

Is this a document that toxicologists

Computer Data forTrichloroethylene --produced and markedfor identification asCommonwealth ExhibitNo. C-146.

I show you a document that has been

Can you identify that for me?

This is EPA's IRIS printout for trichlo

Have you reviewed that document?

Yes .

Is it reliable?

Yes .

Is this a document that toxicologists

12

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4Exhibit No. C-147.

5BY MS. WASICEK:

6Q I show you a document that has been

7marked C-147. Can you identify that for me?

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1972

Computer Data for1,1,1-Trichloroethane --produced and markedfor identificationas Commonwealth

A This is the IRIS printout for 1,1,1-

trichloroethane.

Q And have you reviewed that document?

A Yes.

Q Is this a reliable document?

A Yes.



clusion?

A Yes .

Health Assessment Documentfor Trichloroethylene --produced and marked foridentification as Common-wealth Exhibit No. C-150.

BY MS. WASICEK:22


marked C-150. Can you identify that for me?

A This is the Health Assessment Document

1 • 1973

2 for Trichloroethy1ene published by EPA in 1983. ^)


4 A Yes.

5 Q Is that a reliable document?

6 A Yes.

7 Q Is this a document that toxicologists

8 reasonably rely upon in reaching a professional con-

9 elusion?

10 A Yes .

11 Guengerich, "Role of HumanCytochrome P-450 11 E 1 in

12 the Oxidation of Many LowMolecular Weight Cancer

13 Suspects" -- produced and~j marked for identification

14 as Commonwealth Exhibit ,No. C-151.

15 BY MS. WASICEK:16

Q I show you a document that has been17

marked C-151. Have you reviewed that document?18

A Yes.19

Q Could you please identify it for me?20

A This is a research paper. The title is,' 21

"Role of Human Cytochrome P-450 11 E 1 in the Oxida-22

tion of Many Low Molecular Weight Cancer Suspects."23

Q Is this a reliable document?24

A Yes .25

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Q Was it peer reviewed?

A Yes.



clusion?

A Yes .

Cooper, "Poisoning by Drugsand Chemicals" -- producedand marked for identificationas Commonwealth ExhibitNo. C-152.

BY MS. WASICEK:


marked as C-152. Can you identify that for me,

pi ease?

A This is an excerpt from an occupational

textbook from 1958. The title is, "Poisoning by Drugs

and Chemicals."

Q . Have you reviewed that document?

A Yes .

Q Is that a reliable document?

A Yes .



clusion?

A Yes..

AR305U55

1 . 1975

2 :. Fan, "Trichl oroethyl ene: * «XWater Contamination and

3 Health Risk Assessment" --produced and marked for


5BY MS. WASICEK:

6Q I show you a document marked C-154.

7Could you identify that for me?

8A This is a research paper called "Tri-

9chloroethylene: Water Contamination and Health Risk

10Assessment."

nQ Have you reviewed that document?

12A Yes .

"1 Q Is this a reliable document?14 -A Yes.15

Q Is it a document that a toxicologist16

reasonably relies upon in reaching a professional17

conclusion?18

A Yes.19

Q Have you reviewed all of these documents20

that we have just gone through in reaching profession-21

al conclusions in this matter?22

A Yes.23 Q For ease of reference in the record,24 when I refer to the scientific literature you have25

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1976

reviewed to give opinions in this matter, do you un-

derstand that I am referring to Commonwealth Exhibits

C-122, 135, 136, 137, 138, 140, 142, 143, 145, 146,

147, 150, 151, 152, and 154?

A Yes .

MS. WASICEK: Is that acceptable to the

Court, also, Your Honor?

JUDGE FITZPATRICK: Yes. That's accept-

able t.hat you are using that as a collective refer-

ence, yes .

MS. WASICEK: Thank you.

I would like to refer to some other

documents at this point.

Volatile Contaminantsof Drinking Water,247 Science 141,Abelson, 1990 --produced and markedfor identificationas CommonwealthExhibit No. C-144.

19BY MS. WASICEK:

20Q I show you a document that has been

2.1marked. C-144. Can you identify that document?

A This is an editorial that appeared in

"Science" called "Volatile Contaminants of Drinking

Water."

—11 1 197 7_

2 Q Did you review this document? v)

3 A Yes.

4 Q How would toxicologists use a document

5 like this?

6 A This is a typical kind of confirmatory

7 document.

8 Q Is the use that you made of it, a con-

9 firmatory use?

10 A Yes.

n Q Rather than as a basis for your expert

12 opinions?

13 A Right.~~j• 14 Q Who is the author of this document?

15 A Philip Abelson.

16 Q Who is he?

17 A He at the time, I think, was president

18 of AAAS and editor of the "Science" magazine.

19 Q How would you gauge his stature in the

20 field?

21 A Imminent.

22 BY JUDGE FITZPATRICK:

23 Q What do you mean by "confirmatory," Dr.

24 Harper?

25 A I reviewed many other documents. Some

3R3051»58

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we r.e,; research articles. Some were more in the opinion

category, such as this, in which the material that he

cites, he doesn't give a reference for. But because

of his stature, I think he is right about it.

MS. WASICEK: Your Honor, for clarifica-

tion, the documents I am discussing now as opposed to

the ones we just went through, the confirmatory docu-

ments, we are not asserting that she bases her opinion

on the confirmatory documents, that they only se.rve to

confirm, rather than serve as the basis of her opin-

ions.

JUDGE FITZPATRICK: All right.

DER Hygienic InformationGuide No. 6, Trichloro-ethylene (1971) --produced and marked foridentification as Common-wealth Exhibit No. C-141.

17BY MS. WASICEK:

18Q Similarly, I show you an exhibit marked

19C-141. Would you identify that document for me?

20A This is a Hygiene Information Guide for

Trichloroethylene that was published by the Department

of Environmental Resources in 1971.

Q Does this fall into the class of a con-

firmatory document?

ftR305i»59

1 1979

2 ; A It's confirmatory and it's useful be-

3 cause .it shows the occupational standards that existed

4 at the time as far as worker safety goes.

5 Q What use did you make of this document?

6 A Specifically to indicate what the compa-

7 nies in the State were supposed to do as far as worker

8 safety and exposure control .

9 Q But you did not use that as a basis for

10 scientific evidence or opinions?

11 A ' No.

12 Q It was only in regard to showing safe

13 practices?

14 A Right.

15 Q And you would label that a confirmatory

16 document?

17 A The. same category, right.

18 Ozone Protection - MethylChloroform ban offers

19 single greatest step,Environmental Policy

20 Alert, 1989 - producedand marked for identi-

21 fication as CommonwealthExhibit No. C-139.

22 BY MS. WASICEK:23 Q I show you a document labeled C-139.24

What is that document?25

flR305i»60

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1980

~ -. A This is a news article from one of our

environmental weeklies. The title is, "Ozone Protec-

tion - Methyl Chloroform ban offers single greatest

step." Methyl Chloroform being 1,1,1-trichloroethane.

Q Have you reviewed that document?

A Yes .

Q Have you used this as a confirmatory

document only?

A Right. Just confirmatory.

Q Would toxicologists reasonably use that

as a confirmatory document?

A Yes, for specific points.n"Phase-out of MethylChloroform Use Calledfor by NRDC in ProtectOzone Layer" -- producedand marked for identifi-cation as Commonwealth

17 "

15

16

Exhibit No. C-153.

18 BY MS. WASICEK:

19 Q I show you a document marked C-153. Can

20 you identify that document for me?

21 A This is also a news article from a week-

22 ly summary published by the Bureau of National Af-

23 fairs. The title is, "Phase-out of Methyl Chloroform

24 Use Called for by NRDC to Protect Ozone Layer."


AR305i*6l

1 i 1981L

2 ...... A Yes.

3 Q Have you made confirmatory use only of

4 it?

5 A Yes .

6 Q I would like you to refer just a moment

7 to C-122. You stated that you reviewed that document,

8 is that correct?

9 A Yes.

10 Q What is that document?

n A The ATSDR Toxicological Profile for

12' Trichl oroethyl ene .

13 Q That's TCE?

1 14 A Right .

,5 Q What does "ATSDR" stand for?

16 A The Agency for Toxic Substances and

17 Disease Registry.

18 Q How is this document particularly used

19 by professional toxicologists?

20 A It is a review of the literature --

2i toxicology, cancer, all the various health effects in

22 animals and humans of whatever particular chemical

23 it's prepared for.

24

25

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6

7Exhibit No. C-131.

8BY MS. WASICEK:

9Q I am going to show you a different kind

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1982

Sample Results ofWestinghouse and itsConsultants from WorkPlan, "Summary ofHistorical On-SiteAnalytical Data" and"Summary of ResidentialWell Analytical Data" --produced and markedfor identificationas Commonwealth

of document. I show .you a document labeled C-131.

Can you identify that for me, please?

A This is a summary of analytical data for

groundwater for VOC's. There are two parts to it.

The first three pages -- Table 2-5 -- is on-site data.

The rest of it -- Table 2-12 -- is the residential

data.

Q Have you reviewed this document?

A Yes .

MS. WASICEK: Your Honor, at this time,

I would move to admit Commonwealth Exhibit C-131.

This has been stipulated to by the parties for admis-

sibility.

JUDGE FITZPATRICK: Is there any objec-

tion, Mr. Armstrong?

14 or

12 -:•• . MR. ARMSTRONG: I don't know, Your Hon-v

3 or. I am checking into whether there's a stipulation,

4 because if there is, then I don't have any objection.

5 JUDGE FITZPATRICK: All right. Fine.

6 MS. WASICEK: That should be in the

7 Ninth Amended Response.

8 (Mr. Komoroski checking documents)

9 MR. KOMOROSKI: We have so stipulated,

10 Your Honor.

11 JUDGE FITZPATRICK: So, there's no ob-

12 jection, then, to C-131, to the admissibi1ity?

13 MR. KOMOROSKI: No ob jec ti on, . Your Hon-

15 JUDGE FITZPATRICK: C-131 is admitted.

16 Commonwealth ExhibitNo. C-131, previously

17 identified, admittedinto evidence.

18MS. WASICEK: That's all right with you,

19too, Mr. Armstrong?

20MR. ARMSTRONG: It's all right. Any-

21thing the Judge says is all right with me.

22JUDGE FITZPATRICK: It's admitted.

23 (Discussion off-the-record)24

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1984

BY -MS . WASICEK:

Q Dr. Harper, you stated that you reviewed

C-131 and these other documents, and you stated that

TCE does belong to the volatile organic compound

class?

A Yes.

Q Is TCE a volatile chlorinated solvent

within that class?

A Yes. It's kind of overlapping defini-

tions there.

Q Are you aware of uses that had been made

of TCE?

A Yes. It has been used extensively as a

vapor and liquid degreaser, as an anesthetic, fumi-

gant, analgesic, disinfectant, chemical intermediate

and a variety of minor uses such as spot cleaner.

Q Has it ever been misused as a substance

of abuse?

A Yes, quite extensively.

Q What do you mean by that? What is the

substance of abuse-in this sense?

A It's addictive or causes habituation.e>

That has been known since the 1920's and it has been

reported in some of these occupational textbooks as

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2 very, common.

3 Q Dr . Harper, what is meant by an acute

4 health effect?

5 A It's generally any adverse health effect

6 that occurs after two weeks of exposure, or less.

7 Q What is meant by a chronic health ef-

8 feet?

9 A Generally, it's an effect that requires

10 more than two weeks to appear.

n Q Does exposure to TCE have any health

12 effects that are acute or chronic?

13 A It has both.

14 Q Can you state some of the general cate i

15 gories of acute or chronic effects of TCE?

16 A The acute effects are related to central

17 nervous system, anesthetic kinds of things, for in-

13 stance.

19 Some of the chronic effects, the primary

20 one that we're interested in is cancer. It also caus-

21 es neurological effects.

22 There are other acute effects on various

23 organs or tissues within the body.

24 Q Generally, the acute effects relate to

,25 longevity?

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1986

A Well -- death.

Q You're saying that death is one of the

acute effects of exposure to TCE?

A Yes, it has occurred on numerous instan-

ces on overexposure.

Q Why is cancer a health effect of concern

to toxicologists?

A It's irreversible and it's progressive.

So, once the process is started, it doesn't reverse

itself.

Q Breaking down these categories somewhat.

What health effects, if any, on the central nervous

system result from human exposure to TCE?

A These have been reported since 1915, the

turn of the century. There's 20 or 25 symptoms that

are listed, including things like headache, nausea,

vomiting, sleepiness, insomnia, dizziness or vertigo,

euphoria, tremors, 1ightheadedness, drunkenness,

slowed reaction time, disturbances in visual percep-

tion or double vision, memory loss, difficulty in

learning new tasks -- a whole variety of things like

that.

They're related to three general kinds

of effects. One is the anesthetic kind of effect

19 1""*2 which would be the sleepiness, 1 ightheadedness,

3 ness, even unconsciousness, ccma and death.

4 A second kind of effect -- it has ef-

5 fects that are very similar to alcohol and it metabol-

6 ically interacts with alcohol. Those kind of effects

7 are the euphoria, feeling of 1 ightheadedness , or it's

8 been called feeling drunk. Other things related to

9 that like insomnia, some of the other kind of atypical

10 reactions to alcohol that TCE also causes.

11 Then there is a third category of gener-

12 al effects that are in the heading of solvent synd-

_ 13 rome. These are things like slowed reaction time,

•• 14 emotional instability, irritability, some of the

15 ry effects, dexterity kinds of effects, other neuro-

16 logical effects like tremors.*

17 Not allôf these occur in each person.

18 In one person exposed to TCE, they may react by com-

19 plaining of fatigue or tiredness or sleepiness. An-

20 other person might complain of insomnia, just like

21 people respond differently to alcohol.

22 Q So that different people react differ-

23 ently to exposure to these substances?

24 A Right .

25 Q Here, we're talking TCE?

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1988

A Yes.

Q You brought up solvent syndrome. Can

you describe the metabolic interaction of TCE when a

human has also ingested alcohol?

A That's really the "degreasers" flush

more than the solvent syndrome.

"Degreasers" flush -- it's appeared in

the literature for a long time. What .it is, is the

intolerance to alcohol if the person is chronically

exposed to TCE. The symptoms, there's a basal dila-

tion. A person gets real flushed because if they have

already been exposed to TCE and then they drink alco-

hol, they don't detoxify the alcohol as fast as they

should. So, they get detoxic reactions to alcohol.

Q Why don't they detoxify the TCE or alco-

hol as fast as they should?

A .They are metabolized by exactly the same

enzyme system as to alcohol, really all of the*chlori-

nated solvents.

Q What does that mean in regard to the

enzyme that is trying to detoxify either TCE or alco-

hol?

A Well, practically speaking, it has al-

ready been used up trying to detoxify the TCE, so that

1 — I1 • •1 1939

2 when/there is an extra challenge of alcohol, the al

3 hoi is-not detoxified.

4 Q What health effects, if any, on the

5 cardiac system results from human exposure to TCE?

6 A This has also been known since the

7 1920's or 1930's and mentioned in every textbook. It

8 causes what's known as cardiac sensi tization . What

9 this is, is it tends to cause arrhythmias and to sen-

10 sitize the heart to adrenalin or epinephrine which the

11 body naturally produces in response to exertion or

12 stress .

13 A lot of the fatalities that have been

•> 14 reported for TCE are most likely due to cardiac sen? j

15 tization, but unrecognized until the person dropped

16 dead.

17 Q How long have people been aware of the

18 effect of TCE, both on the cardiac system and on the

19 central nervous system?

20 A The first report was 1915. There have

21 been really a steady stream of reports -- hundreds and

22 hundreds of occupational poisonings, anesthetic ad-

23 . verse reactions. Every decade, there's another five

24 or ten papers and they're all cited both in the cur-

25 rent literature and in the occupational textbooks from

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the-:40's, 50's, and 60's.

Q Describe the health effects, if any,

upon mucous membranes of persons who are exposed to

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TCE?

A Like any of the solvents, it dissolves

the lipids that are in the skin, so it's an irritant.

It tends to cause reddening, dryness, irritation of

the skin and mucous membranes which are eyes, nose,

throat, even the lungs.

Q So that they irritate the skin, eyes,

nose, and throat?

A Right.

Q What health effects, if any, on internal

organs result from human exposure to TCE?

A There have been reports all along that

it also causes liver and kidney damage. Some of this,

it was not known whether it was due to pure TCE or to

the stabilizers in technical grade TCE, but they're

consistent reports, and if one looks at the cellular

level, it's not as important an effect as the cardiac

sensitization or the central nervous system effects,

but there most likely are harmful effects to both the

liver and the kidney.

This is particularly important because

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2 we-get into some of the risk factors for reacting

3 adversely to TCE. If a person has any underlying

4 problem, for instance, with the liver, like hepatitis

5 or cirrhosis, metabolic disease -- it places the liver

6 at increased risk. In looking back at the medical

7 literature, it's likely that a lot of the liver re-

8 ports were actually due to an underlying liver problem

9 that was really exacerbated by TCE.

10 Q Does TCE affect the level or interfere

n with triglycerides?

12 A It interferes with triglyceride metabo-

13 lism. Therefore, the triglyceride level in the blood-

-I n stream. That's kind of a general metabolic impair-v J

ment.

Q In regard to central nervous system

effects, are there effects from exposure to TCE in

regard to depression of breathing?

A Yes, because breathing is regulated

through the brain. That's one of the effects that is

related to coma and eventual death.

Q Do you have different effects at differ-

ent levels of exposure to TCE in regard to this de-

pression of breathing?

A For any of the symptoms, you would have

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1992

diff.erent 1 evel s of effects, depending on the exposure

as well as on the individual sensitivity.

Q Are all these health effects resulting

from TCE that you've described, documented in people

who have been exposed to TCE?

A Those are really all based on either

occupational groups -of workers or on, for instance,

aesthetic patients.

Q Have these health effects been document-

ed in animals, also?

A The mechanisms have been studied in

animals and actually, the human studies actually trig-

J " gered the animal studies.

Q Is that usual?

A It's usually the other way around, but

in this case, so much was known in people before we

knew in an animal, before we did the confirmatory

animal experiments, that it's unusual how much we knew

about TCE all throughout its history of use.

Q What conditions, if any, make persons at

special risk for developing adverse effects from expo-

sure to TCE?

A If you look through the literature,

almost anything. Any heart problem .increases the

flR305b73

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2 chance that you'll have a cardiac response to TCE an x

3 heart disease is the most common cause of death in the

4 country.

5 Any kind of neurological or psychologi-

6 cal problem will put a person at increased risk for

7 neurological symptoms.

8 Any kidney problem or liver problem like

9 we've mentioned, the hepatitis and so on, will put

10 those organs at increased risk within that particular

11 person.

12 Anybody who doesn't metabolize quite as

13 efficiently as the normal healthy adult male worker,

14 such as the elderly, pregnant women, children, in- v \

15 fants, and so on, are also at increased risk.

16 Q Are unborn children -- fetuses --

17 affected by this?

13 . A Yes.

19 Q How long does it take for something like

20 TCE to show up in the system of a fetus after the

21 mother is exposed?

22 A About two minutes.

23 Q That is for TCE?

24 A Yes.

25 Q Why are pregnant and elderly people at

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. - 1994

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A There's really two reasons. One is that

in infants, for instance, the brain is not totallyt

developed, so their brain is at increased risk for the

neurological system. It's at increased risk to any-

thing in that both children and the elderly do not

metabolize or detoxify chemicals as efficiently as

adults do in general and healthy adult male workers in

particular, which is what all the occupational stan-

dards are set to protect.

Q You've talked about the word "metabo-

lized." Could you explain to us what metabolize and

metabolism means?

A Simply speaking, it's the biochemical.

steps that the body takes to remove the substance.

It's a multi-step process and there are steps along

the way that are more toxic, can be more toxic than

the original chemical. The end result is to make the

chemical more easily exhaled or more easily excreted

in the urine, which is the primary route of excretion,

it's the detoxication pathway.

Q Can you describe the interaction, if

any, of TCE with caffeine that has been ingested by

humans? '

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1995

A Both caffeine and nicotine are stimu-

lants. They affect the heart. Therefore, exposure to

either of those would potentiate the effects of TCE or

TCA and vice versa. Pre-exposure to one of the sol-

vents would increase the adverse effects to caffeine

and nicotine.

Should I define "potentiate?1'

Q Please go ahead?

A That's when the effect of both chemicals

together is worse than the effect of either one by

themselves, or the response to the one chemical is

made worse by pre-exposure to another chemical.

Q Can you describe the metabolic interac j

tion of TCE with TCA in humans?

A Again, they are both metabolized by

identically the same enzyme system. Therefore, expo-

sure to one either competes with the detoxification of

the second one — that's the primary way in which you

think of interactions.

Q You said that "potentiate" means that it

makes the effects of a substance worse when there's

more than one substance present than it would be if it

were alone.

Do TCE and TCA potentiate each other?

• o

1996

2 . A Yes. And TCE and TCA and alcohol, and

3 TCE and TCA and the other chlorinated solvents, and

* caffeine and nicotine, and even other chemicals that

5 we haven't talked about like benzene and toluene --

6 benzene in particular which is also metabolized by the

7 same enzymes.

8 Q So that if TCE is present along with one

9 of these other substances like TCA, it will make ad-

10 verse health effects of either of those substances

11 worse than you would expect if they were alone?

12 A Right.

13 JUDGE FITZPATRICK: Ms. Wasicek, do you

14 have any objection to taking our lunch break now?

15 MS. WASICEK: No, Your Honor.

16 JUDGE FITZPATRICK: All right. Let's

17 take a break until 2:00. It's my intention to go to

18 4:30 this afternoon.

19

20 (Lunch recess from 1:00 to 2:00 p.m.)

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