neoneto mcq answers.doc

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1. Ans. E

Explanation: Sudden infant death syndrome (SIDS) usually does not occur in the first month of

life. Usual age is 1 month to 1 year. SIDS should not e confused !ith apnea of prematurity" ecause one is not related to the other. Apnea of prematurity occurs in the first month of life.

#. Ans. D. $horioamnionitis is the asolute contraindication for prenatal steroids.

Also decreases the incidence of %E$" E&S" de'elopmental delay

post natal gro!th not ad'ersely affected

Do not increase puerperal sepsis. Dexamethasone is more eneficial in reducing I

*etamethasone is no! preferred

+. Ans. itamin A (,"--- IU I + times/!0 for !0) in 2*3 infants reduces the ris0

*4D5 result of lung in6ury in infants re7uiring mechanical 'entilation and supplemental oxygen

4rimarily a disease of 81"--- gm or 8#9 !ee0s

Early use of nasal $4A4 and rapid extuation !ith transition to nasal $4A4 are associated !ith a

decreased ris0 of *4D.

. Ans. A. %eonatal mortality rate is the numer of deaths in first #9 days of life for 1"--- li'e

irths. Stillirths are not considered !hile calculating this.

,. Ans. A. ponderal index !t/ht+ (cue) ;!t in 0g and ht. in meters)

or ponderal index 1-- <!t/ht+ (cue)= ;!t in grams and ht in centimeters>

here in this 7uestion"

4I 1-- <#---/,-?,-?,-= ;i.e.cue of ,- in denominator> 1-- <#/1#,= 1.@

%ormal more than #

In asymmetric IUB" less than #

Symmetric IUB more than #

@. Ans. *.



Cetracycline5 retarded physical gro!th

alproate5 spinal dysraphism" cleft palate" cardiac and neurological

2ithium5 Estein anomaly

4rednisolone5 oral clefts

Isotretinoin5 congenital heart disease

. Ans. *. olic acid protects almost t!o third cases of neural tue defects" not all.

olic acid talets should e ta0en periconceptionally" ie atleast + months efore concei'ing and

has to e continued for + months after ecoming pregnant.

Che dose is --microgram per day.

*ut if there is past or family history of neural tue defects" the dose is , mg per day.

9. Ans: $. IUB is a main feature" facial dysmorphism" seiFure disorder" ADD

Due to maternal inta0e of alcohol during pregnancy

G. Ans. D. 4remature rupture of memranes (4B&) is associated !ith ris0s of early5onsetsepsis and premature irth. *oth should contriute to neonatal" not postneonatal" mortality.

&ther ris0 factors for E&S

1-. Ans. A. Esophageal atresia is most often associated !ith polyhydramnios" as are other upper

intestinal ostructi'e lesions or disorders of fetal s!allo!ing and neural tue defects.

4otter syndrome5 ilateral renal anomaly leading to se'ere oligamnios5 due to pressure effect"

anormal face" $CE. Also lung hypoplasia. 4rune elly syndrome5 also called Criad Syndrome

and Eagle *arrette Syndrome !ith deficient adominal muscles" undescended testes and urinary

tract anomalies" ecause of se'ere urethral ostruction in fetal life.

4olyhydramnios in anencephaly" CE" intestinal atresias" neuromuscular diseases

Anencephaly" ydrocephaly" Cracheoesophageal fistula

Duodenal atresia

Spina ifida $left lip or palate



$ystic adenomatoid lung malformation

Diaphragmatic hernia

Syndromes: Achondroplasia

Hlippel5eil Crisomy 19

Crisomy #1

C&B$ (toxoplasmosis" other agents" ruella" cytomegalo'irus" herpes simplex)

ydrops fetalis

ultiple congenital anomalies

&ther: Diaetes mellitus

C!in5t!in transfusion (recipient)

etal anemia

etal heart failure

4olyuric renal disease

%euromuscular diseases

%onimmune hydrops

$hylothorax

Ceratoma

11. Ans. A. aternal lupus may affect the fetal cardiac conduction system" and it produces

radycardia y the de'elopment of an antiody immune5related process

1#. Ans. E. &ther conditions are Apert syndrome" cleidocranial dysostosis" hydrocephalus"

hypophosphatasia" prematurity" 'it D deficiency ric0ets" trisomies.

Cay5Sach’s disease is a neurodegenerati'e disorder" not manifesting in ne!orn period

2arge head" exaggerated startle response and cherry red spot are manifestations

&I associated !ith !ormian ones

&ther conditions eing py0nodysostosis" cleido cranial dysplasia" hypophosphatasia"

hypoparathyroidism.

Drug used in the management of &I to impro'e one strength and to reduce fracture incidence5

*isphosphonates (I pulse pamidronate" oral alendronate)

1+. Ans. $. ost of the natal teeth are isolated entities" not part of any syndrome.

Usually in the lo!er incisor position. Chese are shed efore the deciduous teeth erupt.

Extraction is indicated only if it interferes !ith feeding or loose so that may get aspitated.



In hypothyroidism" dentition is li0ely to e delayed.

1. Ans. A. Chey represent entrapped epithelium. Another site is tip of penis.

*enign

&nly reassurance needed

1,. Ans *

1@. Ans. A

1. Ans. *. Che Apgar score helps to rapidly assess the need to resuscitate neonates after irth.

Although it has some 'alue in predicting neonatal mortality and cereral palsy" it has a poor

positi'e predicti'e 'alue.

ost children !ith cereral palsy ha'e had normal Apgar scores" !hile neonates !ith lo! Apgar

scores do not uni'ersally get cereral palsy.

19. Ans. E. IC4 in mother5 maternal antiodies cross the placenta and result in thromocytopenia.

yotonic dystrophy in mother5 se'ere neonatal form of myotonic dystrophy in ay.

S2E5 congenital heart loc0" anemia" thromocytopenia" neutropenia" rash.

4HU5 teratogenic effect of ele'ated phenyl alanine5 microcephaly" B" $D

1G. Ans. D.

Different !ays to pre'ent hypothermia are Incuators" radiant !armers" 0eeping the ay in

thermal neutral en'ironment" H$" plexiglass shields" head cap and ody clothing.

$old stress is !hen chest is !arm and extremities cold to touch.

#-. Ans. *.

8#9 !ee0s5 no proper suc0ing efforts" no propulsi'e motility of gut5 needs I fluids.

#95+1 !ee0s5 no suc05s!allo!5reath coordination5 orogastric feeding (ga'age" % tue).

+#5+ !ee0s5 slightly mature suc0ing5 paladai/spoon feeding of E*.



+ !ee0s5 mature suc0ing pattern" coordinated s!allo!ing and reathing5 reast feeding.

Expressed reast mil0 can e stored for @ hours in room temperature and # hours in refrigerator.

#1. Ans. $.

*allard scoring system" assess physical maturity and neuromuscular maturity.

4hysical5 s0in" crease" reast nodule" scrotum" clitoris and laia" ear cartilage" lanugo

##. Ans. D.

Also * complex" $" D" E 'itamins.

Iron supplements usually after 1 mo of irth (once irth !eight is douled)

#+. Ans. E

#. Ans. $.

Superiosteal in location

$rosses the one only if there is a fracture.

*eneath the aponeurosis is sugaleal haematoma" usually follo! 'accum deli'ery or in

coagulopathy.

luctuant s!elling

#,. Ans. D. 4hototherapy is clearly indicated. Aspiration or incision and drainage (I J D) should

not e done to manage a cephalohematoma.

#@. And. D

#. Ans: A. 42 occurs in preterm aies" enhanced 'ulneraility of immature oligodendroglia

to oxidati'e stress due to ischemia" infections or inflammatory insults

2o!er lims more affected" intelligence usually normal

Athetoid/extrapyramidal/chorioathetoid $45 status marmoratus of asal ganglia" 0ernicterus

leading on to lesions of glous pallidus

Sanat and Sarnat staging of IE5 ild5 hyperalert5 stage I



If seiFure present" stage II

Se'ere5 stage III" stuporous or comatose" flaccid" decererate

#9. Ans. A. ost Er palsies resol'e rapidly !ith immoiliFation" rehailitation" and positioning.

If there is no impro'ement et!een +5@ mo" a referral for surgical e'aluation is indicated. Er’s

palsy and fracture cla'icle can lead on to asymmetric oro

#G. Ans: A.

Al'eoli tend to collapse especially during expiration due to lo! end expiratory pressures. Co 0eep

them patent" a positi'e pressure has to e maintained. Chis is y using $4A4

+-. Ans. $. Acrocyanosis is normal at this time" and if the physical examination is also normal

there is no ris0 of a serious underlying disorder. 3arming is all that may e needed

+1. Ans. $.

$linical features are suggesti'e of D

D usually occurs in preterm aies

Becurrence in silings and consanguinity of parents points to an autosomal recessi'e condition

K BDS (D) @-59-L elo! #9!ee0s

K Bis0 is less if 4B&" antenatal corticosteroids" maternal hypertension" maternal heroin use

K Cype # al'eolar cells5 surfactants are produced (mainly dipalmitoyl phosphatidyl choline" ie

lecithin" 4h" apoproteins and cholesterol)

K Beduce surface tension5 pre'ent atelectasis at end expiration

K Surfactant in amniotic fluid5 y #95+# !ee0s" mature le'els y +, !ee0s

K Symptoms can start after minutes of irth. 2ate onset5 thin0 of other causes

K runting and retractions are important signs" symptoms and signs pea0 !ithin + days

K Impro'ement is heralded y spontaneous diuresis

K M ray5 fine reticulogranular pattern !ith air ronchogram" !hite lung



K DD5 E&S" CA4$" 44%" $$A" CC%*" congenital al'eolar proteinosis (congenital

surfactant protein * deficiency)

K 4re'ention5 timing of electi'e $S" 2:S ratio assessment" a'oiding asphyxia" etamethasone 9hour efore deli'ery for fetuses elo! + !ee0s" early rescue !ith surfactant

K Creatment5 $4A4" assisted mechanical 'entilation" exogenous surfactant therapy" inhaled nitric

oxide

K edical closure of 4DA of prematurity5 I indomethacin or I iuprofen

K *4D5 result of lung in6ury in infants re7uiring mechanical 'entilation and supplemental oxygen

K &ther enefits of prenatal steroids5 I" 4DA" %E$" *4D" pneumothotax" E&S" de'elopmental

delay. %o protection against B&4

K Dexamethasone5 increased ris0 of peri'entricular leucomalacia

K Sha0e test5 gastric aspirate or A mixed !ith asolute alcohol" sha0e for 1, secs and 0eep for

1, minutes. Stale ules on top indicates lung maturity

K %ile lue sulphate test is another test

K 2:S ratio # (+., in case of D)

K Saturated phosphatidyl choline ,--micgm/d2 (phosphatidyl glycerol est in D)

K Bespiratory stimulants used in apnoea of prematurity5 $affeine" aminophylline" doxapram. Also

$4A4 can e used.

K $onfirmation of EC tue position5 end tidal $&# measurement

+#. Ans. $. &ther!ise called BDS type #.

&ccur oth in term and preterm.

ore in electi'e $S.

Spontaneous impro'ement !ith in + days.

Air ronchogram is seen in D.



++. Ans. A.

ore common on left side 9,L" ,L ilateral.

Defect in posterolateral part of diaphragm (*ochdale0)

Scaphoid adomen

a6or limiting factor for sur'i'al is pulmonary hypoplasia and resultant pulmonary hypertension.

,-L can e diagnosed efore # !ee0s y US.

K $ongenital loar emphysema5 left upper loe is commonly affected.

K Air trapping" lung herniation" atelectasis of other loes.

K Usually not symptomatic at irth" ut usually in the neonatal period itself.

K 2oectomy or in mild cases conser'ati'e management

+. Ans. $.

runting is common" particularly after a cesarean section !ithout prior laor.

If grunting persists eyond +- min or if there are other signs of distress" the child should e

e'aluated for sepsis" respiratory distress syndrome (BDS)" or congenital heart disease.

+,. Ans. *.

Apnea from any cause is treated !ith securing a patent air!ay and instituting 'entilation.

If ag mas0 'entilation is ineffecti'e" endotracheal intuation should e performed

%aloxone (%arcan) can e gi'en only after the ay is 'entilated.

+@. Delayed passage of meconium 2oo0 for imperforate anus/AB/myelomeningocele

If normal" contrast enema (to r/o atresia or stenosis) (do not r/o D as transitional Fone formed

only after + !ee0s)

If normal" rectal suction iopsy or anorectal manometry to r/o D

ull thic0ness rectal iopsy for confirmation





Se'ere dehydration and shoc0 can occur.

Adominal distension is not mar0ed .

econium ileus usually occur in those !ith $.

econium is thic0 and 'iscid due to the asence of pancreatic enFymes.

+G. Ans. A.

*o!el sounds are reduced or asent in %E$.

Intestinal necrosis" occurring mainly in preterm aies

Etiological Criad5 intestinal ischaemia" enteral nutrition and pathogenic organisms

$lassic triad of symptoms5 adominal distension" ilious 'omiting and lood in stools

etaolic triad5 metaolic acidosis" hyponatremia and thromocytopenia.

K reatest ris0 factor5 prematurity.

K Adominal tenderness an important feature

K 2ess !ith human mil0.

K 4neumatosis intestinalis (gas accumulation in the sumucosa of the o!el !all).

K 4ortal 'enus gas5 late manifestation.

K 4erforation !ith air under diaphragm

K Staging of %E$5 modified *ell’s staging

-. Ans. *.

All infants !ith ile stained emesis should e e'aluated radiologically.

A plain film or HU* film is not sufficient to detect all anormalities ut should e done efore a

arium intestinal series.

1. Ans. A.



Chis is a case of typical physiologic 6aundice.

%o treatment is needed except for clinical monitoring of 6aundice y assessing its cephalo caudal

progression.

Chis can e the 6aundice of reast fed ay" ut stoppage of reast feeding is not recommended.

Chis modality is recommended for reast mil0 6aundice !hich is due to the presence of

glucuronidase in some reast mil0.

Chis prolem occur usually after 1 !ee0 of life" iliruin le'el can reach high le'els so that e'en

0ernicterus can de'elop

#. Ans. $. 4hysiological Oaundice5&ccurs in @-59-L of ne!orns

%e'er efore # hours

%e'er 1,mgL

%e'er 1 days in preterm and 1- days in term

Uncon6ugated hyperiliruinemia" ie con6ugated iliruin ne'er #mgL or #-L of total

iliuin (!hiche'er is less)

+. Ans. *

Boughly 1mg/0g iliruin per each 1-- gm ody !eight is considered safe

If 6aundice detected early" more li0ely that further increase !ill occur5 so early inter'ention

If immune hemolysis" more li0ely to !orsen

*irth asphyxia" sepsis" other neurologic prolems5 *** may not e intact

. Ans. * *iliary atresia has to e ruled out

,. Ans. *. *iliruin asors light maximally in the lue range (#-–- nm).

4hotoisomerisation5 re'ersile reaction con'erting the toxic nati'e uncon6ugated P" 1,P5iliruin

into an uncon6ugated configurational isomer P"1,E5iliruin" !hich can then e excreted in ile!ithout con6ugation



Structural isomerisation5 to lumiruin" an irre'ersile structural isomer 5 can e excreted y the

0idneys in the uncon6ugated state5 most effecti'e !hen intensi'e phototherapy is gi'en

K 4hotooxidation– least effecti'e

K *ronFe ay syndrome5 if there is significant amount of direct reacting iliruin

K %ot an indication to stop phototherapy

K ray ay syndrome (the cardio'ascular collapse associated !ith high doses of

chloramphenicol in ne!orns)

K *lue diaper syndrome (isolated tryptophan malasorption)

@. Ans. D. Done 'ia umilical 'ein

*lood 'olume5 9- ml/0g

Doule 'olume exchange5 1@- ml/0g lood is used

*lood group5 (for A*& incompatiility) mother ’s group (&)" Bh compatile !ith mother and ay

Bh incompatiility5 preferaly & negati'e

%eeds cross matching !ith oth mother ’s and ay’s lood

. Ans. *. &rganism in the maternal genital tract or in the deli'ery area

&ccurs !ithin first # hours of life

4redisposing factors5 2*3" 4B&" foul smelling li7uor" multiple 4s" maternal fe'er

Usually manifest as pneumonia" meningitis rare

K Sepsis screen52eucopenia !ith C2$ 8,"---/cmm

A%$ 819--5elie'ed to e the est predictor

*and cell count more than #-L



Ele'ated $B4 1mg/d2

igh micro ESB (normal 'alue postnatal day J+ or any 'alue 1,)

astric aspirate containing neutrophills (e'idence of chorioamnionitis)

K Sclerema53ooden hardening of s0in and sucutaneous tissue

*etter appreciated on chee0

Associated underlying conditions include pneumonia" septicemia" hypothermia" metaolic

acidosis" respiratory distress syndrome" congenital heart defects" gastroenteritis" and intestinal

ostruction

4oor prognosis" mortality high

9. Cerm ne!orn5 can ha'e (-5+#) cells/mm+

4% 15#/mm+ (up to @1L)

%o B*$s

4rotein G- (#-51-)mgL

lucose5 ,# (+511G)mg/d2

@-L of lood glucose

4reterms also" similar 'alues

%o B*$s in nontraumatic sample

Ans: *

G. Ans. $.

Usual organisms causing meningitis in ne!orn Hlesiella" E. coli" Staphylococcus" BSA"

2isteria" 4seudomonas

,-. Ans. A

,1. Ans. $

,#. Ans<c=



,-L of neonatal seiFures are sutle

3orst prognosis5 myoclonic seiFures

*est prognosis5 focal clonic

Aetiologically" SA and late onset hypocalcaemia5 est neurode'elopmental outcome

4eculiarities of neonatal seiFures

K eneraliFed seiFures are uncommon

K aximum incidence of seiFures compared to any other age group

K ocal seiFure need not mean a localiFed prolem" for eg" hypoglycemia can produce focal

seiFures.

K Immaturity of ner'ous system is the reason for this.

K ocal seiFures5 localiFed structural lesions" infections" suarachnoid hemorrhage

K ultifocal clonic" tonic" myoclonic and sutle seiFures are other types

K Sutle5 che!ing mo'ements" excessi'e sali'ation" apnoea" lin0ing" nystagmus" icycling and

pedaling mo'ements" cyanosis

K eatures !hich differentiate seiFures from non epileptic acti'ity in ne!orn5 presence of

autonomic changes" cannot e suppressed y gentle restraint" not enhanced y sensory stimuli.

K $ommon metaolic causes5 hypoglycemia" hypocalcaemia" hypomagnesaemia" hypo or

hypernatraemia

K $ommonest cause of neonatal seiFures5 IE

,+. Ans: a.

&ther causes of neonatal seiFures5 irth trauma" intracranial leeds" structural rain anormalities"

inorn errors of metaolism" 0ernicterus"

,. Ans: a.



Bis0 of hypoglycemia5 1,5#,L" usually asymptomatic

Bis0 of hypoglycemia is higher if mother is ha'ing o'ert diaetes

aximum ris0 et!een 1 and + hours" spontaneous reco'ery y 5@ hours

Early appearance of 6itteriness" tremulousness" hyperexcitaility" hypotonia" lethargy" poor

suc0ing5 indicate hypoglycemia" late appearance indicate hypocalcemia

$ommonest cardiac prolem is asymmetric hypertrophy of inter'entricular septum

K &ther prolems encountered are

K polycythaemia"

K neonatal 6aundice"

K irth trauma related to macrosomia"

K Bespiratory distress syndrome

K congenital anomalies of heart and ner'ous system (AS" $audal Begression syndrome)

K ost important hormone influencing gro!th in fetal period5 insulin

K During infancy5 thyroxine"

K After infancy" gro!th hormone

K &ther important causes of hypoglycemia in ne!orn5 prematurity" hypothermia" hypoxia"

maternal hyperglycemia in laor" IUB"

K Creatment5 symptomatic5#ml/0g of 1-LDextrose I. o'er 1 minute follo!ed y glucose

infusion @59mg/0g/minute

K Asymptomatic" #,mgL5 oral feeds and monitoring

K Drug used in hyperinsulinemia– hydrocortisone" diaFoxide" octreotide" partial pancreatectomy

,,. Ans. E. Chis is a classic prolem in infants of diaetic mothers (IDs).

All 6ittery children should e examined carefully and concern for a seiFure considered.

If the child appears normal and has no anormal eye mo'ements" and if the motion is sensory5



dependent" is only present during acti'e !a0efulness" and is stopped y simple pressure" it is

proaly not a seiFure.

%onetheless" it could e associated !ith a serious prolem such as hypocalcemia" hypoglycemia"

or opiate !ithdra!al.

IDs are often more 6ittery than other infants ut are also at ris0 for hypoglycemia and

hypocalcemia

,@. Ans. $.

*ay is proaly ha'ing galactosemia

,. Ans. A. $A.

emale presents !ith 'irilisation and clitoromegaly" amiguous genitalia.

In males" difficult to identify.

%ormal genitalia" phallus may e prominent" hyperpigmentation of scrotum" nipples may gi'e a

clue.

Autosomal recessi'e"

$ommonest5 #15 hydroxylase deficiency.

115 hydroxylase5 has hypertension.

4renatal treatment5 if female" gi'e prednisolone to mother

,9. Ans. E. Ans!ers A to D" other congenital heart defects" postnatal hemorrhage ($%S" li'er"

spleen)" and sepsis can all produce hypotension.

,G. Ans. *. Apt Cest

Used to detect the presence or asence of fetal lood (7ualitati'e) in a 'aginal discharge to rule out

'asa pre'ia late in pregnancy

K Co detect the origin of a neonatal loody 'omiting !hether itQs a genuine upper I

hemorrhage/hemoptysis or simply a s!allo!ed maternal lood during deli'ery or from crac0ed

nipple. In Hleihauer5*et0e Cest" the sample is maternal peripheral smear and is used to see ho!

much of fetal lood (7uantitati'e) has een transfused into the maternal serum in order to assess

the ris0 of isoimmuniFation and thence the ris0 of hemolytic disease of the ne!orn.



K *oth of them relies on the fact that is resistant to al0ali (Apt) and acids (Hleihauer) and so

the A containing B*$s (maternal) !ill e hemolyFed ut not the fetal B*$s as they contain the

.

K alse positi'e5 if mother has ele'ated as in sic0le cell disease

@-. Ans. A.

Early onset5 !ithin # hours of irth

Usually secondary to maternal drugs (phenoarital" phenytoin" !arfarin" rifampin" isoniaFid) that

interfere !ith 'itamin H

other can e gi'en 'itamin H to pre'ent this.

$lassic5 #5 days" mainly I leed" incidence is aout #L if 'it H prophylaxis not gi'en at irth.

K 2ate D%5 after 1 !ee0" upto @ mo.

K Usually intracranial" usually secondary to cholestasis

K 4rognosis depends on primary prolem.

K Apt test5 4rinciple is fetal resist al0ali denaturation

K Co pin0 supernatant" add 1L %a&.

K 4IHA5 undercaroxylated forms of the proteins that are normally caroxylated in the presence

of 'itamin H.

K D%5 first anormality5 increased 4IHA" then ele'ated 4C" then only" a4CC

@1. Ans. $. inor clinical prolems in first !ee0 of life are

ilia

ongolian spots

4eeling of s0in

*reast engorgement



%atal teeth

aginal mucoid discharge

K 4hysiological phymosis

K Erythema toxicum

K Stor0 ites

K Sucon6uncti'al haemorrhage

K Epstein pearl" palatal and prepuceal

K aginal leeding

K ymenal tags

K 4hysiologic 6aundice

@#. *oth a and d are correct.

*etter ans!er !ill e d

*ulging fontanel is seen in acute 'itamin A toxicity

@+. $raniofacial ne'us is the ans!er

In S3S" port !ine stain o'er the distriution of trigeminal ner'e" present at irth" do not disappear

%ipple li0e lesions on ody due to 'enous malformation !ith similar lesions in intestine leading to

I leed5 lue ruer le ne'us syndrome

K Stor0 ites" angel’s 0isses" de'il’s pinch or salmon patches5 present at irth" later disappear

K Stra!erry angiomas5 usually asent or 'ery small at irth" gradually increase up to 1 year" then

gradually regress and disappear y , years

@. Ans!er is A" occur in aout ,-L of term ne!orns"

Bepresent collection of eosinophils. $ultures are sterile. %o treatment needed



Cransient neonatal pustular melanosis5 + types of lesions" superficial pustules" ruptured pustules

!ith central scales and hyperpigmentation" hyperpigmented macules.

Aggregation of polymorphs" culture sterile" no therapy needed" present at *irth

@,. Ans: .

Associated anormalities" include intestinal prolems (eg" ec0el di'erticulum" intestinal atresia)"

genetic syndromes (eg" *ec0!ith53iedemann" trisomy 19)" and congenital heart disease.

Umilical hernia has s0in and peritoneal co'er intact

&mphalocele5 peritoneal present" s0in asent

astroschisis5 no peritoneal or s0in co'er

@@. Ans. $

@. Ans. E. $hild ause is often associated !ith ilateral retinal hemorrhages" especially in !hat

is called the Rsha0en ay syndrome.“

Betinal hemorrhages may e noted immediately after irth. %onetheless" they should all resol'e

y 1 mo of age

@9. Ans. $.

3idest rectal diameter is usually smaller than !idest sigmoid diameter in D e'en in the asence

of significant transition Fone

3idest diameter of rectum/ that of sigmoid loop

@G. Ans. *

-. Ans. $

1. Ans. $

#. Ans. D. It is sporadic

+.

. 5555

,. Ans. D



@. All the ao'e

. Ans. E

9. Ans. D

G. All the ao'e

9-. Ans. $

91. Ans. *

9#. Ans. A

9+. Ans. A

9. Ans. *

9,. Ans. $

9@. Ans. D

9. Ans.

99. Ans. D

9G. Ans. A

G-. Ans. D

G1. Ans. D

G#. Ans. D

G+. Ans. $

G. Ans. A

G,. Ans. *

G@. Ans. D

G. Ans. $



G9. Ans. *

GG. Ans. D

1--. Ans. *

1-1. Ans. D

1-#. Ans. $

1-+. Ans. $

1-. Ans. A

1-,. Ans. A

1-@. Ans. $

1-. Ans. $

1-9. Ans. $

1-G. Ans. D

ilia: Ciny cysts" usually found on the face" occurring in up to -L of ne!orns

Epstein pearls: $ysts found on the palate of approximately @L of ne!orns

*ohn nodules: Al'eolar cysts

All three forms represent cystic retention of 0eratin" appear and resol'e in the first month" and can

e present at irth. Chey are !hite" 15 to #5mm papules that can e found singularly or in clusters

11-. Ans. A

111. Ans. D

11#. Ans. E. Seen more in exposure to cold en'ironment

11+. Ans. D

11. Ans. E



11,. Ans. A

11@. Ans. *

11. Ans. A

119. Ans.

11G. Ans. D

1#-. Ans. $

1#1. Ans. A

1##. Ans. *

1#+. Ans.

1#. Ans. *

1#,. Ans. A

1#@. Ans. *

1#. Ans. *

1#9. Ans. *

1#G. Ans. *

1+-. Ans. D

1+1. Ans. *

1+#. Ans. $

1++. Ans. D

1+. Ans. A

1+,. Ans. A

1+@. Ans. A



1+. Ans. E

1+9. Ans. D

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neoneto mcq answers.doc

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