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More than 30 trinucleotide repeat expansion diseases have been identified
Such diseases often display “genetic anticipation.”
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In the SOS response inE. coli, activated RecAcleaves the LexA repressor and leads toinduction of a numberof proteins, includingPol V
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Note– this is not reallyrepair. It’s an exampleof “damage tolerance.” (Also, the detailed mechanism is not exactly as depicted here.)
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