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Page 1: EKG Interpretation

EKG Interpretation

UNC Emergency MedicineMedical Student Lecture

Series

Page 2: EKG Interpretation

Objectives The Basics Interpretation Clinical Pearls Practice

Recognition

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The Normal Conduction System

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Lead Placement

aVF

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All Limb Leads

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Precordial Leads

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EKG Distributions Anteroseptal: V1, V2, V3,

V4 Anterior: V1–V4 Anterolateral: V4–V6, I,

aVL Lateral: I and aVL Inferior: II, III, and aVF Inferolateral: II, III, aVF,

and V5 and V6

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Waveforms

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Interpretation Develop a systematic approach to

reading EKGs and use it every time The system we will practice is:

Rate Rhythm (including intervals and

blocks) Axis Hypertrophy Ischemia

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Rate Rule of 300- Divide 300 by the

number of boxes between each QRS = rate

Number of big boxes

Rate

1 300

2 150

3 100

4 75

5 60

6 50

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Rate HR of 60-100 per minute is normal HR > 100 = tachycardia HR < 60 = bradycardia

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Differential Diagnosis of Tachycardia

Tachycardia

Narrow Complex

Wide Complex

Regular STSVTAtrial flutter

ST w/ aberrancySVT w/ aberrancy

VT

Irregular A-fibA-flutter w/ variable conductionMAT

A-fib w/ aberrancy

A-fib w/ WPWVT

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What is the heart rate?

(300 / 6) = 50 bpm

www.uptodate.com

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Rhythm Sinus

Originating from SA node

P wave before every QRS

P wave in same direction as QRS

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What is this rhythm?Normal sinus rhythm

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Normal Intervals PR

0.20 sec (less than one large box)

QRS 0.08 – 0.10 sec (1-2

small boxes) QT

450 ms in men, 460 ms in women

Based on sex / heart rate

Half the R-R interval with normal HR

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Prolonged QT Normal

Men 450ms Women 460ms

Corrected QT (QTc) QTm/√(R-R)

Causes Drugs (Na channel blockers) Hypocalcemia, hypomagnesemia, hypokalemia Hypothermia AMI Congenital Increased ICP

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Blocks AV blocks

First degree block PR interval fixed and > 0.2 sec

Second degree block, Mobitz type 1 PR gradually lengthened, then drop QRS

Second degree block, Mobitz type 2 PR fixed, but drop QRS randomly

Type 3 block PR and QRS dissociated

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What is this rhythm?

First degree AV block PR is fixed and longer than 0.2 sec

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What is this rhythm?

Type 1 second degree block (Wenckebach)

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What is this rhythm?

Type 2 second degree AV block Dropped QRS

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What is this rhythm?

3rd degree heart block (complete)

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The QRS Axis

Represents the overall direction of the heart’s activity

Axis of –30 to +90 degrees is normal

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The Quadrant Approach QRS up in I and up in aVF =

Normal

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What is the axis?

Normal- QRS up in I and aVF

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Hypertrophy Add the larger S wave of V1 or V2

in mm, to the larger R wave of V5 or V6.

Sum is > 35mm = LVH

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Ischemia Usually indicated by ST changes

Elevation = Acute infarction Depression = Ischemia

Can manifest as T wave changes Remote ischemia shown by q

waves

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What is the diagnosis?

Acute inferior MI with ST elevation in leads II, III, aVF

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What do you see in this EKG?

ST depression II, III, aVF, V3-V6 = ischemia

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Let’s PracticeThe sample EKGs were obtained from the following text:

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Normal Sinus Rhythm

Mattu, 2003

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First Degree Heart Block

PR interval >200ms

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Accelerated Idioventricular

Ventricular escape rhythm, 40-110 bpm

Seen in AMI, a marker of reperfusion

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Junctional Rhythm

Rate 40-60, no p waves, narrow complex QRS

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Hyperkalemia

Tall, narrow and symmetric T waves

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Wellen’s Sign

ST elevation and biphasic T wave in V2 and V3Sign of large proximal LAD lesion

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Brugada Syndrome

RBBB or incomplete RBBB in V1-V3 with convex ST elevation

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Brugada Syndrome Autosomal dominant genetic

mutation of sodium channels Causes syncope, v-fib, self

terminating VT, and sudden cardiac death

Can be intermittent on EKG Most common in middle-aged males Can be induced in EP lab Need ICD

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Premature Atrial Contractions

Trigeminy pattern

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Atrial Flutter with Variable Block

Sawtooth wavesTypically at HR of 150

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Torsades de Pointes

Notice twisting pattern

Treatment: Magnesium 2 grams IV

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Digitalis

Dubin, 4th ed. 1989

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Lateral MI

Reciprocal changes

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Inferolateral MI

ST elevation II, III, aVF

ST depression in aVL, V1-V3 are reciprocal changes

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Anterolateral / Inferior Ischemia

LVH, AV junctional rhythm, bradycardia

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Left Bundle Branch Block

Monophasic R wave in I and V6, QRS > 0.12 secLoss of R wave in precordial leadsQRS T wave discordance I, V1, V6Consider cardiac ischemia if a new finding

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Right Bundle Branch Block

V1: RSR prime pattern with inverted T waveV6: Wide deep slurred S wave

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First Degree Heart Block, Mobitz Type I (Wenckebach)

PR progressively lengthens until QRS drops

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Supraventricular Tachycardia

Narrow complex, regular; retrograde P waves, rate <220

Retrograde P waves

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Right Ventricular Myocardial Infarction

Found in 1/3 of patients with inferior MI

Increased morbidity and mortality

ST elevation in V4-V6 of Right-sided EKG

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Ventricular Tachycardia

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Prolonged QT

QT > 450 ms

Inferior and anterolateral ischemia

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Second Degree Heart Block, Mobitz Type II

PR interval fixed, QRS dropped intermittently

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Acute Pulmonary Embolism

SIQIIITIII in 10-15%

T-wave inversions, especially occurring in inferior and anteroseptal simultaneously

RAD

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Wolff-Parkinson-White Syndrome

Short PR interval <0.12 secProlonged QRS >0.10 secDelta waveCan simulate ventricular hypertrophy, BBB and previous MI

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Hypokalemia

U wavesCan also see PVCs, ST depression, small T waves

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Thank You

Any Questions?


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