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    Maternal & Child Nursing CareSecond Edition

    Marcia L. London Patricia W. Ladewig Jane W. Ball Ruth C. Bindler

    Lecture Notes

    Chapter 15Pregnancy at Risk: Gestational OnsetMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Causes of Bleeding During the First and Second Trimester Abortion: Expulsion of the fetus before 20 weeks gestation

    Expulsion of fetus less than 500g Spontaneous: Occur naturally Induced: Caused by medical or surgical means

    Medical therapy: Bed rest and abstinence from sex

    Persistent bleeding: Hospitalization IV therapy or blood transfusions Dilatationand curettage (D&C) or suction evacuation 2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Miscarriage Related to chromosomal abnormalities Classification

    Threatened abortion Imminent abortion Complete abortion Incomplete abortion Missed abortion Recurrent abortion Septic abortion

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    AFIGURE 151 Types of spontaneous abortion. A, Threatened. The cervix is not dilated, and the placenta is still attached to the uterine wall, but some bleeding occurs. B, Imminent. The placenta has separated from the uterine wall, the cervix has dilated, and the amount of bleeding has increased. C, Incomplete. The embryoor fetus has passed out of the uterus, but the placenta remains.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 by

    Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    BFIGURE 151 (continued) Types of spontaneous abortion. A, Threatened. The cervix is not dilated, and the placenta is still attached to the uterine wall, but somebleeding occurs. B, Imminent. The placenta has separated from the uterine wall,the cervix has dilated, and the amount of bleeding has increased. C, Incomplete.The embryo or fetus has passed out of the uterus, but the placenta remains.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 by

    Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    CFIGURE 151 (continued) Types of spontaneous abortion. A, Threatened. The cervix is not dilated, and the placenta is still attached to the uterine wall, but somebleeding occurs. B, Imminent. The placenta has separated from the uterine wall,the cervix has dilated, and the amount of bleeding has increased. C, Incomplete.The embryo or fetus has passed out of the uterus, but the placenta remains.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 by

    Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Spontaneous Abortion: Treatment Bed rest Abstinence from coitus D&C or suction evacuation Rh immune globulin

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Spontaneous Abortion: Nursing Care Assess the amount and appearance of any vaginal bleeding Monitor the womans vitalsigns and degree of discomfort Assess need for Rh immune globulin Assess fetalheart rate Assess the responses and coping of the woman and her familyMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Ectopic Pregnancy: Risk Factors Tubal damage Previous pelvic or tubal surgery Endometriosis Previous ectopic pregnancy Presence of an IUD High levels of progesterone

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Je

    rsey 07458

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    Ectopic Pregnancy: Risk Factors (contd) Congenital anomalies of the tube Use of ovulation-inducing drugs Primary infertility Smoking Advanced maternal age

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Je

    rsey 07458

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    Causes of Bleeding During First Half of Pregnancy Ectopic pregnancy

    Implantation of fertilized ovum in site other than uterus Mortality rates declin

    ed almost 90% Initially symptoms of pregnancy Positive hCG present in blood andurine Chorionic villi grow into tube wall or implantation site Rupture and bleeding into the abdominal cavity occurs 2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Causes of Bleeding During First Half of Pregnancy (contd) Ectopic pregnancy

    Result is sharp unilateral pain and syncope Referred shoulder pain Lower abdominal pain Vaginal bleeding

    Medical therapy: Intramuscular methotrexate if future pregnancy desired Surgicaltherapy: Salpingostomy or salpingectomyMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    PATHOPHYSIOLOGY ILLUSTRATED: ECTOPIC PREGNANCY Various implantation sites in ectopic pregnancy. The most common site is within the fallopian tube, hence the name tubal pregnancy.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Ectopic Pregnancy: Nursing Care Assess the appearance and amount of vaginal bleeding Monitor vital signs Assessthe womans emotional status and coping abilities Evaluate the couples informational needs Provide post-operative careMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Gestational trophoblastic disease Gestational trophoblastic disease

    Pathologic proliferation of trophoblastic cells Includes:

    hydatidiform mole Invasive mole (chorioadenoma destruens) Choriocarcinoma, a form of cancer

    Initially, clinical picture similar to pregnancy Classic signs: Uterine enlargement greater than gestational age, vaginal bleeding 2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Gestational Trophoblastic Disease: Symptoms Vaginal bleeding Anemia Passing of hydropic vesicles Uterine enlargement greaterthan expected for gestational age Absence of fetal heart sounds Elevated hCGMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Gestational Trophoblastic Disease: Symptoms Low levels of MSAFP Hyperemesis gravidarum Preeclampsia

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Causes of Bleeding During First Half of Pregnancy (contd) Therapy: Suction evacuation of the mole

    Uterine curettage for removal of placental fragments Hysterectomy for excessivebleeding

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    FIGURE 152 Hydatidiform mole. A common sign is vaginal bleeding, often brownish (the characteristic prune juice appearance) but sometimes bright red. In this figure, some of the hydropic vessels are being passed. This occurrence is diagnosticfor hydatidiform mole.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Incompetent Cervix Associated with repeated second trimester abortions Possible causes

    Cervical trauma Infection Congenital cervical or uterine anomalies Increased uterine volume (as with a multiple gestation)

    Diagnosis: Positive history of repeated second trimester abortionsMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Treatment: Surgical Procedures Shirodkar procedure (cerclage) Modification of it by McDonald Reinforces the weakened cervix Purse-string suture is placed in cervix Done in first trimester orearly in second trimester Cesarean birth may be planned Suture may be cut at term and vaginal birth permittedMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 074

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    FIGURE 153 A cerclage or purse-string suture is inserted in the cervix to preventpreterm cervical dilatation and pregnancy loss. After placement the string is tightened and secured anteriorly.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Nursing Interventions Monitor women for premature labor Monitor for premature rupture of membranes Teach client

    Signs of premature labor Signs of premature rupture of membranes

    Tell client to contact healthcare provider if membranes rupture or labor beginsMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Hyperemesis Gravidarum Exact cause of hyperemesis is unclear Increased levels of hCG may play a role Severe cases: Causes dehydration

    Fluid-electrolyte imbalance Alkalosis Metabolic acidosis Decreased urinary output

    2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Aim of Treatment Control vomiting: Antiemetics Correct fluid and electrolyte imbalance potassiumchloride Correct dehydration: Intravenous (IV) fluids Improve nutritional statatus

    Vitamin supplements Total parenteral nutrition

    2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Nursing Care Supportive Directed at maintaining a relaxed environment Maintaining oral hygiene Monitoring weight Monitoring for signs of complications Once oral feedings resume, food needs to be attractively servedMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Premature Rupture of Membranes Spontaneous rupture of membranes before labor Preterm PROM (PPROM): Rupture of membranes before term Maternal risk of infection increases Risk of abruptio placentae Fetal-newborn: Risk of respiratory distress syndrome Fetal sepsis, malpresentation and prolapse of umbilical cord Increased perinatal morbidity and mortalityMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 by

    Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Premature Rupture of Membranes (contd) Prevention of infection

    Use sterile speculum to detect amniotic fluid Limit digital vaginal examinations

    If maternal signs of infection evident, antibiotic therapy started immediately U

    pon admission to nursery: Infant assessed for sepsis, placed on antibioticMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Premature Rupture of Membranes (contd) Absence of infection and gestation age less than 37

    Hospitalization and bed rest Complete blood cell count (CBC) C-reactive proteinand urinalysis Continuous or intermittent fetal monitoring Regular nonstress tests (NSTs) or biophysical profiles Maternal vital signs assessed every 4 hours Re

    gular laboratory evaluations 2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Premature Rupture of Membranes (contd) Absence of infection /gestation age less than 37weeks

    Fetal lung maturity studies Maternal corticosteroid administration Bed rest withbathroom privileges Monitor temperature and pulse every 4 hours Keep fetal movement chart and have weekly NST Call healthcare provider for signs of complicatio

    ns 2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

    If sent home: Discharge instructions

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Preterm Labor Labor that occurs between 20 and 37 weeks gestation Documented uterine contractions (4 in 20 minutes or 8 in 1 hour) Documented cervical change Cervical dilatation of greater than 1 cm Cervical effacement of 80% or more Chart page 347Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007 byPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    TABLE 151Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    Risk Factors for Spontaneous Preterm Labor. 2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    TABLE 152

    Self-Care Measures to Prevent Preterm Labor. 2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Preterm Labor (contd) Management

    Assessment of cervicovaginal fibronectin Assessment of cervical length via ultrasound Obtaining history of previous preterm birth Assess for the presence of infections Educating clients about preterm labor Assessing for early signs and symp

    toms Maternal laboratory studies 2007 by Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Preterm Labor (contd) Management

    IV infusion: Promotes maternal hydration Tocolysis: Medications used to stop labor -adrenergic agonists and magnesium sulfate Prostaglandin synthetase inhi

    itors Calcium channel

    lockers

    2007

    y Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

    Tocolytics

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Preterm La

    or (contd) Nursing management

    Identify woman at risk Assess the progress of la

    or Administration of medications Teach how to recognize onset of la

    or Provide information a

    out community resources

    Assess impact of la

    or on mother and fetus

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007

    y Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Signs and Symptoms of Preterm La

    or Uterine contractions occurring every 10 minutes or less Mild menstrual-like cramps felt low in the ad

    omen Constant or intermittent feeling of pelvic pressure Rupture of mem

    ranes Low, dull

    ackache, which may

    e constant or intermittentMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

    yPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Signs and Symptoms of Preterm La

    or (contd) A change in vaginal discharge A

    dominal cramping with or without diarrhea

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007

    y Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Classification and Pathophysiology Classification

    Gestational (or transient) hypertension Preeclampsia-eclampsia Chronic hypertension Chronic hypertension with superimposed preeclampsia or eclampsia

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007

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    Preeclampsia-eclampsia Definition

    Blood pressure of 140/90 or higher on two occasions at least 6 hours apart accom

    panied

    y proteinuria Signs of impending eclampsia include:

    Scotomata,

    lurred vision, epigastric pain, vomiting, persistent or severe headache, neurologic hyperactivity, pulmonary edema, or cyanosis 2007

    y Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Characteristics of Preeclampsia Maternal vasospasm Decreased perfusion to virtually all organs Decrease in plasma volume Activation of the coagulation cascade Alterations in glomerular capillary endothelium EdemaMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    PATHOPHYSIOLOGY ILLUSTRATED: PRECLAMPSIA A, In a normal pregnancy, the passive quality of the spiral arteries permits increased

    lood flow to the placenta. B, In preclampsia vasoconstriction of the myometrial se

    ment of the spiral arteriesoccurs.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

    yPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Maternal Risks Hyperreflexia and headache Seizures, renal failure and a

    ruptio placentae Disseminated intravascular coagulation (DIC) Ruptured liver and pulmonary em

    olism HELLP syndrome (Hemolysis, Elevated Liver enzymes, and Low Platelet count)Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

    yPearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    Fetal-Neonatal Risks Small for gestational age (SGA) Premature Hypermagnesemia (Magnesium sulfate administration to mother) Increased mor

    idity and mortality

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007

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    rsey 07458

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    Clinical Manifestations and Diagnosis Mild preeclampsia

    BP 140/90 mm Hg or higher 1+ proteinuria may occur Liver enzymes may

    e elevatedminimally Edema may

    e present BP 160/110 mm Hg or higher measurements, 6 hoursapart

    2007

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    Severe preeclampsia

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Clinical Manifestations and Diagnosis Severe preeclampsia

    Proteinuria 5 g in a 24-hour urine collection Dipstick urine protein 31 to 41 on2 random samples Samples must

    e o

    tained at least 4 hours apart Visual or cere

    ral distur

    ances Grand mal convulsion May occur antepartum, intrapartum, or pos

    tpartum

    Eclampsia

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007

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    Management Home care of mild preeclampsia

    Client monitors her

    lood pressure Measures weight and tests urine protein dailyRemote NSTs performed daily or

    i-weekly Advised to report signs of worsening preeclampsia Bed rest and moderate to high protein diet Fetal evaluation

    2007

    y Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

    Hospital care of mild preeclampsia

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    Management (contd) Severe preeclampsia

    Bed rest Diet: High-protein, moderate-sodium Anticonvulsants: Magnesium sulfateFluid and electrolyte replacement Corticosteroids and antihypertensive drugs

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007

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    Management (contd) Eclampsia

    Anticonvulsants: Bolus of magnesium sulfate Sedation and other anticonvulsants:Dilantin Diuretics to treat pulmonary edema Furosemide (Lasix) Digitalis: For circulatory failure Strict monitoring of intake and output

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Management (contd) Nursing care

    Monitor vital signs and auscultate lungs Evaluate fetal heart rate patterns Moni

    tor urinary output and urine protein hourly Check specific gravity of the urinehourly Weigh the woman daily at the same time Assess deep tendon reflexes and clonus 2007

    y Pearson Education, Inc. Pearson Prentice Hall Upper Saddle River, New Jersey 07458

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    TABLE 153Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    Deep Tendon Reflex Rating Scale 2007

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    FIGURE 154 To elicit clonus, with the knee flexed and the leg supported, sharplydorsiflex the foot, hold it momentarily, and then release it. Normally the footreturns to its usual position of plantar flexion. Clonus is present if the foot jerks or taps against the examiners hand. If so, the num

    er of taps or

    eats of clonus is recorded.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    HELLP Hemolysis, elevated liver enzymes, and low platelet

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    Chronic Hypertensive Disease Chronic hypertension exists when the

    lood pressure is 140/90mm Hg or higher

    efore pregnancy or

    efore the 20th week of gestation, or when hypertension persists 42 days following child

    irth. Gestational hypertenison- occurs midpregnancy without proteinuria

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    DIC Definition Disseminated intravascular coagulation (DIC) is a systemic process producing

    oth throm

    osis an hemorrhage. It involves

    Exposure of

    lood to procoagulants Formation of fi

    rin in the circulation Fi

    rinolysis Depletion of clotting factors End-organ damage

    2007

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    Rh Alloimmunization: Causes Rh-negative woman carries an Rh-positive fetus Fetal red

    lood cells cross intomaternal circulation Response: Production of Rh anti

    odies Transfer of RBCs usually occurs at

    irth The first child is not affected Su

    sequent pregnancy

    Rh anti

    odies enter the fetal circulation Result: Hemolysis of fetal red

    lood c

    ells and fetal anemia 2007

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    AFIGURE 155 Rh alloimmunization sequence. A, Rh-positive father and Rh-negative mother. B, Pregnancy with Rh-positive fetus. Some Rh-positive

    lood enters the mothers

    loodstream. C, As the placenta separates, the mother is further exposed tothe Rh-positive

    lood. D, Anti-Rh-positive anti

    odies (triangles) are formed. E,In su

    sequent pregnancies with an Rh-positive fetus, Rh-positive red

    lood cells are attacked

    y the anti-Rh-positive maternal anti

    odies, causing hemolysis of

    the red

    lood cells in the fetus.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    BFIGURE 155 (continued) Rh alloimmunization sequence. A, Rh-positive father and Rhnegative mother. B, Pregnancy with Rh-positive fetus. Some Rh-positive

    lood enters the mothers

    loodstream. C, As the placenta separates, the mother is furtherexposed to the Rhpositive

    lood. D, Anti-Rh-positive anti

    odies (triangles) areformed. E, In su

    sequent pregnancies with an Rh-positive fetus, Rh-positive red

    lood cells are attacked

    y the anti-Rhpositive maternal anti

    odies, causing hem

    olysis of the red

    lood cells in the fetus.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    CFIGURE 155 (continued) Rh alloimmunization sequence. A, Rh-positive father and Rhnegative mother. B, Pregnancy with Rh-positive fetus. Some Rh-positive

    lood enters the mothers

    loodstream. C, As the placenta separates, the mother is furtherexposed to the Rhpositive

    lood. D, Anti-Rh-positive anti

    odies (triangles) areformed. E, In su

    sequent pregnancies with an Rh-positive fetus, Rh-positive red

    lood cells are attacked

    y the anti-Rhpositive maternal anti

    odies, causing hem

    olysis of the red

    lood cells in the fetus.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    DFIGURE 155 (continued) Rh alloimmunization sequence. A, Rh-positive father and Rhnegative mother. B, Pregnancy with Rh-positive fetus. Some Rh-positive

    lood enters the mothers

    loodstream. C, As the placenta separates, the mother is furtherexposed to the Rhpositive

    lood. D, Anti-Rh-positive anti

    odies (triangles) areformed. E, In su

    sequent pregnancies with an Rh-positive fetus, Rh-positive red

    lood cells are attacked

    y the anti-Rhpositive maternal anti

    odies, causing hem

    olysis of the red

    lood cells in the fetus.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    EFIGURE 155 (continued) Rh alloimmunization sequence. A, Rh-positive father and Rhnegative mother. B, Pregnancy with Rh-positive fetus. Some Rh-positive

    lood enters the mothers

    loodstream. C, As the placenta separates, the mother is furtherexposed to the Rhpositive

    lood. D, Anti-Rh-positive anti

    odies (triangles) areformed. E, In su

    sequent pregnancies with an Rh-positive fetus, Rh-positive red

    lood cells are attacked

    y the anti-Rhpositive maternal anti

    odies, causing hem

    olysis of the red

    lood cells in the fetus.Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    Rh Alloimmunization: Fetal and Neonatal Risks Anemia Hemolytic syndrome Erythro

    lastosis fetalis

    Marked fetal edema, called hydrops fetalis Congestive heart failure Marked jaundice

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007

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    Rh Alloimmunization: Prevention Screen for Rh incompati

    ility and sensitization

    Take a history Identify Rh-negative woman Anti

    ody screen (indirect Coom

    s test)Identifies if woman is sensitized Give injection of 300 mcg Rh immune glo

    ulin

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007

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    Rh Alloimmunization: Prevention (contd) Give Rh immune glo

    ulin in the following cases

    Pregnant Rh-women who have no anti

    ody titer At 28 weeks gestational age Mother whose

    a

    ys father is Rh positive or unknown After each a

    ortion and within 72 hours postpartum Amniocentesis and placenta previa Invasive procedures that may cau

    se

    leeding

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007

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    TABLE 154Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    Rh Alloimmunization 2007

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    ABO Incompati

    ility Cause: Mother has type O

    lood and infant has A, B, or AB

    Anti-A and anti-B anti

    odies occur naturally During pregnancy maternal anti

    odie

    s cross placenta Cause hemolysis of the fetal red

    lood cells Unlike Rh incompati

    ility, first infant is often involved, no evidence of repeated sensitization,no antepartal treatment 2007

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    ABO Incompati

    ility (contd) Creates hyper

    iliru

    inemia in the infant Hyper

    iliru

    inemia is treated with phototherapy Assess for potential for ABO incompati

    ility - type O mother and type Aor B father Following

    irth

    New

    orn assessed carefully Asses for development of hyper

    iliru

    inemia

    Unlike Rh incompati

    ility, it cannot

    e preventedMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    Effects of Surgical Procedures First trimester surgery: Increase incidence of a

    ortion Increased incidence of fetal mortality Low-

    irth-weight (less than 2500 g) infants Increased incidence of preterm la

    or Increased incidence of intrauterine growth restriction Ina

    ilityto perform some diagnostic procedures (x-ray) - may hinder diagnosis of diseaseduring pregnancyMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

    y

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    Special Considerations Surgery during early second trimester decreases risk of complication During surgery, wedge placed under mothers hip prevents uterine compression of major

    lood vessels Insertion of nasogastric tu

    e to decrease vomiting An indwelling catheter

    Prevents

    ladder distension Facilitates monitoring of output

    2007

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    Special Considerations (contd) Fetal heart rate must

    e monitored electronically during and after surgery Postoperatively

    Encourage to turn,

    reathe deeply, and cough Encourage use of ventilation therapy Early am

    ulation to prevent complications

    Discharge teaching is very importantMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    Impact of Trauma During Pregnancy Types of trauma

    Blunt trauma Penetrating injuries Gunshot wounds Falls Direct assaults Maternalshock Premature la

    or or spontaneous a

    ortion 2007

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    rsey 07458

    Causes: Motor vehicle accident - most common

    Impact

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    Impact of Trauma During Pregnancy (contd) Maternal mortality: From head trauma or hemorrhage

    Uterine rupture is rare Placental a

    ruption High rate of fetal mortality Premature

    irth

    Traumatic separation of the placenta

    Early rupture of mem

    ranesMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    Treatment Major injuries

    Life-saving measures for woman Esta

    lishing an airway Control external

    leeding

    Administer IV fluid to alleviate shock Kept on her left side to prevent furtherhypotension Oxygen is administered at 100% Exploratory surgery may

    e necessary 2007

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    Treatment (contd) Fetus near term and uterus damaged: Cesarean section Fetus immature

    Uterus can

    e repaired Pregnancy continue to term

    Evaluation of fetal heart rate and movement Minor injuries

    Fetal monitoring for minimum of 4 hours Signs of o

    stetric complications such asuterine

    leeding Monitoring for 24 hours is recommended 2007

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    Physical A

    use During Pregnancy Incidence: 4% to 8% May result in loss of pregnancy Preterm la

    or, low-

    irth-weight infants, and fetal death A

    used women have higher rates of complications

    Anemia, infection, and low weight gain First- and second-trimester

    leeding 2007

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    rsey 07458

    Be alert for non-specific signsMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

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    Physical A

    use During Pregnancy (contd) Management: Early detection Ask a

    out a

    use at several prenatal visits Client may only disclose a

    use after knowing her caregivers Assess old scars on parts ofthe

    ody Be alert for signs of

    ruising: Target areas of violence during pregnancy

    Clients

    reasts A

    domen or genitalia 2007

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    Treatment Create an accepting, nonjudgmental environment Allow client to express her concerns She needs to

    e aware of community resources

    Emergency shelters Police, legal, and social services Counseling

    Client has to make decision to seek assistanceMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    Prenatal Infections Toxoplasmosis: Protozoan toxoplasma gondii Transmission

    Eating raw or undercooked meat Contact with the feces of infected cats

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007

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    Prenatal Infections (contd) Fetal-neonatal risks

    Fetal infection Severe fetal disease or death

    Severe neonatal disorders Treatment

    Sulfadiazine and pyrimethamine Given after the first trimester

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler

    2007

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    Prenatal Infections (contd) Ru

    ella: Virus Transmission: Across placenta to fetus

    Fetal neonatal infection Infant should

    e isolated Ru

    ella syndrome Vaccinationof all children Vaccination of women of reproductive age 2007

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    rsey 07458

    Treatment: Prevention

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    Prenatal Infections (contd) Cytomegalovirus: Virus Transmission

    Across placenta to fetus Cervical route during

    irth

    Fetal infection Fetal death Neonatal disorders Treatment: Currently none exist

    Maternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    Prenatal Infections (contd) Herpes simplex virus: HSV-1 or HSV-2 Transmission: Ascending infection during

    irth

    After mem

    ranes rupture Transplacental: Rare

    Neonatal infection Treatment: Antiviral therapy (acyclovir) Active herpes lesion: Cesarean section No evidence of genital infection exists, vaginal

    irth is preferredMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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    Prenatal Infections (contd) Group B streptococcal infection (GBS) -

    acterial infection Transmission: Vertical from mother during

    irth

    From colonized nursing personnel From colonized infants

    Neonatal infection treated with anti

    iotics Prevention

    Early identification Anti

    iotic prophylaxis 2007

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    Prenatal Infections (contd) Other Infections

    Urinary tract infections Vaginal infections Sexually transmitted infections

    Maternal infections may cause spontaneous a

    ortions. Some evidence links infecti

    on and prematurity Risk of maternal and fetal mor

    idity and mortality Early diagnosis and treatment is necessaryMaternal & Child Nursing Care, 2/e By London / Ladewig / Ball / Bindler 2007

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